| Publication |
Sentence |
Publish Date |
Extraction Date |
Species |
| R S Jope, M Simonato, K Lall. Acetylcholine content in rat brain is elevated by status epilepticus induced by lithium and pilocarpine. Journal of neurochemistry. vol 49. issue 3. 1987-09-23. PMID:3612132. |
the concentration of acetylcholine in the cortex, hippocampus, and striatum decreased prior to the onset of spike activity or status epilepticus. |
1987-09-23 |
2023-08-11 |
rat |
| R S Jope, M Simonato, K Lall. Acetylcholine content in rat brain is elevated by status epilepticus induced by lithium and pilocarpine. Journal of neurochemistry. vol 49. issue 3. 1987-09-23. PMID:3612132. |
once status epilepticus began, the concentration of acetylcholine increased over time in the cortex and hippocampus, reaching peak levels that were 461% and 304% of control levels, respectively, after 2 h of seizures. |
1987-09-23 |
2023-08-11 |
rat |
| R S Jope, M Simonato, K Lall. Acetylcholine content in rat brain is elevated by status epilepticus induced by lithium and pilocarpine. Journal of neurochemistry. vol 49. issue 3. 1987-09-23. PMID:3612132. |
synaptosomes prepared from the cortex and from the hippocampus of rats with status epilepticus had elevated concentrations of acetylcholine: in the hippocampus the acetylcholine was principally in the cytoplasmic fraction, whereas in the cortex the acetylcholine was elevated in both the cytoplasmic and the vesicular fractions. |
1987-09-23 |
2023-08-11 |
rat |
| J P Vicedomini, J V Nadle. A model of status epilepticus based on electrical stimulation of hippocampal afferent pathways. Experimental neurology. vol 96. issue 3. 1987-07-02. PMID:3582552. |
a model of status epilepticus based on electrical stimulation of hippocampal afferent pathways. |
1987-07-02 |
2023-08-11 |
human |
| R K Yu, J A Holley, L J Macala, D D Spence. Ganglioside changes associated with temporal lobe epilepsy in the human hippocampus. The Yale journal of biology and medicine. vol 60. issue 2. 1987-05-27. PMID:3554791. |
to understand better the molecular and cellular events associated with status epilepticus, a multifaceted analysis has begun on hippocampal tissues therapeutically removed from patients with temporal lobe epilepsy. |
1987-05-27 |
2023-08-11 |
human |
| b' M Ingvar, J Folbegrova, B K Siesj\\xc3\\xb. Metabolic alterations underlying the development of hypermetabolic necrosis in the substantia nigra in status epilepticus. Journal of cerebral blood flow and metabolism : official journal of the International Society of Cerebral Blood Flow and Metabolism. vol 7. issue 1. 1987-03-16. PMID:3805159.' |
in the pag, the metabolite changes during status epilepticus were similar to those reported for cerebral cortex and hippocampus. |
1987-03-16 |
2023-08-11 |
rat |
| R S Slovite. Decreased hippocampal inhibition and a selective loss of interneurons in experimental epilepsy. Science (New York, N.Y.). vol 235. issue 4784. 1987-02-03. PMID:2879352. |
the occurrence of seizure activity in human temporal lobe epilepsy or status epilepticus is often associated with a characteristic pattern of cell loss in the hippocampus. |
1987-02-03 |
2023-08-11 |
human |
| D C McIntyre, K A Stokes, N Edso. Status epilepticus following stimulation of a kindled hippocampal focus in intact and commissurotomized rats. Experimental neurology. vol 94. issue 3. 1987-01-21. PMID:3780907. |
status epilepticus following stimulation of a kindled hippocampal focus in intact and commissurotomized rats. |
1987-01-21 |
2023-08-11 |
rat |
| D C McIntyre, K A Stokes, N Edso. Status epilepticus following stimulation of a kindled hippocampal focus in intact and commissurotomized rats. Experimental neurology. vol 94. issue 3. 1987-01-21. PMID:3780907. |
status epilepticus of either a nonconvulsive, partial, or generalized form was provoked in rats by 60 min of electrical stimulation of a kindled focus in the posterior-ventral hippocampus. |
1987-01-21 |
2023-08-11 |
rat |
| D C McIntyre, K A Stokes, N Edso. Status epilepticus following stimulation of a kindled hippocampal focus in intact and commissurotomized rats. Experimental neurology. vol 94. issue 3. 1987-01-21. PMID:3780907. |
following spontaneous offset of the nonconvulsive status epilepticus, minor pathology occurred which was largely restricted to the hippocampus, whereas partial or generalized status epilepticus produced considerable bilateral damage in the hippocampus, basolateral amygdala, and pyriform cortex. |
1987-01-21 |
2023-08-11 |
rat |
