| Publication |
Sentence |
Publish Date |
Extraction Date |
Species |
| Ilaria Martinelli, Andrea Ghezzi, Elisabetta Zucchi, Giulia Gianferrari, Laura Ferri, Cristina Moglia, Umberto Manera, Luca Solero, Rosario Vasta, Antonio Canosa, Maurizio Grassano, Maura Brunetti, Letizia Mazzini, Fabiola De Marchi, Cecilia Simonini, Nicola Fini, Marco Vinceti, Marcello Pinti, Adriano Chi\\xc3\\xb2, Andrea Calvo, Jessica Mandriol. Predictors for progression in amyotrophic lateral sclerosis associated to SOD1 mutation: insight from two population-based registries. Journal of neurology. 2023-09-05. PMID:37668704. |
predictors for progression in amyotrophic lateral sclerosis associated to sod1 mutation: insight from two population-based registries. |
2023-09-05 |
2023-09-07 |
Not clear |
| Ilaria Martinelli, Andrea Ghezzi, Elisabetta Zucchi, Giulia Gianferrari, Laura Ferri, Cristina Moglia, Umberto Manera, Luca Solero, Rosario Vasta, Antonio Canosa, Maurizio Grassano, Maura Brunetti, Letizia Mazzini, Fabiola De Marchi, Cecilia Simonini, Nicola Fini, Marco Vinceti, Marcello Pinti, Adriano Chi\\xc3\\xb2, Andrea Calvo, Jessica Mandriol. Predictors for progression in amyotrophic lateral sclerosis associated to SOD1 mutation: insight from two population-based registries. Journal of neurology. 2023-09-05. PMID:37668704. |
uncovering distinct features and trajectories of amyotrophic lateral sclerosis (als) associated with sod1 mutations (sod1-als) can provide valuable insights for patient' counseling and stratification for trials, and interventions timing. |
2023-09-05 |
2023-09-07 |
Not clear |
| Akhtar Atiya, Abdullatif Bin Muhsinah, Mohammed Alrouji, Fahad Alhumaydhi, Waleed Al Abdulmonem, Mohammad Abdullah Aljasir, Sharaf E Sharaf, Mohammad Furkan, Rizwan Hasan Khan, Moyad Shahwan, Anas Shams. Unveiling promising inhibitors of superoxide dismutase 1 (SOD1) for therapeutic interventions. International journal of biological macromolecules. 2023-09-04. PMID:37666395. |
any dysregulation of sod1 activity is linked with cancer pathogenesis and neurodegenerative disorders, such as amyotrophic lateral sclerosis (als). |
2023-09-04 |
2023-09-07 |
Not clear |
| Lisa Morichon, Christophe Hirtz, Laurent Tiers, Alexandre Mezghrani, C\\xc3\\xa9dric Raoul, Florence Esselin, Elisa De La Cruz, Jean-Pierre Julien, William Camu, Sylvain Lehman. Ultrasensitive digital immunoassays for SOD1 conformation in amyotrophic lateral sclerosis. Bioanalysis. 2023-08-31. PMID:37650499. |
ultrasensitive digital immunoassays for sod1 conformation in amyotrophic lateral sclerosis. |
2023-08-31 |
2023-09-07 |
Not clear |
| Soledad Marton, Ernesto Miquel, Joaqu\\xc3\\xadn Acosta-Rodr\\xc3\\xadguez, Santiago Fontenla, Gabriela Libisch, Patricia Cassin. SOD1 ASN neuro. vol 15. 2023-08-30. PMID:37644868. |
sod1 amyotrophic lateral sclerosis (als) is a fatal neurodegenerative disease characterized by upper and lower motor neuron (mn) degeneration. |
2023-08-30 |
2023-09-07 |
Not clear |
| Shilpa Sharma, Vijay Raj Tomar, Shashank Dee. Mechanism of the interaction of toxic SOD1 fibrils with two potent polyphenols: curcumin and quercetin. Physical chemistry chemical physics : PCCP. 2023-08-21. PMID:37602388. |
amyotrophic lateral sclerosis (als) is a debilitating neurodegenerative disease commonly caused due to the aggregation of superoxide dismutase 1 (sod1) protein. |
2023-08-21 |
2023-09-07 |
Not clear |
| Jos\\xc3\\xa9 R Monteiro Neto, Gabriela D Ribeiro, Rayne S S Magalh\\xc3\\xa3es, Cristian Follmer, Tiago F Outeiro, Elis C A Eleutheri. Glycation modulates superoxide dismutase 1 aggregation and toxicity in models of sporadic amyotrophic lateral sclerosis. Biochimica et biophysica acta. Molecular basis of disease. 2023-08-09. PMID:37558009. |
