| Publication |
Sentence |
Publish Date |
Extraction Date |
Species |
| E Hirsch, O C Snead, M Vergnes, F Gille. Corpus callosotomy in the lithium-pilocarpine model of seizures and status epilepticus. Epilepsy research. vol 11. issue 3. 1992-11-12. PMID:1396533. |
the lesion of the hippocampal commissure may have contributed to the protective effect of scc, since animals with an isolated lesion of the hippocampal commissure without scc survived the status and showed an increased latency to seizure and status epilepticus. |
1992-11-12 |
2023-08-11 |
rat |
| J B Perlin, S B Churn, E W Lothman, R J DeLorenz. Loss of type II calcium/calmodulin-dependent kinase activity correlates with stages of development of electrographic seizures in status epilepticus in rat. Epilepsy research. vol 11. issue 2. 1992-08-03. PMID:1319899. |
'continuous' hippocampal stimulation (chs), a new model of non-convulsive limbic status epilepticus (se), mimics the progression of electrographic changes characteristic in human se and allows for quantitation of post-stimulus seizure severity. |
1992-08-03 |
2023-08-11 |
human |
| R S Jope, X G. Seizures increase acetylcholine and choline concentrations in rat brain regions. Neurochemical research. vol 16. issue 11. 1992-06-22. PMID:1815138. |
status epilepticus induced by (i) coadministration of lithium and pilocarpine caused massive increases in the concentration of acetylcholine in the cerebral cortex and hippocampus, (ii) a high dose of pilocarpine did not cause an increase of acetylcholine, and (iii) kainate increased acetylcholine, but the magnitude was lower than with the lithium/pilocarpine model. |
1992-06-22 |
2023-08-11 |
rat |
| R S Jope, X G. Seizures increase acetylcholine and choline concentrations in rat brain regions. Neurochemical research. vol 16. issue 11. 1992-06-22. PMID:1815138. |
the finding that the acetylcholine concentration increases in two models of status epilepticus in the cortex and hippocampus is in direct contrast with many in vitro reports in which excessive stimulation causes depletion of acetylcholine. |
1992-06-22 |
2023-08-11 |
rat |
| K Inoue, K Morimoto, K Sato, N Yamada, S Otsuk. Mechanisms in the development of limbic status epilepticus and hippocampal neuron loss: an experimental study in a model of status epilepticus induced by kindling-like electrical stimulation of the deep prepyriform cortex in rats. Acta medica Okayama. vol 46. issue 2. 1992-06-04. PMID:1533485. |
mechanisms in the development of limbic status epilepticus and hippocampal neuron loss: an experimental study in a model of status epilepticus induced by kindling-like electrical stimulation of the deep prepyriform cortex in rats. |
1992-06-04 |
2023-08-11 |
rat |
| G Le Gal La Salle, G Rougon, A Vali. The embryonic form of neural cell surface molecule (E-NCAM) in the rat hippocampus and its reexpression on glial cells following kainic acid-induced status epilepticus. The Journal of neuroscience : the official journal of the Society for Neuroscience. vol 12. issue 3. 1992-04-16. PMID:1545243. |
the embryonic form of neural cell surface molecule (e-ncam) in the rat hippocampus and its reexpression on glial cells following kainic acid-induced status epilepticus. |
1992-04-16 |
2023-08-11 |
rat |
| T F Freund, A Ylinen, R Miettinen, A Pitkänen, H Lahtinen, K G Baimbridge, P J Riekkine. Pattern of neuronal death in the rat hippocampus after status epilepticus. Relationship to calcium binding protein content and ischemic vulnerability. Brain research bulletin. vol 28. issue 1. 1992-04-09. PMID:1347249. |
pattern of neuronal death in the rat hippocampus after status epilepticus. |
1992-04-09 |
2023-08-11 |
rat |
| T F Freund, A Ylinen, R Miettinen, A Pitkänen, H Lahtinen, K G Baimbridge, P J Riekkine. Pattern of neuronal death in the rat hippocampus after status epilepticus. Relationship to calcium binding protein content and ischemic vulnerability. Brain research bulletin. vol 28. issue 1. 1992-04-09. PMID:1347249. |
the pattern of hippocampal cell death has been studied following hippocampal seizure activity and status epilepticus induced by 110-min stimulation of the perforant pathway in awake rats. |
1992-04-09 |
2023-08-11 |
rat |
| B Meldru. Excitotoxicity and epileptic brain damage. Epilepsy research. vol 10. issue 1. 1992-03-31. PMID:1790773. |
hippocampal lesions occurring acutely after status epilepticus show a high degree of selectivity for hilar interneurones, ca1 pyramidal neurones and ca3 pyramidal neurones. |
1992-03-31 |
2023-08-11 |
Not clear |
| C M DeGiorgio, U Tomiyasu, P S Gott, D M Treima. Hippocampal pyramidal cell loss in human status epilepticus. Epilepsia. vol 33. issue 1. 1992-02-28. PMID:1733757. |
