| Publication |
Sentence |
Publish Date |
Extraction Date |
Species |
| G W Mathern, G Price, C Rosales, J K Pretorius, A Lozada, D Mendoz. Anoxia during kainate status epilepticus shortens behavioral convulsions but generates hippocampal neuron loss and supragranular mossy fiber sprouting. Epilepsy research. vol 30. issue 2. 1998-07-13. PMID:9600545. |
in rats, this study determined the impact of systemic hypoxia during late kainate-induced status epilepticus on hippocampal neuron loss and mossy fiber sprouting. |
1998-07-13 |
2023-08-12 |
human |
| G W Mathern, G Price, C Rosales, J K Pretorius, A Lozada, D Mendoz. Anoxia during kainate status epilepticus shortens behavioral convulsions but generates hippocampal neuron loss and supragranular mossy fiber sprouting. Epilepsy research. vol 30. issue 2. 1998-07-13. PMID:9600545. |
these results indicate that rats exposed to kainate-induced status epilepticus for over 1 h and then a hypoxic insult had a shorter duration of convulsive status, decreased hippocampal neuron densities and greater fd mossy fiber sprouting than controls and the amount of neuronal damage and sprouting was slightly more than animals subjected to 6 h of kainate-induced status. |
1998-07-13 |
2023-08-12 |
human |
| L Giménez-Llort, E Martínez, L Camón, N de Ver. Long-term effects of status epilepticus induced by kainic acid on hippocampal polyamines. Neuroreport. vol 9. issue 5. 1998-07-01. PMID:9579694. |
long-term effects of status epilepticus induced by kainic acid on hippocampal polyamines. |
1998-07-01 |
2023-08-12 |
rat |
| L Giménez-Llort, E Martínez, L Camón, N de Ver. Long-term effects of status epilepticus induced by kainic acid on hippocampal polyamines. Neuroreport. vol 9. issue 5. 1998-07-01. PMID:9579694. |
in the present study we found that 2 and 3 weeks after status epilepticus induced by kainic acid, rats had increased concentrations of putrescine (3- and 1.7-fold, respectively) and spermidine (1.6- and 1.4-fold, respectively) in the hippocampus. |
1998-07-01 |
2023-08-12 |
rat |
| C Montécot, L Rondi-Reig, V Springhetti, J Seylaz, E Pinar. Inhibition of neuronal (type 1) nitric oxide synthase prevents hyperaemia and hippocampal lesions resulting from kainate-induced seizures. Neuroscience. vol 84. issue 3. 1998-07-01. PMID:9579784. |
(1) hippocampal nitric oxide synthase activity was measured under basal conditions, at 1 h after the onset of the status epilepticus and at 24 h after its termination (n = 4-6 per group). |
1998-07-01 |
2023-08-12 |
rat |
| C Montécot, L Rondi-Reig, V Springhetti, J Seylaz, E Pinar. Inhibition of neuronal (type 1) nitric oxide synthase prevents hyperaemia and hippocampal lesions resulting from kainate-induced seizures. Neuroscience. vol 84. issue 3. 1998-07-01. PMID:9579784. |
(3) hippocampal damage was revealed by cresyl violet staining and evaluated with a lesion score seven days after status epilepticus (n = 12 per group). |
1998-07-01 |
2023-08-12 |
rat |
| C Montécot, L Rondi-Reig, V Springhetti, J Seylaz, E Pinar. Inhibition of neuronal (type 1) nitric oxide synthase prevents hyperaemia and hippocampal lesions resulting from kainate-induced seizures. Neuroscience. vol 84. issue 3. 1998-07-01. PMID:9579784. |
hippocampal nitric oxide synthase activity was not significantly modified during status epilepticus or the following day in vehicle-treated rats. |
1998-07-01 |
2023-08-12 |
rat |
| C Montécot, L Rondi-Reig, V Springhetti, J Seylaz, E Pinar. Inhibition of neuronal (type 1) nitric oxide synthase prevents hyperaemia and hippocampal lesions resulting from kainate-induced seizures. Neuroscience. vol 84. issue 3. 1998-07-01. PMID:9579784. |
during status epilepticus, 7-nitroindazole significantly reduced the increase in hippocampal blood flow by 70% and prevented any increase in the tissue partial pressure of oxygen. |
1998-07-01 |
2023-08-12 |
rat |
| M Gruentha. Electroencephalographic and histological characteristics of a model of limbic status epilepticus permitting direct control over seizure duration. Epilepsy research. vol 29. issue 3. 1998-05-29. PMID:9551784. |
status epilepticus is induced by continuous, unilateral hippocampal stimulation. |
1998-05-29 |
2023-08-12 |
Not clear |
| D W Gross, L M Li, F Anderman. Catastrophic deterioration and hippocampal atrophy after childhood status epilepticus. Annals of neurology. vol 43. issue 5. 1998-05-21. PMID:9585371. |
catastrophic deterioration and hippocampal atrophy after childhood status epilepticus. |
1998-05-21 |
2023-08-12 |
Not clear |
| U C Wieshmann, F G Woermann, L Lemieux, S L Free, P A Bartlett, S J Smith, J S Duncan, J M Stevens, S D Shorvo. Development of hippocampal atrophy: a serial magnetic resonance imaging study in a patient who developed epilepsy after generalized status epilepticus. Epilepsia. vol 38. issue 11. 1998-05-18. PMID:9579926. |
