All Relations between Epilepsy and hippocampus

Publication Sentence Publish Date Extraction Date Species
Yao Ding, Shan Wang, Yan Jiang, Yi Yang, Manman Zhang, Yi Guo, Shuang Wang, Mei-ping Din. Fructose-1,6-diphosphate protects against epileptogenesis by modifying cation-chloride co-transporters in a model of amygdaloid-kindling temporal epilepticus. Brain research. vol 1539. 2014-06-20. PMID:24095797. in hippocampal neurons, cation-chloride co-transporters (cccs) are suggested to play interesting roles in epilepsy by modulating γ-aminobutyric acid (gaba)ergic activity through controlling gabaa receptor-mediated reversal potential. 2014-06-20 2023-08-12 human
Libai Yang, Xiaodong Cai, Jueqian Zhou, Shuda Chen, Yishu Chen, Ziyi Chen, Qian Wang, Ziyan Fang, Liemin Zho. STE20/SPS1-related proline/alanine-rich kinase is involved in plasticity of GABA signaling function in a mouse model of acquired epilepsy. PloS one. vol 8. issue 9. 2014-06-16. PMID:24058604. also, there are no data clearly characterizing spak expression in cortical or hippocampal neurons or confirming an association between spak and epilepsy. 2014-06-16 2023-08-12 mouse
Libai Yang, Xiaodong Cai, Jueqian Zhou, Shuda Chen, Yishu Chen, Ziyi Chen, Qian Wang, Ziyan Fang, Liemin Zho. STE20/SPS1-related proline/alanine-rich kinase is involved in plasticity of GABA signaling function in a mouse model of acquired epilepsy. PloS one. vol 8. issue 9. 2014-06-16. PMID:24058604. our data suggests that spak is involved in the plasticity of gaba signaling function in acquired epilepsy via adjustment of [cl(-)]i in hippocampal neurons. 2014-06-16 2023-08-12 mouse
Tanja Grimminger, Katharina Pernhorst, Rainer Surges, Pitt Niehusmann, Lutz Priebe, Marec von Lehe, Per Hoffmann, Sven Cichon, Susanne Schoch, Albert J Becke. Levetiracetam resistance: Synaptic signatures & corresponding promoter SNPs in epileptic hippocampi. Neurobiology of disease. vol 60. 2014-06-15. PMID:24018139. to this end, we have used our unique access to analyze hippocampal tissue from pharmacoresistant tle patients who underwent epilepsy surgery for seizure control (n = 53) stratified according to a priori lev responders versus patients with impaired lev-response. 2014-06-15 2023-08-12 Not clear
Elena L Paley, George Perry, Olga Sokolov. Tryptamine induces axonopathy and mitochondriopathy mimicking neurodegenerative diseases via tryptophanyl-tRNA deficiency. Current Alzheimer research. vol 10. issue 9. 2014-06-15. PMID:24117115. we identified axonal defects in hippocampus of tryptamine- treated mice similar to those observed in human brain of patients with alzheimer's disease, multiple sclerosis and epilepsy using anti-trprs site-directed antibodies. 2014-06-15 2023-08-12 mouse
E A Scholl, F E Dudek, J J Ekstran. Neuronal degeneration is observed in multiple regions outside the hippocampus after lithium pilocarpine-induced status epilepticus in the immature rat. Neuroscience. vol 252. 2014-06-11. PMID:23896573. the claim from several researchers using animal models of acquired epilepsy that the immature brain can develop epilepsy without evidence of hippocampal neuronal death raises the possibility that neuronal death in some of these other regions may also be important for epileptogenesis. 2014-06-11 2023-08-12 rat
E A Scholl, F E Dudek, J J Ekstran. Neuronal degeneration is observed in multiple regions outside the hippocampus after lithium pilocarpine-induced status epilepticus in the immature rat. Neuroscience. vol 252. 2014-06-11. PMID:23896573. the present study used the lithium pilocarpine model of acquired epilepsy in immature animals to assess which structures outside the hippocampus are injured acutely after status epilepticus. 2014-06-11 2023-08-12 rat
Brita Fritsch, Janine Reis, Maciej Gasior, Rafal M Kaminski, Michael A Rogawsk. Role of GluK1 kainate receptors in seizures, epileptic discharges, and epileptogenesis. The Journal of neuroscience : the official journal of the Society for Neuroscience. vol 34. issue 17. 2014-06-09. PMID:24760837. kainate receptors containing the gluk1 subunit have an impact on excitatory and inhibitory neurotransmission in brain regions, such as the amygdala and hippocampus, which are relevant to seizures and epilepsy. 2014-06-09 2023-08-13 mouse
Ardalan Aarabi, Bin H. Seizure prediction in hippocampal and neocortical epilepsy using a model-based approach. Clinical neurophysiology : official journal of the International Federation of Clinical Neurophysiology. vol 125. issue 5. 2014-06-05. PMID:24374087. seizure prediction in hippocampal and neocortical epilepsy using a model-based approach. 2014-06-05 2023-08-12 Not clear
Mahmood Motamedi, Ali Zandieh, Alireza Hajimirzabeigi, Majid Tahsini, Fatemeh Vakhshiteh, Elham Rahimia. Hippocampal body changes in pure partial onset sleep and pure partial onset waking epileptic patients. Neurological sciences : official journal of the Italian Neurological Society and of the Italian Society of Clinical Neurophysiology. vol 34. issue 9. 2014-06-02. PMID:23283529. the aim of the current study was to evaluate for the first time the hippocampal changes in patients with pure sleep and pure waking epilepsy. 2014-06-02 2023-08-12 human
