| Publication |
Sentence |
Publish Date |
Extraction Date |
Species |
| C E Ribak, T Z Bara. Selective death of hippocampal CA3 pyramidal cells with mossy fiber afferents after CRH-induced status epilepticus in infant rats. Brain research. Developmental brain research. vol 91. issue 2. 1996-12-06. PMID:8852375. |
previous studies of crh-induced status epilepticus in infant rats demonstrated neuronal loss in several limbic structures, including the ca3 region of the hippocampus. |
1996-12-06 |
2023-08-12 |
rat |
| C E Ribak, T Z Bara. Selective death of hippocampal CA3 pyramidal cells with mossy fiber afferents after CRH-induced status epilepticus in infant rats. Brain research. Developmental brain research. vol 91. issue 2. 1996-12-06. PMID:8852375. |
thus, ca3 pyramidal neurons that are linked via mossy fibers to the tri-synaptic excitatory hippocampal circuit die subsequent to crh-induced status epilepticus. |
1996-12-06 |
2023-08-12 |
rat |
| A Pitkänen, J Tuunanen, T Halone. Vigabatrin and carbamazepine have different efficacies in the prevention of status epilepticus induced neuronal damage in the hippocampus and amygdala. Epilepsy research. vol 24. issue 1. 1996-12-05. PMID:8800633. |
vigabatrin and carbamazepine have different efficacies in the prevention of status epilepticus induced neuronal damage in the hippocampus and amygdala. |
1996-12-05 |
2023-08-12 |
rat |
| A Pitkänen, J Tuunanen, T Halone. Vigabatrin and carbamazepine have different efficacies in the prevention of status epilepticus induced neuronal damage in the hippocampus and amygdala. Epilepsy research. vol 24. issue 1. 1996-12-05. PMID:8800633. |
the present study compares the efficacy of carbamazepine (20 mg/kg/day) and vigabatrin (250 mg/kg/day) in preventing hippocampal and amygdaloid damage in the perforant pathway stimulation model of status epilepticus in the rat. |
1996-12-05 |
2023-08-12 |
rat |
| M G Naffah-Mazzacoratti, M I Bellíssimo, E A Cavalheir. Profile of prostaglandin levels in the rat hippocampus in pilocarpine model of epilepsy. Neurochemistry international. vol 27. issue 6. 1996-03-08. PMID:8574174. |
the results show, in hippocampus of rats, increase of pg f2 alpha and pg d2 during status epilepticus, increase of pg d2 during the silent period and increase of pg e2 and pg d2 during the chronic phase, when compared with control group. |
1996-03-08 |
2023-08-12 |
rat |
| J Kapur, D A Coulte. Experimental status epilepticus alters gamma-aminobutyric acid type A receptor function in CA1 pyramidal neurons. Annals of neurology. vol 38. issue 6. 1996-01-23. PMID:8526461. |
there is a reduction of gamma-aminobutyric acid (gaba)-mediated inhibition of the ca1 pyramidal region of the hippocampus during status epilepticus (se). |
1996-01-23 |
2023-08-12 |
rat |
| M Dragunow, K Preston, J Dodd, D Young, P Lawlor, D Christi. Clusterin accumulates in dying neurons following status epilepticus. Brain research. Molecular brain research. vol 32. issue 2. 1996-01-18. PMID:7500839. |
to further investigate the role of clusterin in nerve cell death its expression was measured in the rat brain at various times after status epilepticus (se) induced by 1 h of hippocampal stimulation, by using in situ hybridization, immunocytochemistry, and immunoblotting. |
1996-01-18 |
2023-08-12 |
rat |
| T Halonen, R Miettinen, A Toppinen, J Tuunanen, T Kotti, P J Riekkine. Vigabatrin protects against kainic acid-induced neuronal damage in the rat hippocampus. Neuroscience letters. vol 195. issue 1. 1995-12-18. PMID:7478243. |
we studied the neuroprotective effect of vigabatrin (gamma-vinyl gaba, vgb) in the rat hippocampus after status epilepticus (se) induced by kainic acid (ka). |
1995-12-18 |
2023-08-12 |
rat |
| D A Rempe, P S Mangan, E W Lothma. Regional heterogeneity of pathophysiological alterations in CA1 and dentate gyrus in a chronic model of temporal lobe epilepsy. Journal of neurophysiology. vol 74. issue 2. 1995-12-07. PMID:7472385. |
extracellular and intracellular recording techniques were employed in brain slice preparations to characterize responses of hippocampal tissue in the post-self sustaining limbic status epilepticus (post-sslse) model of chronic temporal lobe epilepsy (tle) as compared with responses in slices from control animals. |
1995-12-07 |
2023-08-12 |
Not clear |
| P S Mangan, D A Rempe, E W Lothma. Changes in inhibitory neurotransmission in the CA1 region and dentate gyrus in a chronic model of temporal lobe epilepsy. Journal of neurophysiology. vol 74. issue 2. 1995-12-07. PMID:7472386. |
extracellular and intracellular recordings were obtained in combined hippocampal-parahippocampal slices > or = 1 mo after a period of self-sustaining limbic status epilepticus (sslse) induced by continuous hippocampal stimulation. |
1995-12-07 |
2023-08-12 |
Not clear |
