| Publication |
Sentence |
Publish Date |
Extraction Date |
Species |
| B S Shankaranarayana Rao, T R Raju, B L Met. Self-stimulation of lateral hypothalamus and ventral tegmentum increases the levels of noradrenaline, dopamine, glutamate, and AChE activity, but not 5-hydroxytryptamine and GABA levels in hippocampus and motor cortex. Neurochemical research. vol 23. issue 8. 1999-05-21. PMID:9704594. |
self-stimulation of lateral hypothalamus and ventral tegmentum increases the levels of noradrenaline, dopamine, glutamate, and ache activity, but not 5-hydroxytryptamine and gaba levels in hippocampus and motor cortex. |
1999-05-21 |
2023-08-12 |
rat |
| B S Shankaranarayana Rao, T R Raju, B L Met. Self-stimulation of lateral hypothalamus and ventral tegmentum increases the levels of noradrenaline, dopamine, glutamate, and AChE activity, but not 5-hydroxytryptamine and GABA levels in hippocampus and motor cortex. Neurochemical research. vol 23. issue 8. 1999-05-21. PMID:9704594. |
self-stimulation experience resulted in a significant (p < 0.001) increase in the levels of noradrenaline, dopamine, glutamate and ache activity but not 5-hydroxytryptamine and gaba levels in hippocampus and motor cortex. |
1999-05-21 |
2023-08-12 |
rat |
| V Derkach, A Barria, T R Soderlin. Ca2+/calmodulin-kinase II enhances channel conductance of alpha-amino-3-hydroxy-5-methyl-4-isoxazolepropionate type glutamate receptors. Proceedings of the National Academy of Sciences of the United States of America. vol 96. issue 6. 1999-05-20. PMID:10077673. |
ltp in the ca1 field of the hippocampus requires activation of ca2+/calmodulin-kinase ii (cam-kii), which phosphorylates ser-831 in the glur1 subunit of the alpha-amino-3-hydroxy-5-methyl-4-isoxazolepropionate glutamate receptor (ampa-r), and this activation/phosphorylation is thought to be a postsynaptic mechanism in ltp. |
1999-05-20 |
2023-08-12 |
Not clear |
| M E Gilbert, C M Mack, S M Lasle. Chronic developmental lead exposure and hippocampal long-term potentiation: biphasic dose-response relationship. Neurotoxicology. vol 20. issue 1. 1999-05-19. PMID:10091860. |
we have observed a similar profile in hippocampal glutamate release employing a similar range of exposure levels, i.e., reduction of glutamate release that is absent at higher concentrations of pb in the drinking water (lasley et al., 1998). |
1999-05-19 |
2023-08-12 |
rat |
| H Katsumori, R A Baldwin, C G Wasterlai. Reverse transport of glutamate during depolarization in immature hippocampal slices. Brain research. vol 819. issue 1-2. 1999-05-17. PMID:10082873. |
reverse transport of glutamate during depolarization in immature hippocampal slices. |
1999-05-17 |
2023-08-12 |
rat |
| H Katsumori, R A Baldwin, C G Wasterlai. Reverse transport of glutamate during depolarization in immature hippocampal slices. Brain research. vol 819. issue 1-2. 1999-05-17. PMID:10082873. |
we studied the source of extracellular glutamate released by hippocampal slices obtained from p14 or adult rats, during 50 mm k+ depolarization by using two potent inhibitors of na+-dependent glutamate transport: l-trans-pyrrolidine-2,4-dicarboxylate (pdc), which is a relatively non-selective inhibitor of various glutamate transporter subtypes and dihydrokainic acid (dhk), a specific inhibitor of the glial transporter, glt-1. |
1999-05-17 |
2023-08-12 |
rat |
| H Katsumori, R A Baldwin, C G Wasterlai. Reverse transport of glutamate during depolarization in immature hippocampal slices. Brain research. vol 819. issue 1-2. 1999-05-17. PMID:10082873. |
these data suggest that most depolarization-induced glutamate release in immature hippocampal slices is due to reversal of transport through a pdc-sensitive na+-dependent glutamate transporter, presumably acting on presynaptic or cytoplasmic neuronal pools, and is not due to exocytosis from vesicular pools. |
1999-05-17 |
2023-08-12 |
rat |
| M K Shelton, K D McCarth. Mature hippocampal astrocytes exhibit functional metabotropic and ionotropic glutamate receptors in situ. Glia. vol 26. issue 1. 1999-05-13. PMID:10088667. |
mature hippocampal astrocytes exhibit functional metabotropic and ionotropic glutamate receptors in situ. |
1999-05-13 |
2023-08-12 |
Not clear |
| M K Shelton, K D McCarth. Mature hippocampal astrocytes exhibit functional metabotropic and ionotropic glutamate receptors in situ. Glia. vol 26. issue 1. 1999-05-13. PMID:10088667. |
in previous studies, neither metabotropic nor ionotropic glutamate receptor-mediated responses were detected by imaging ca2+ in astrocytes from mature (p21-p42) animals, suggesting astrocyte glutamate receptors only contribute to hippocampus physiology during development. |
1999-05-13 |
2023-08-12 |
Not clear |
