| Publication |
Sentence |
Publish Date |
Extraction Date |
Species |
| Roberto Cilia, Andrea Landi, Francesco Vergani, Erik Sganzerla, Gianni Pezzoli, Angelo Antonin. Extradural motor cortex stimulation in Parkinson's disease. Movement disorders : official journal of the Movement Disorder Society. vol 22. issue 1. 2007-04-05. PMID:17083104. |
outcome measures included changes in mean medication dosage (levodopa and dopamine agonists), unified parkinson's disease rating scale (updrs parts ii-iii and item 39), and dyskinesias (abnormal involuntary movements scale [aims]). |
2007-04-05 |
2023-08-12 |
Not clear |
| Y Naidu, K Ray Chaudhur. Transdermal rotigotine: a new non-ergot dopamine agonist for the treatment of Parkinson's disease. Expert opinion on drug delivery. vol 4. issue 2. 2007-04-03. PMID:17335409. |
studies in animal models and humans suggest that continuous dopaminergic stimulation could be achieved by the infusions of different dopamine agonists or levodopa, and may significantly reduce the risk of dyskinesias associated with treatment strategies utilising pulsatile treatment options. |
2007-04-03 |
2023-08-12 |
Not clear |
| Ralph Ankenman, Michael F Salvator. Low dose alpha-methyl-para-tyrosine (AMPT) in the treatment of dystonia and dyskinesia. The Journal of neuropsychiatry and clinical neurosciences. vol 19. issue 1. 2007-03-20. PMID:17308229. |
dystonias and dyskinesias seem to have their origin in inconsistent regulation of dopamine function in dopamine pathways. |
2007-03-20 |
2023-08-12 |
Not clear |
| F Sedel, M J Ribeiro, P Remy, N Blau, J-M Saudubray, Y Agi. Dihydropteridine reductase deficiency: levodopa's long-term effectiveness without dyskinesia. Neurology. vol 67. issue 12. 2007-01-16. PMID:17190955. |
despite the use of levodopa from age 3 months, he exhibited no dyskinesia or dopaminergic cell loss as suggested by normal pet imaging of the dopamine transporter. |
2007-01-16 |
2023-08-12 |
Not clear |
| Przemysław Nowak, Joanna Dabrowska, Aleksandra Bortel, Izabela Biedka, Grazyna Szczerbak, Grzegorz Słomian, Richard M Kostrzewa, Ryszard Bru. Histamine H3 receptor agonist- and antagonist-evoked vacuous chewing movements in 6-OHDA-lesioned rats occurs in an absence of change in microdialysate dopamine levels. European journal of pharmacology. vol 552. issue 1-3. 2007-01-04. PMID:17055481. |
the gradual induction of dopamine d(1) receptor supersensitivity is known as a priming phenomenon, and this process is thought to underlie not only the appearance of vacuous chewing movements in humans with tardive dyskinesia, but also the onset of motor dyskinesias in l-dihydroxyphenylalanine (l-dopa)-treated parkinson's disease patients. |
2007-01-04 |
2023-08-12 |
rat |
| Naomi P Visanji, Mark J Millan, Jonathan M Brotchi. Actions at sites other than D(3) receptors mediate the effects of BP897 on l-DOPA-induced hyperactivity in monoamine-depleted rats. Experimental neurology. vol 202. issue 1. 2006-12-20. PMID:16814282. |
attenuation of levodopa-induced dyskinesia by normalizing dopamine d(3) receptor function. |
2006-12-20 |
2023-08-12 |
rat |
| Brad J Kolls, Mark Stac. Apomorphine: a rapid rescue agent for the management of motor fluctuations in advanced Parkinson disease. Clinical neuropharmacology. vol 29. issue 5. 2006-11-14. PMID:16960475. |
in advance-stage disease, fluctuations in motor function, variability in response to dopaminergic therapy, and dyskinesias related to increasing doses of dopamine agonists and levodopa, present a variety of challenges to a managing physician. |
2006-11-14 |
2023-08-12 |
Not clear |
| b' J-C Corvol, J-A Girault, D Herv\\xc3\\xa. [Role and regulation of dopamine D1 receptors in the striatum: implications for the genesis of dyskinesia in Parkinson\'s disease]. Revue neurologique. vol 162. issue 6-7. 2006-11-08. PMID:16840977.' |
[role and regulation of dopamine d1 receptors in the striatum: implications for the genesis of dyskinesia in parkinson's disease]. |
2006-11-08 |
2023-08-12 |
Not clear |
| Judith A Strong, Arif Dalvi, Fredy J Revilla, Alok Sahay, Frederick J Samaha, Jeffrey A Welge, Jianhua Gong, Maureen Gartner, Xia Yue, Lei Y. Genotype and smoking history affect risk of levodopa-induced dyskinesias in Parkinson's disease. Movement disorders : official journal of the Movement Disorder Society. vol 21. issue 5. 2006-10-18. PMID:16435402. |
we determined whether particular dopamine and opioid receptor polymorphisms were associated with risk of earlier onset of dyskinesia side effects during levodopa therapy. |
2006-10-18 |
2023-08-12 |
human |
| Concepció Marin, Esther Aguilar, José A Obes. Coadministration of entacapone with levodopa attenuates the severity of dyskinesias in hemiparkinsonian rats. Movement disorders : official journal of the Movement Disorder Society. vol 21. issue 5. 2006-10-18. PMID:16437585. |
levodopa-induced dyskinesias (lids) have been associated with a sequence of events that includes pulsatile stimulation of dopamine receptors. |
2006-10-18 |
2023-08-12 |
rat |