different sod1 proteoforms are implicated## in both familial and sporadic cases of amyotrophic lateral sclerosis (als), an aging-associated disease that affects motor neurons. |
2023-08-09 |
2023-08-16 |
human |
| Shamchal Bakavayev, Alexandra Stavsky, Shirel Argueti-Ostrovsky, Galit Yehezkel, Yael Fridmann-Sirkis, Zeev Barak, Daniel Gitler, Adrian Israelson, Stanislav Enge. Blocking an epitope of misfolded SOD1 ameliorates disease phenotype in a model of amyotrophic lateral sclerosis. Brain : a journal of neurology. 2023-07-03. PMID:37394908. |
blocking an epitope of misfolded sod1 ameliorates disease phenotype in a model of amyotrophic lateral sclerosis. |
2023-07-03 |
2023-08-14 |
mouse |
| Biying Yang, Jingrui Pan, Xiao-Ni Zhang, Hongxuan Wang, Lei He, Xiaoming Rong, Xiangpen Li, Ying Pen. NRF2 activation suppresses motor neuron ferroptosis induced by the SOD1 Neurobiology of disease. 2023-06-23. PMID:37352984. |
nrf2 activation suppresses motor neuron ferroptosis induced by the sod1 the progressive neurodegenerative disease amyotrophic lateral sclerosis (als) is caused by a decline in motor neuron function, resulting in worsened motor impairments, malnutrition, respiratory failure and mortality, and there is a lack of effective clinical treatments. |
2023-06-23 |
2023-08-14 |
Not clear |
| Chengyou Zheng, Weifen Li, Tahir Ali, Ziting Peng, Jieli Liu, Zhengying Pan, Jinxing Feng, Shupeng L. Ibrutinib Delays ALS Installation and Increases Survival of SOD1 Journal of neuroimmune pharmacology : the official journal of the Society on NeuroImmune Pharmacology. 2023-06-16. PMID:37326908. |
ibrutinib delays als installation and increases survival of sod1 amyotrophic lateral sclerosis (als) is a fatal multisystem degenerative disorder with minimal available therapeutic. |
2023-06-16 |
2023-08-14 |
Not clear |
| Shilpa Sharma, Vijay Raj Tomar, Shashank Dee. Myricetin: A Potent Anti-Amyloidogenic Polyphenol against Superoxide Dismutase 1 Aggregation. ACS chemical neuroscience. 2023-06-14. PMID:37314311. |
amyotrophic lateral sclerosis (als) is believed to be caused by the aggregation of misfolded or mutated superoxide dismutase 1 (sod1). |
2023-06-14 |
2023-08-14 |
Not clear |
| Stefania Santarelli, Chiara Londero, Alessia Soldano, Carlotta Candelaresi, Leonardo Todeschini, Luisa Vernizzi, Paola Bellost. Frontiers in neuroscience. vol 17. 2023-06-05. PMID:37274187. |
indeed, despite the physiological and morphological differences between the fly and the human brain, most of the biochemical and molecular aspects regulating protein homeostasis, including autophagy, are conserved between the two species.in this review, we will provide an overview of the most common neurodegenerative proteinopathies, which include polyq diseases (huntington's disease, spinocerebellar ataxia 1, 2, and 3), amyotrophic lateral sclerosis (c9orf72, sod1, tdp-43, fus), alzheimer's disease (app, tau) parkinson's disease (a-syn, parkin and pink1, lrrk2) and prion diseases, highlighting the studies using drosophila that have contributed to understanding the conserved mechanisms and elucidating the role of autophagy in these diseases. |
2023-06-05 |
2023-08-14 |
human |
| Snehal Aouti, Sivaraman Padavattan, Balasundaram Padmanabha. Structure-based discovery of an antipsychotic drug, paliperidone, as a modulator of human superoxide dismutase 1: a potential therapeutic target in amyotrophic lateral sclerosis. Acta crystallographica. Section D, Structural biology. 2023-05-19. PMID:37204819. |
aggregates of the antioxidant superoxide dismutase 1 (sod1) are one of the major contributors to the pathogenesis of amyotrophic lateral sclerosis (als). |
2023-05-19 |
2023-08-14 |
human |
| Akira Hosomi, Chinatsu Okachi, Yudai Fujiwar. Human SOD1 is secreted via a conventional secretion pathway in Saccharomyces cerevisiae. Biochemical and biophysical research communications. vol 666. 2023-05-14. PMID:37182284. |