development of hippocampal atrophy: a serial magnetic resonance imaging study in a patient who developed epilepsy after generalized status epilepticus. |
1998-05-18 |
2023-08-12 |
Not clear |
| I M Germano, E F Sperbe. Transplacentally induced neuronal migration disorders: an animal model for the study of the epilepsies. Journal of neuroscience research. vol 51. issue 4. 1998-05-11. PMID:9514201. |
in rats with severe nmd, kainic acid-induced status epilepticus produced hippocampal damage in the ca3/4 region. |
1998-05-11 |
2023-08-12 |
human |
| P S Mangan, E W Lothma. Profound disturbances of pre- and postsynaptic GABAB-receptor-mediated processes in region CA1 in a chronic model of temporal lobe epilepsy. Journal of neurophysiology. vol 76. issue 2. 1998-03-16. PMID:8871236. |
intracellular recordings were obtained in pyramidal cells from combined hippocampal/parahippocampal control slices and slices obtained > or = 1 mo after a period of self-sustaining limbic status epilepticus (sslse) induced by continuous hippocampal stimulation. |
1998-03-16 |
2023-08-12 |
Not clear |
| K Thompson, A M Holm, A Schousboe, P Popper, P Micevych, C Wasterlai. Hippocampal stimulation produces neuronal death in the immature brain. Neuroscience. vol 82. issue 2. 1998-03-11. PMID:9466446. |
these combined data demonstrate that the immature brain can incur damage as a result of prolonged seizure-like hippocampal activity mimicking status epilepticus in immature rats. |
1998-03-11 |
2023-08-12 |
rat |
| L K Friedman, E F Sperber, S L Moshé, M V Bennett, R S Zuki. Developmental regulation of glutamate and GABA(A) receptor gene expression in rat hippocampus following kainate-induced status epilepticus. Developmental neuroscience. vol 19. issue 6. 1998-03-05. PMID:9445090. |
developmental regulation of glutamate and gaba(a) receptor gene expression in rat hippocampus following kainate-induced status epilepticus. |
1998-03-05 |
2023-08-12 |
rat |
| L K Friedman, E F Sperber, S L Moshé, M V Bennett, R S Zuki. Developmental regulation of glutamate and GABA(A) receptor gene expression in rat hippocampus following kainate-induced status epilepticus. Developmental neuroscience. vol 19. issue 6. 1998-03-05. PMID:9445090. |
in rat pups resistant to kainate seizure-induced hippocampal neurodegeneration by silver impregnation, glutamate or gaba(a) alpha1-receptor mrnas were unaltered in ca3 neurons 24 h after status epilepticus. |
1998-03-05 |
2023-08-12 |
rat |
| A M Mazarati, C G Wasterlai. Blockers of NMDA receptor restore paired-pulse inhibition in the rat dentate gyrus lesioned by perforant path stimulation. Neuroscience letters. vol 234. issue 2-3. 1998-01-15. PMID:9364516. |
the ¿dormant basket cell' hypothesis postulates, that after status epilepticus, inhibitory interneurons in the hippocampus are deafferented from their excitatory inputs. |
1998-01-15 |
2023-08-12 |
rat |
| E M Aronica, J A Gorter, M C Paupard, S Y Grooms, M V Bennett, R S Zuki. Status epilepticus-induced alterations in metabotropic glutamate receptor expression in young and adult rats. The Journal of neuroscience : the official journal of the Society for Neuroscience. vol 17. issue 21. 1997-11-20. PMID:9334430. |
in the present study we used in situ hybridization to examine possible changes in mglur expression at the level of the hippocampus after status epilepticus in postnatal day 10 (p10) pup and adult (p40) rats. |
1997-11-20 |
2023-08-12 |
rat |
| D A Rempe, E H Bertram, J M Williamson, E W Lothma. Interneurons in area CA1 stratum radiatum and stratum oriens remain functionally connected to excitatory synaptic input in chronically epileptic animals. Journal of neurophysiology. vol 78. issue 3. 1997-11-06. PMID:9310439. |
rats were made chronically epileptic by a period of continuous electrical stimulation of the hippocampus, which establishes an acute condition of self-sustained limbic status epilepticus (sslse). |
1997-11-06 |
2023-08-12 |
human |
| T Kotti, T Halonen, J Sirviö, P Riekkinen, R Miettine. Comparison of NADPH diaphorase histochemistry, somatostatin immunohistochemistry, and silver impregnation in detecting structural and functional impairment in experimental status epilepticus. Neuroscience. vol 80. issue 1. 1997-10-23. PMID:9252225. |
in the present study we investigated whether the loss or survival of nicotinamide adenine dinucleotide phosphate (nadph) diaphorase-containing neurons, which are known to synthesize nitric oxide, would be an useful indicator for evaluating the structural and functional state of the rat hippocampus after status epilepticus that is induced by intraperitoneal injection of kainic acid. |
1997-10-23 |
2023-08-12 |
rat |