a pilot case-control quantitative study of the hippocampus in patients with severe status epilepticus was performed to identify specific patterns of pyramidal cell loss. |
1992-02-28 |
2023-08-11 |
human |
| R S Jope, G V Johnson, M S Bair. Seizure-induced protein tyrosine phosphorylation in rat brain regions. Epilepsia. vol 32. issue 6. 1992-01-16. PMID:1720736. |
this increase was evident in all three regions examined: cerebral cortex, hippocampus, and striatum; it occurred abruptly with onset of generalized status epilepticus (se) and remained elevated for at least 90 min. |
1992-01-16 |
2023-08-11 |
rat |
| K E VanLandingham, E W Lothma. Self-sustaining limbic status epilepticus. I. Acute and chronic cerebral metabolic studies: limbic hypermetabolism and neocortical hypometabolism. Neurology. vol 41. issue 12. 1992-01-16. PMID:1745354. |
recently, we developed a model of nonconvulsive, self-sustaining limbic status epilepticus (sslse) in which electrographic seizures persist 12 to 24 hours after 90 minutes of continuous hippocampal stimulation. |
1992-01-16 |
2023-08-11 |
rat |
| K E VanLandingham, E W Lothma. Self-sustaining limbic status epilepticus. I. Acute and chronic cerebral metabolic studies: limbic hypermetabolism and neocortical hypometabolism. Neurology. vol 41. issue 12. 1992-01-16. PMID:1745354. |
on the basis of our findings, we postulate a feedback network involving the hippocampus and neighboring parahippocampal structures (the hippocampal-parahippocampal "loop") as a critical substrate for establishing limbic system status epilepticus. |
1992-01-16 |
2023-08-11 |
rat |
| K E VanLandingham, E W Lothma. Self-sustaining limbic status epilepticus. II. Role of hippocampal commissures in metabolic responses. Neurology. vol 41. issue 12. 1992-01-16. PMID:1745355. |
in prior work, we developed a model of self-sustaining limbic status epilepticus (sslse) induced by continuous hippocampal stimulation (chs). |
1992-01-16 |
2023-08-11 |
Not clear |
| F Seitelberger, H Lassmann, O Hornykiewic. Some mechanisms of brain edema studied in a kainic acid model. Acta neurobiologiae experimentalis. vol 50. issue 4-5. 1991-11-13. PMID:2130648. |
subcutaneous injection of ka into rats (10 mg/kg in saline 10 mg/ml; ph 7.0) induced longlasting status epilepticus followed by damage of cns tissue in the entorhinal/pyriform cortex and in the hippocampus. |
1991-11-13 |
2023-08-11 |
rat |
| b' E F Sperber, K Z Haas, P K Stanton, S L Mosh\\xc3\\xa. Resistance of the immature hippocampus to seizure-induced synaptic reorganization. Brain research. Developmental brain research. vol 60. issue 1. 1991-10-28. PMID:1717181.' |
in contrast to adult rats, these seizures did not produce ca3/ca4 neuronal loss, mossy fiber sprouting or changes in paired-pulse excitation or inhibition in the hippocampus of rat pups tested 2-4 weeks after status epilepticus. |
1991-10-28 |
2023-08-11 |
human |
| D Leifer, D G Cole, N W Kowal. Neuropathologic asymmetries in the brain of a patient with a unilateral status epilepticus. Journal of the neurological sciences. vol 103. issue 2. 1991-09-30. PMID:1715386. |
autopsy study of a patient who died after an episode of prolonged unilateral status epilepticus revealed neuronal loss in the hippocampus on the epileptic side, with gliosis confined to the ca1 and ca3 fields. |
1991-09-30 |
2023-08-11 |
human |
| K Yamada, M D Saltarelli, J T Coyl. [3H]hemicholinium-3 binding in rats with status epilepticus induced by lithium chloride and pilocarpine. European journal of pharmacology. vol 195. issue 3. 1991-09-17. PMID:1651249. |
the specific binding of [3h]hch-3 in cortex and hippocampus from rats with status epilepticus increased to 2- to 3-fold of control while the striatal [3h]hch-3 binding increased minimally. |
1991-09-17 |
2023-08-11 |
rat |
| E A Cavalheir. GAD-immunoreactive neurons are preserved in the hippocampus of rats with spontaneous recurrent seizures. Brazilian journal of medical and biological research = Revista brasileira de pesquisas medicas e biologicas. vol 23. issue 6-7. 1991-09-05. PMID:2129265. |
in the present study glutamate decarboxylase immunoreactivity (gad-ir) was used to quantify gabaergic neurons in the hippocampus of rats exhibiting spontaneous recurrent seizures following pilocarpine-induced status epilepticus. |
1991-09-05 |
2023-08-11 |
rat |
| M Lerner-Natoli, G Rondouin, M Belaidi, M Baldy-Moulinier, J M Kamenk. N-[1-(2-thienyl)cyclohexyl]-piperidine (TCP) does not block kainic acid-induced status epilepticus but reduces secondary hippocampal damage. Neuroscience letters. vol 122. issue 2. 1991-06-07. PMID:2027518. |
n-[1-(2-thienyl)cyclohexyl]-piperidine (tcp) does not block kainic acid-induced status epilepticus but reduces secondary hippocampal damage. |
1991-06-07 |
2023-08-11 |
rat |