| Publication |
Sentence |
Publish Date |
Extraction Date |
Species |
| N N Koberskaia, L R Zenkov, V V Zakharov, I S Preobrazhenskai. [Cognitive potential P300 in dementia with Levy's bodies and Alzheimer's disease]. Zhurnal nevrologii i psikhiatrii imeni S.S. Korsakova. vol 105. issue 1. 2005-05-24. PMID:15704487. |
neurodegenerative diseases accompanied by cognitive impairment (dementia with levy's bodies--dlb and alzheimer's disease--ad) and others) are becoming more widespread due to increasing population life expectancy. |
2005-05-24 |
2023-08-12 |
Not clear |
| Melissa Ray, Iwo Bohr, J Michael McIntosh, Clive Ballard, Ian McKeith, Sylvie Chalon, Denis Guilloteau, Robert Perry, Elaine Perry, J A Court, Margaret Piggot. Involvement of alpha6/alpha3 neuronal nicotinic acetylcholine receptors in neuropsychiatric features of Dementia with Lewy bodies: [(125)I]-alpha-conotoxin MII binding in the thalamus and striatum. Neuroscience letters. vol 372. issue 3. 2005-01-05. PMID:15542244. |
dementia with lewy bodies (dlb) is a neurodegenerative disease associated with a range of neuropsychiatric symptoms and reduced expression of neuronal nicotinic acetylcholine receptors (nachrs) in neocortex, hippocampus, thalamus and basal ganglia. |
2005-01-05 |
2023-08-12 |
Not clear |
| Francesca Cormack, Alistair Gray, Clive Ballard, Martin J Tové. A failure of 'pop-out' in visual search tasks in dementia with Lewy Bodies as compared to Alzheimer's and Parkinson's disease. International journal of geriatric psychiatry. vol 19. issue 8. 2004-10-25. PMID:15290700. |
the pattern of neural damage in dementia with lewy bodies (dlb) led us to hypothesize that patients with dlb would be particularly impaired on parallel ('pop-out') search tasks, relative to serial search tasks, and that this would serve to distinguish dlb from other forms of neurodegenerative disease, particularly alzheimer's disease (ad). |
2004-10-25 |
2023-08-12 |
Not clear |
| Dominique Petit, Jean-François Gagnon, Maria Livia Fantini, Luigi Ferini-Strambi, Jacques Montplaisi. Sleep and quantitative EEG in neurodegenerative disorders. Journal of psychosomatic research. vol 56. issue 5. 2004-09-21. PMID:15172204. |
this paper reviews current knowledge on sleep problems, sleep architecture changes and quantitative eeg alteration brought on by various neurodegenerative diseases, such as alzheimer's disease (ad), progressive supranuclear palsy (psp), rem sleep behavior disorder (rbd), parkinson's disease (pd), dementia with lewy bodies (dlb), multiple system atrophy msa, huntington's disease and creutzfeldt-jakob disease, in comparison to normal aging. |
2004-09-21 |
2023-08-12 |
Not clear |
| Qiu-Lan Ma, Piu Chan, Mitsunobu Yoshii, Kenji Uéd. Alpha-synuclein aggregation and neurodegenerative diseases. Journal of Alzheimer's disease : JAD. vol 5. issue 2. 2003-09-16. PMID:12719631. |
later, the discovery of two missense mutations (g88c and g209a), which resulted in ala30pro (a30p) and ala53thr (a53t) substitutions, of the alpha-synuclein gene in certain autosomal-dominant early onset familial parkinson's disease (pd) has greatly promoted the understanding of the role of alpha-synuclein in the pathogenesis of neurodegenerative diseases, such as pd, dementia with lewy bodies (dlb) and multiple system atrophy (msa) [5,6,51,75]. |
2003-09-16 |
2023-08-12 |
human |
| Joshua P Fessel, Christine Hulette, Suzanne Powell, L Jackson Roberts, Jing Zhan. Isofurans, but not F2-isoprostanes, are increased in the substantia nigra of patients with Parkinson's disease and with dementia with Lewy body disease. Journal of neurochemistry. vol 85. issue 3. 2003-06-17. PMID:12694390. |
in this study, we attempted to compare the levels of f2-isops and isofs esterified in phospholipids in the substantia nigra (sn) from patients with pd to those of age-matched controls as well as patients with other neurodegenerative diseases, including dementia with lewy body disease (dlb), multiple system atrophy (msa), and alzheimer's disease (ad). |
2003-06-17 |
2023-08-12 |
Not clear |
| Larissa A Munishkina, Cassandra Phelan, Vladimir N Uversky, Anthony L Fin. Conformational behavior and aggregation of alpha-synuclein in organic solvents: modeling the effects of membranes. Biochemistry. vol 42. issue 9. 2003-06-16. PMID:12614167. |
intracellular proteinaceous inclusions (lewy bodies and lewy neurites) of alpha-synuclein are pathological hallmarks of neurodegenerative diseases such as parkinson's disease, dementia with lewy bodies (dlb), and multiple systemic atrophy. |
2003-06-16 |
2023-08-12 |
Not clear |
| K L Newell, B T Hyman, J H Growdon, E T Hedley-Whyt. Application of the National Institute on Aging (NIA)-Reagan Institute criteria for the neuropathological diagnosis of Alzheimer disease. Journal of neuropathology and experimental neurology. vol 58. issue 11. 1999-11-17. PMID:10560657. |
in studying several other neurodegenerative diseases, such as dlb, which shows neuropathological and clinical overlap with ad, the staging of neurofibrillary changes offered potential diagnostic refinement. |
1999-11-17 |
2023-08-12 |
human |
| H Arai, Y Morikawa, M Higuchi, T Matsui, C M Clark, M Miura, N Machida, V M Lee, J Q Trojanowski, H Sasak. Cerebrospinal fluid tau levels in neurodegenerative diseases with distinct tau-related pathology. Biochemical and biophysical research communications. vol 236. issue 2. 1997-08-28. PMID:9240421. |
our study suggests that tau may accumulate in the csf of patients with certain neurodegenerative diseases other than ad and that measurement of csf-tau may not distinguish ad from dlb. |
1997-08-28 |
2023-08-12 |
Not clear |