| M K Shelton, K D McCarth. Mature hippocampal astrocytes exhibit functional metabotropic and ionotropic glutamate receptors in situ. Glia. vol 26. issue 1. 1999-05-13. PMID:10088667. |
for this study, we imaged astrocytes in p31-p38 hippocampal slices to determine if metabotropic and ionotropic glutamate receptor activation elevates intracellular calcium in mature astrocytes. |
1999-05-13 |
2023-08-12 |
Not clear |
| M K Shelton, K D McCarth. Mature hippocampal astrocytes exhibit functional metabotropic and ionotropic glutamate receptors in situ. Glia. vol 26. issue 1. 1999-05-13. PMID:10088667. |
these results demonstrate that astrocytes in mature hippocampus have functional ionotropic and metabotropic glutamate receptors that regulate astrocytic calcium levels. |
1999-05-13 |
2023-08-12 |
Not clear |
| Z C Ye, H Sontheime. Metabotropic glutamate receptor agonists reduce glutamate release from cultured astrocytes. Glia. vol 25. issue 3. 1999-05-12. PMID:9932873. |
we investigated the effects of metabotropic glutamate receptor (mglur) agonists on glutamate transport and [glu]o in primary hippocampal astrocytic cultures. |
1999-05-12 |
2023-08-12 |
rat |
| G J Brewe. Age-related toxicity to lactate, glutamate, and beta-amyloid in cultured adult neurons. Neurobiology of aging. vol 19. issue 6. 1999-05-11. PMID:10192215. |
neurons were isolated from embryonic, young- and old-aged rat hippocampus, cultured in serum-free medium and exposed to lactic acid, glutamate or beta-amyloid. |
1999-05-11 |
2023-08-12 |
rat |
| K Inoue, S Koizumi, S Ueno, M Tsud. [Signal transduction through ATP receptors in brain and the development of new drugs]. Nihon yakurigaku zasshi. Folia pharmacologica Japonica. vol 112 Suppl 1. 1999-05-10. PMID:10190130. |
in the paper we presented the data that atp inhibits the glutamate release in cultured hippocampal neurons. |
1999-05-10 |
2023-08-12 |
Not clear |
| K Inoue, S Koizumi, S Ueno, M Tsud. [Signal transduction through ATP receptors in brain and the development of new drugs]. Nihon yakurigaku zasshi. Folia pharmacologica Japonica. vol 112 Suppl 1. 1999-05-10. PMID:10190130. |
this and the report revealing that atp protected against cell death by glutamate suggest that atp may be playing a role in the protection of the hippocampus from over-stimulation. |
1999-05-10 |
2023-08-12 |
Not clear |
| S U Devaskar, P A Rajakumar, R B Mink, R A McKnight, S Thamotharan, S J Hick. Effect of development and hypoxic-ischemia upon rabbit brain glucose transporter expression. Brain research. vol 823. issue 1-2. 1999-05-07. PMID:10095018. |
further, while hypoxic-ischemia did not significantly affect brain glut 1 mrna and protein, it altered glut 3 mrna levels in a region-specific manner, with a three-fold increase in the cerebral cortex, a two-fold increase in the hippocampus, and a 50% increase in the caudate nucleus (p<0.05). |
1999-05-07 |
2023-08-12 |
mouse |
| Y Zhang, P Lipto. Cytosolic Ca2+ changes during in vitro ischemia in rat hippocampal slices: major roles for glutamate and Na+-dependent Ca2+ release from mitochondria. The Journal of neuroscience : the official journal of the Society for Neuroscience. vol 19. issue 9. 1999-05-07. PMID:10212290. |
cytosolic ca2+ changes during in vitro ischemia in rat hippocampal slices: major roles for glutamate and na+-dependent ca2+ release from mitochondria. |
1999-05-07 |
2023-08-12 |
rat |
| S M Sequeira, A P Carvalho, C M Carvalh. Both protein kinase G dependent and independent mechanisms are involved in the modulation of glutamate release by nitric oxide in rat hippocampal nerve terminals. Neuroscience letters. vol 261. issue 1-2. 1999-05-06. PMID:10081919. |
both protein kinase g dependent and independent mechanisms are involved in the modulation of glutamate release by nitric oxide in rat hippocampal nerve terminals. |
1999-05-06 |
2023-08-12 |
rat |
| S M Sequeira, A P Carvalho, C M Carvalh. Both protein kinase G dependent and independent mechanisms are involved in the modulation of glutamate release by nitric oxide in rat hippocampal nerve terminals. Neuroscience letters. vol 261. issue 1-2. 1999-05-06. PMID:10081919. |
we compared the effects of sodium nitroprusside (snp), and of 8-bromo guanosine 3',5'-cyclic monophosphate (8-brcgmp), on the 4-aminopyridine (4-ap)-evoked ca2+-dependent release of glutamate from hippocampal nerve terminals and further investigated the role of protein kinase g (pkg) in this mechanism. |
1999-05-06 |
2023-08-12 |
rat |
| M Hüll, J Eistetter, B L Fiebich, J Baue. Glutamate but not interleukin-6 influences the phosphorylation of tau in primary rat hippocampal neurons. Neuroscience letters. vol 261. issue 1-2. 1999-05-06. PMID:10081920. |
glutamate but not interleukin-6 influences the phosphorylation of tau in primary rat hippocampal neurons. |
1999-05-06 |
2023-08-12 |
rat |