| E W Lothman, D A Rempe, P S Manga. Changes in excitatory neurotransmission in the CA1 region and dentate gyrus in a chronic model of temporal lobe epilepsy. Journal of neurophysiology. vol 74. issue 2. 1995-12-07. PMID:7472387. |
extracellular and intracellular recordings were obtained from in vitro hippocampal-parahippocampal slices > or = 1 mo after a period of self-sustaining limbic status epilepticus (sslse) induced by continuous hippocampal stimulation. |
1995-12-07 |
2023-08-12 |
Not clear |
| D Young, M Draguno. Neuronal injury following electrically induced status epilepticus with and without adenosine receptor antagonism. Experimental neurology. vol 133. issue 2. 1995-09-22. PMID:7649219. |
in the present study we compared neuronal death following status epilepticus (se) induced in the presence of 8-cyclopentyl-1,3-dimethylxanthine (8-cpt), an a1-adenosine receptor antagonist, with that following se induced by continuous hippocampal stimulation. |
1995-09-22 |
2023-08-12 |
Not clear |
| M B Harrison, M D Shumate, E W Lothma. Opioid peptide expression in models of chronic temporal lobe epilepsy. Neuroscience. vol 65. issue 3. 1995-08-17. PMID:7609877. |
using in situ hybridization, we examined the expression of prodynorphin and preproenkephalin messenger rna acutely following induction of kindling with recurrent seizures and in two models of chronic temporal lobe epilepsy: (i) rats fully kindled with rapidly recurring hippocampal seizures; and (ii) rats surviving after self-sustaining limbic status epilepticus induced with focal electrical stimulation of the hippocampus. |
1995-08-17 |
2023-08-12 |
rat |
| M M Okazaki, D A Evenson, J V Nadle. Hippocampal mossy fiber sprouting and synapse formation after status epilepticus in rats: visualization after retrograde transport of biocytin. The Journal of comparative neurology. vol 352. issue 4. 1995-05-25. PMID:7721998. |
hippocampal mossy fiber sprouting and synapse formation after status epilepticus in rats: visualization after retrograde transport of biocytin. |
1995-05-25 |
2023-08-12 |
rat |
| R Schmidt-Kastner, A Tomac, B Hoffer, S Bektesh, B Rosenzweig, L Olso. Glial cell-line derived neurotrophic factor (GDNF) mRNA upregulation in striatum and cortical areas after pilocarpine-induced status epilepticus in rats. Brain research. Molecular brain research. vol 26. issue 1-2. 1995-03-16. PMID:7854063. |
using limbic motor status epilepticus induced by pilocarpine to activate neurons in motor and limbic areas, we now demonstrate gdnf mrna signals in the striatum, hippocampus and cortex using in situ hybridisation. |
1995-03-16 |
2023-08-12 |
rat |
| R Schmidt-Kastner, A Tomac, B Hoffer, S Bektesh, B Rosenzweig, L Olso. Glial cell-line derived neurotrophic factor (GDNF) mRNA upregulation in striatum and cortical areas after pilocarpine-induced status epilepticus in rats. Brain research. Molecular brain research. vol 26. issue 1-2. 1995-03-16. PMID:7854063. |
increases in gdnf mrna after status epilepticus in hippocampus and neocortex indicate additional roles for gdnf. |
1995-03-16 |
2023-08-12 |
rat |
| S Shimosaka, Y T So, R P Simo. Deep prepiriform cortex modulates kainate-induced hippocampal injury. Neuroscience. vol 61. issue 4. 1995-03-02. PMID:7838380. |
status epilepticus induced these markers of neuronal injury in the ca1 and ca3a regions of the hippocampus, thalamus, piriform cortex and the amygdaloid complex. |
1995-03-02 |
2023-08-12 |
Not clear |
| E H Bertram, J F Cornet. The evolution of a rat model of chronic spontaneous limbic seizures. Brain research. vol 661. issue 1-2. 1995-03-01. PMID:7834366. |
the seizures begin following an episode of limbic status epilepticus induced by continuous electrical stimulation of the hippocampus, and they persist for more than a year (longest duration followed). |
1995-03-01 |
2023-08-12 |
rat |
| V Ridoux, A Valin, M Synguelakis, G Le Gal La Sall. Ex vivo culture of adult microglial cells from previously lesioned rat brains. Comptes rendus de l'Academie des sciences. Serie III, Sciences de la vie. vol 317. issue 3. 1995-01-19. PMID:7994611. |
in an attempt to study more precisely the glial cells involved in reactions following specific brain injuries, we tried to culture cells derived from surgically-lesioned rat brains or adult rat hippocampus previously treated with kainic acid, a convulsant which induces status epilepticus associated with structural modifications. |
1995-01-19 |
2023-08-12 |
rat |
| R D Tien, G J Felsber. The hippocampus in status epilepticus: demonstration of signal intensity and morphologic changes with sequential fast spin-echo MR imaging. Radiology. vol 194. issue 1. 1995-01-17. PMID:7997562. |
the hippocampus in status epilepticus: demonstration of signal intensity and morphologic changes with sequential fast spin-echo mr imaging. |
1995-01-17 |
2023-08-12 |
Not clear |