| Concepció Marin, Esther Aguilar, José A Obes. Coadministration of entacapone with levodopa attenuates the severity of dyskinesias in hemiparkinsonian rats. Movement disorders : official journal of the Movement Disorder Society. vol 21. issue 5. 2006-10-18. PMID:16437585. |
long-acting agents producing continuous dopaminergic stimulation are less likely to prime for dyskinesia than short-acting drugs that produce pulsatile stimulation of dopamine receptors. |
2006-10-18 |
2023-08-12 |
rat |
| Jean-Jacques Soghomonia. L-DOPA-induced dyskinesia in adult rats with a unilateral 6-OHDA lesion of dopamine neurons is paralleled by increased c-fos gene expression in the subthalamic nucleus. The European journal of neuroscience. vol 23. issue 9. 2006-10-06. PMID:16706847. |
l-dopa-induced dyskinesia in adult rats with a unilateral 6-ohda lesion of dopamine neurons is paralleled by increased c-fos gene expression in the subthalamic nucleus. |
2006-10-06 |
2023-08-12 |
rat |
| Jean-Jacques Soghomonia. L-DOPA-induced dyskinesia in adult rats with a unilateral 6-OHDA lesion of dopamine neurons is paralleled by increased c-fos gene expression in the subthalamic nucleus. The European journal of neuroscience. vol 23. issue 9. 2006-10-06. PMID:16706847. |
the objective of this work was to study the effects of acute or chronic systemic administration of l-dopa to adult rats with a unilateral 6-hydroxydopamine (6-ohda) lesion of dopamine neurons on c-fos expression in the stn and test the hypothesis that these effects correlate with l-dopa-induced dyskinesias. |
2006-10-06 |
2023-08-12 |
rat |
| Wassilios Meissner, Paula Ravenscroft, René Reese, Daniel Harnack, Rudolf Morgenstern, Andreas Kupsch, Henrik Klitgaard, Bernard Bioulac, Christian E Gross, Erwan Bezard, Thomas Borau. Increased slow oscillatory activity in substantia nigra pars reticulata triggers abnormal involuntary movements in the 6-OHDA-lesioned rat in the presence of excessive extracellular striatal dopamine. Neurobiology of disease. vol 22. issue 3. 2006-08-24. PMID:16531050. |
since electrophysiological correlates of l-dopa-induced dyskinesia (lid) are almost unknown, changes of striatal dopamine (da) transmission and electrophysiological activity of the substantia nigra pars reticulata (snr) were recorded before and after acute l-dopa administration in sham-operated and 6-hydroxydopamine (6-ohda)-lesioned rats that were previously treated with vehicle or l-dopa for 10 days. |
2006-08-24 |
2023-08-12 |
rat |
| Lance A Smith, Michael J Jackson, Louisa Johnston, Mikko Kuoppamaki, Sarah Rose, Ghassan Al-Barghouthy, Susanna Del Signore, Peter Jenne. Switching from levodopa to the long-acting dopamine D2/D3 agonist piribedil reduces the expression of dyskinesia while maintaining effective motor activity in MPTP-treated primates. Clinical neuropharmacology. vol 29. issue 3. 2006-07-27. PMID:16772809. |
switching from levodopa to the long-acting dopamine d2/d3 agonist piribedil reduces the expression of dyskinesia while maintaining effective motor activity in mptp-treated primates. |
2006-07-27 |
2023-08-12 |
Not clear |
| E L Lane, C Winkler, P Brundin, M A Cenc. The impact of graft size on the development of dyskinesia following intrastriatal grafting of embryonic dopamine neurons in the rat. Neurobiology of disease. vol 22. issue 2. 2006-07-25. PMID:16406222. |
the impact of graft size on the development of dyskinesia following intrastriatal grafting of embryonic dopamine neurons in the rat. |
2006-07-25 |
2023-08-12 |
rat |
| Ernest Arena. Engineering a dopaminergic phenotype in stem/precursor cells: role of Nurr1, glia-derived signals, and Wnts. Annals of the New York Academy of Sciences. vol 1049. 2006-06-07. PMID:15965107. |
recent results from clinical trials using fetal tissue grafts in patients with parkinson's disease (pd) have indicated that current surgical strategies for dopamine cell replacement therapy need to be improved in order to achieve better functional integration of the grafts and to avoid dyskinesias. |
2006-06-07 |
2023-08-12 |
Not clear |
| Krystof S Bankiewicz, Marcel Daadi, Philip Pivirotto, John Bringas, Laura Sanftner, Janet Cunningham, John R Forsayeth, Jamie L Eberlin. Focal striatal dopamine may potentiate dyskinesias in parkinsonian monkeys. Experimental neurology. vol 197. issue 2. 2006-04-03. PMID:16337943. |
focal striatal dopamine may potentiate dyskinesias in parkinsonian monkeys. |
2006-04-03 |
2023-08-12 |
monkey |
| Eleonora Maries, Jeffrey H Kordower, Yaping Chu, Timothy J Collier, Caryl E Sortwell, Eliza Olaru, Kathleen Shannon, Kathy Steece-Collie. Focal not widespread grafts induce novel dyskinetic behavior in parkinsonian rats. Neurobiology of disease. vol 21. issue 1. 2006-03-09. PMID:16095907. |
in the current study, we employed parkinsonian rats to test whether the distribution of dopamine neuron grafts could differentially alter striatal circuitry and levodopa-induced dyskinesias. |
2006-03-09 |
2023-08-12 |
rat |
| G Ransmay. [The role of dopaminagonists in the treatment of Parkinson's disease]. Praxis. vol 94. issue 42. 2006-03-07. PMID:16277084. |
initial treatment with dopamine agonists has been found to postpone or prevent from motor fluctuations and dyskinesias. |
2006-03-07 |
2023-08-12 |
human |