Mahmood Motamedi, Ali Zandieh, Alireza Hajimirzabeigi, Majid Tahsini, Fatemeh Vakhshiteh, Elham Rahimia. Hippocampal body changes in pure partial onset sleep and pure partial onset waking epileptic patients. Neurological sciences : official journal of the Italian Neurological Society and of the Italian Society of Clinical Neurophysiology. vol 34. issue 9. 2014-06-02. PMID:23283529. hippocampal sclerosis and atrophy were found in nine (25.7 %) and seven (20.0 %) patients with pure sleep epilepsy, and in 12 (34.3 %) and 11 (31.4 %) patients with pure waking epilepsy, respectively (p > 0.05 for the comparison between sleep and waking epilepsy). 2014-06-02 2023-08-12 human
Mahmood Motamedi, Ali Zandieh, Alireza Hajimirzabeigi, Majid Tahsini, Fatemeh Vakhshiteh, Elham Rahimia. Hippocampal body changes in pure partial onset sleep and pure partial onset waking epileptic patients. Neurological sciences : official journal of the Italian Neurological Society and of the Italian Society of Clinical Neurophysiology. vol 34. issue 9. 2014-06-02. PMID:23283529. however, proportion of subjects with rounded hippocampal bodies (15, 42.9 % vs. 3, 8.6 % for patients with sleep and waking epilepsy, respectively) and rounding ratios of both left and right hippocampal bodies (0.66 ± 0.13 and 0.61 ± 0.12, respectively for left and right hippocampal bodies in sleep epileptic patients vs. 0.57 ± 0.11 and 0.55 ± 0.11, respectively for left and right hippocampal bodies in waking epileptic patients) were increased in patients with sleep epilepsy (p < 0.05). 2014-06-02 2023-08-12 human
Prasad S S V Vannemreddy, Andres M Kanner, Michel C Smith, Marvin Rossi, David Wallace, Siddharth N K Vannemreddy, Richard W Byrn. Chronic epilepsy due to low grade temporal lobe tumors and due to hippocampal sclerosis: do they differ in post-surgical outcome? Journal of neuro-oncology. vol 115. issue 2. 2014-05-22. PMID:23955571. chronic epilepsy due to low grade temporal lobe tumors and due to hippocampal sclerosis: do they differ in post-surgical outcome? 2014-05-22 2023-08-12 Not clear
Ana C Coan, Marcia E Morita, Brunno M Campos, Felipe P G Bergo, Bruno Y Kubota, Fernando Cende. Amygdala enlargement occurs in patients with mesial temporal lobe epilepsy and hippocampal sclerosis with early epilepsy onset. Epilepsy & behavior : E&B. vol 29. issue 2. 2014-05-22. PMID:24074891. amygdala enlargement occurs in patients with mesial temporal lobe epilepsy and hippocampal sclerosis with early epilepsy onset. 2014-05-22 2023-08-12 human
Daniel Kenney, Elaine Wirrel. Patient considerations in the management of focal seizures in children and adolescents. Adolescent health, medicine and therapeutics. vol 5. 2014-05-20. PMID:24808722. etiologies are diverse, and range from benign epilepsy syndromes with normal neuroimaging and almost certain remission to focal malformations of cortical development or hippocampal sclerosis with intractable seizures persisting lifelong. 2014-05-20 2023-08-13 Not clear
Leda L Talib, Kette D Valente, Silvia Vincentiis, Wagner F Gatta. Correlation between platelet and brain PLA(2) activity. Prostaglandins, leukotrienes, and essential fatty acids. vol 89. issue 4. 2014-05-19. PMID:23880350. we performed this task assessing pla2 activity in platelets and hippocampus collected simultaneously from 19 patients undergoing temporal lobectomy for treatment of refractory epilepsy. 2014-05-19 2023-08-12 Not clear
Shengjun Wang, Xue Yang, Youting Lin, Xiaoxue Qiu, Hui Li, Xiuhe Zhao, Lili Cao, Xuewu Liu, Yuejiu Pang, Xuping Wang, Zhaofu Ch. Cellular NAD depletion and decline of SIRT1 activity play critical roles in PARP-1-mediated acute epileptic neuronal death in vitro. Brain research. vol 1535. 2014-05-08. PMID:23994215. intense poly(adp-ribose) polymerase-1 (parp-1) activation was implicated as a major cause of caspase-independent cell death in the hippocampal neuronal culture (hnc) model of acute acquired epilepsy (ae). 2014-05-08 2023-08-12 Not clear
Graeme D Jackso. Epilepsy: Hippocampal sclerosis--are we speaking the same language? Nature reviews. Neurology. vol 9. issue 10. 2014-05-08. PMID:23999462. epilepsy: hippocampal sclerosis--are we speaking the same language? 2014-05-08 2023-08-12 Not clear
Graeme D Jackso. Epilepsy: Hippocampal sclerosis--are we speaking the same language? Nature reviews. Neurology. vol 9. issue 10. 2014-05-08. PMID:23999462. a task force of the international league against epilepsy has proposed a new pathology-based classification of three types of hippocampal sclerosis--the most frequent brain lesion in drug-resistant temporal lobe epilepsy. 2014-05-08 2023-08-12 Not clear
Esme Ekizoglu, Erdem Tuzun, Mark Woodhall, Bethan Lang, Leslie Jacobson, Sema Icoz, Nerses Bebek, Candan Gurses, Aysen Gokyigit, Patrick Waters, Angela Vincent, Betul Bayka. Investigation of neuronal autoantibodies in two different focal epilepsy syndromes. Epilepsia. vol 55. issue 3. 2014-05-05. PMID:24502404. our aim was to investigate the prevalence of these autoantibodies in patients with focal epilepsy of unknown cause (feouc) and in the group having mesial temporal lobe epilepsy with hippocampal sclerosis (mtle-hs). 2014-05-05 2023-08-12 Not clear