sod1 is a causative gene of amyotrophic lateral sclerosis (als). |
2023-05-14 |
2023-08-14 |
human |
| Cindy V Ly, Margaret D Ireland, Wade K Self, James Bollinger, Jennifer Jockel-Balsarotti, Hillary Herzog, Peggy Allred, Leah Miller, Michael Doyle, Isabel Anez-Bruzual, Bhavesh Trikamji, Ted Hyman, Tyler Kung, Katherine Nicholson, Robert C Bucelli, Bruce W Patterson, Randall J Bateman, Timothy M Mille. Protein kinetics of superoxide dismutase-1 in familial and sporadic amyotrophic lateral sclerosis. Annals of clinical and translational neurology. 2023-04-29. PMID:37119480. |
accumulation of misfolded superoxide dismutase-1 (sod1) is a pathological hallmark of sod1-related amyotrophic lateral sclerosis (als) and is observed in sporadic als where its role in pathogenesis is controversial. |
2023-04-29 |
2023-08-14 |
Not clear |
| Yoshihiro Kitaoka, Soju Seki, Sou Kawata, Akira Nishiura, Kohei Kawamura, Shin-Ichiro Hiraoka, Mikihiko Kogo, Susumu Tanak. Analysis of Feeding Behavior Characteristics in the Cu/Zn Superoxide Dismutase 1 (SOD1) SOD1G93A Mice Model for Amyotrophic Lateral Sclerosis (ALS). Nutrients. vol 15. issue 7. 2023-04-13. PMID:37049492. |
analysis of feeding behavior characteristics in the cu/zn superoxide dismutase 1 (sod1) sod1g93a mice model for amyotrophic lateral sclerosis (als). |
2023-04-13 |
2023-08-14 |
mouse |
| Takayuki Kondo, Ikuyo Inoue, Kazuhiro Umeyama, Masahito Watanabe, Hitomi Matsunari, Ayuko Uchikura, Kazuaki Nakano, Kayoko Tsukita, Keiko Imamura, Hiroshi Nagashima, Haruhisa Inou. A Transgenic Pig Model With Human Mutant SOD1 Exhibits the Early Pathology of Amyotrophic Lateral Sclerosis. Laboratory investigation; a journal of technical methods and pathology. vol 103. issue 2. 2023-04-11. PMID:37039150. |
a transgenic pig model with human mutant sod1 exhibits the early pathology of amyotrophic lateral sclerosis. |
2023-04-11 |
2023-08-14 |
human |
| Ao Yun Zhang, Chad M Dashnaw, Mayte Gonzalez, Jordan C Koone, Nick A Wells, Colin A Smith, Matthew J Guberman-Pfeffer, Bryan F Sha. Oxidation of Dueling Cysteine Promotes Subunit Exchange in SOD1. ACS chemical neuroscience. 2023-04-06. PMID:37023050. |
the heterodimerization of wild-type (wt) cu, zn superoxide dismutase-1 (sod1) and mutant sod1 might be a critical step in the pathogenesis of sod1-linked amyotrophic lateral sclerosis (als). |
2023-04-06 |
2023-08-14 |
Not clear |
| Katherine L Marshall, Labchan Rajbhandari, Arun Venkatesan, Nicholas J Maragakis, Mohamed H Fara. Enhanced axonal regeneration of ALS patient iPSC-derived motor neurons harboring SOD1 Scientific reports. vol 13. issue 1. 2023-04-05. PMID:37020097. |
enhanced axonal regeneration of als patient ipsc-derived motor neurons harboring sod1 amyotrophic lateral sclerosis (als) is a devastating neurodegenerative disease, characterized by degeneration of upper and lower motor neurons that leads to muscle weakness, paralysis, and death, but the effects of disease-causing mutations on axonal outgrowth of neurons derived from human induced pluripotent stem cells (ipsc)-derived motor neurons (hipsc-mn) are poorly understood. |
2023-04-05 |
2023-08-14 |
human |
| Xiaojiao Xu, Qiu Yang, Zheyi Liu, Rong Zhang, Hang Yu, Manli Wang, Sheng Chen, Guowang Xu, Yaping Shao, Weidong L. Integrative analysis of metabolomics and proteomics unravels purine metabolism disorder in the SOD1 Neurobiology of disease. 2023-03-31. PMID:37001614. |
integrative analysis of metabolomics and proteomics unravels purine metabolism disorder in the sod1 amyotrophic lateral sclerosis (als) is a fatal neurodegenerative disease with progressive paralysis of limbs and bulb in patients, the cause of which remains unclear. |
2023-03-31 |
2023-08-14 |
Not clear |