| D C McIntyre, K A Stokes, N Edso. Status epilepticus following stimulation of a kindled hippocampal focus in intact and commissurotomized rats. Experimental neurology. vol 94. issue 3. 1987-01-21. PMID:3780907. |
further, it was shown that the right hippocampus precipitated status epilepticus with a higher probability than the left hippocampus in both intact and split-brain rats. |
1987-01-21 |
2023-08-11 |
rat |
| J H Swan, B S Meldrum, R P Simo. Hyperglycemia does not augment neuronal damage in experimental status epilepticus. Neurology. vol 36. issue 10. 1986-11-03. PMID:3762942. |
we therefore studied the cytologic features and cerebral content of lactate and glucose in the selectively vulnerable neurons of rat hippocampus after 2 hours of l-allylglycine-induced status epilepticus in rats with mean plasma glucose concentrations of 65, 250, and 480 mg/100 ml. |
1986-11-03 |
2023-08-11 |
rat |
| L Turski, E A Cavalheiro, W A Turski, B S Meldru. Excitatory neurotransmission within substantia nigra pars reticulata regulates threshold for seizures produced by pilocarpine in rats: effects of intranigral 2-amino-7-phosphonoheptanoate and N-methyl-D-aspartate. Neuroscience. vol 18. issue 1. 1986-09-17. PMID:3016600. |
morphological examination of brains from rats treated with n-methyl-d-aspartate in the substantia nigra pars reticulata and subsequently given pilocarpine, 100 mg/kg, which underwent status epilepticus, reveals widespread damage to the amygdala, thalamus, olfactory cortex, substantia nigra, neocortex, and hippocampus. |
1986-09-17 |
2023-08-11 |
rat |
| M Ingva. Cerebral blood flow and metabolic rate during seizures. Relationship to epileptic brain damage. Annals of the New York Academy of Sciences. vol 462. 1986-06-11. PMID:3518570. |
after long periods of status epilepticus, selective neuronal necrosis is incurred in the neocortex (layer iii-iv), in the hippocampus (ca1 and ca4), and in the thalamus (vpl-vpm). |
1986-06-11 |
2023-08-11 |
rat |
| B K Siesjö, T Wieloc. Epileptic brain damage: pathophysiology and neurochemical pathology. Advances in neurology. vol 44. 1986-05-30. PMID:2871725. |
in well oxygenated animals with an adequate cardiovascular function, 2 hr of status epilepticus causes moderate neuronal necrosis in the cerebral cortex (layers 3-4), the hippocampus (ca4 and ca1 pyramidal cells), and the thalamus (ventromedial nuclei). |
1986-05-30 |
2023-08-11 |
rat |
| B S Meldru. Cell damage in epilepsy and the role of calcium in cytotoxicity. Advances in neurology. vol 44. 1986-05-30. PMID:3706026. |
status epilepticus may be followed by the loss of selectively vulnerable neurons in the hippocampus and neocortex. |
1986-05-30 |
2023-08-11 |
Not clear |
| A Lehmann, H Hagberg, I Jacobson, A Hamberge. Effects of status epilepticus on extracellular amino acids in the hippocampus. Brain research. vol 359. issue 1-2. 1986-01-24. PMID:3000520. |
effects of status epilepticus on extracellular amino acids in the hippocampus. |
1986-01-24 |
2023-08-11 |
Not clear |
| J C LaManna, S I Hari. Regional comparisons of brain glucose influx. Brain research. vol 326. issue 2. 1985-04-08. PMID:3971156. |
the higher ratio of glucose influx to glucose utilization in the cerebellum may explain the clinical and experimental findings of relative resistance of the cerebellum to hypoglycemia while the lower tmax in the hippocampus may be the mechanism underlying its selective vulnerability during pathophysiologic conditions associated with marked increments in brain oxidative metabolism, such as status epilepticus. |
1985-04-08 |
2023-08-11 |
rat |
| G Le Gal La Salle, K F Shen, S Feldblu. [Role of the hippocampus, amygdala and the substantia nigra in the evolution of status epilepticus induced by systemic injection of kainic acid in the rat]. Revue d'electroencephalographie et de neurophysiologie clinique. vol 14. issue 3. 1985-04-02. PMID:6528078. |
[role of the hippocampus, amygdala and the substantia nigra in the evolution of status epilepticus induced by systemic injection of kainic acid in the rat]. |
1985-04-02 |
2023-08-12 |
rat |
| M Kaijima, D Riche, S Rousseva, S Moyanova, S Dimov, G Le Gal La Sall. Electroencephalographic, behavioral, and histopathologic features of seizures induced by intra-amygdala application of folic acid in cats. Experimental neurology. vol 86. issue 2. 1984-12-05. PMID:6489501. |
in cases with status epilepticus, edema and neuronal degeneration was observed in the hippocampus, amygdala, thalamic nuclei of the midline, entorhinal cortex, and cerebellum. |
1984-12-05 |
2023-08-12 |
Not clear |