| Publication |
Sentence |
Publish Date |
Extraction Date |
Species |
| Suk-Yun Kang, Dae-Hyun Roh, Jung-Wan Choi, Yeonhee Ryu, Jang-Hern Le. Repetitive Treatment with Diluted Bee Venom Attenuates the Induction of Below-Level Neuropathic Pain Behaviors in a Rat Spinal Cord Injury Model. Toxins. vol 7. issue 7. 2016-04-12. PMID:26184310. |
moreover, sci-induced increase in spinal glia expression was also suppressed by repetitive dbv treatment in the ipsilateral dorsal spinal cord. |
2016-04-12 |
2023-08-13 |
rat |
| Maria M Spruill, Ralph W Kunc. Calbindin-D28K is increased in the ventral horn of spinal cord by neuroprotective factors for motor neurons. Journal of neuroscience research. vol 93. issue 8. 2016-04-01. PMID:25914366. |
after 4 weeks of incubation of spinal cord slices with 1) glial cell line-derived neurotrophic factor (gdnf), 2) neurturin, 3) insulin-like growth factor i (igf-i), or 4) pigment epithelium-derived factor (pedf), the number of calbindin-d28k -immunopositive large neurons (>20 μm) in the ventral horn was higher under the first three conditions, but not after pedf, compared with untreated controls. |
2016-04-01 |
2023-08-13 |
human |
| Safa Shehab, Mehwish Anwer, Divya Galani, Afaf Abdulkarim, Khuloud Al-Nuaimi, Abdullah Al-Baloushi, Saeed Tariq, Nico Nagelkerke, Milos Ljubisavljevi. Anatomical evidence that the uninjured adjacent L4 nerve plays a significant role in the development of peripheral neuropathic pain after L5 spinal nerve ligation in rats. The Journal of comparative neurology. vol 523. issue 12. 2016-03-22. PMID:25631932. |
perk immunoreactivity, which is not detectable in the normal spinal cord, was discernible in neurons (not glia) of the superficial dorsal horn after noxious heat stimuli. |
2016-03-22 |
2023-08-13 |
rat |
| Haruka Omoto, Shinji Matsumura, Manabu Kitano, Shinichiro Miyazaki, Toshiaki Minami, Seiji It. Comparison of mechanisms of allodynia induced by acromelic acid A between early and late phases. European journal of pharmacology. vol 760. 2016-03-08. PMID:25861935. |
activations of nnos and glia in the spinal cord were histochemically examined at 1 h or 1 week after injection of acro-a. |
2016-03-08 |
2023-08-13 |
mouse |
| Amin A Sheikh, Adel Mohame. Ependymal Proliferation: A Conduit for Tricking the Central Nervous System into Bioengineering Itself. Biomedical sciences instrumentation. vol 51. 2016-02-18. PMID:25996733. |
ependymal cells are a type of glial cell lining the ventricles and central canal of the spinal cord. |
2016-02-18 |
2023-08-13 |
Not clear |
| Geeta Ramesh, Peter J Didier, John D England, Lenay Santana-Gould, Lara A Doyle-Meyers, Dale S Martin, Mary B Jacobs, Mario T Philip. Inflammation in the pathogenesis of lyme neuroborreliosis. The American journal of pathology. vol 185. issue 5. 2016-01-21. PMID:25892509. |
histopathology revealed leptomeningitis, vasculitis, and focal inflammation in the central nervous system; necrotizing focal myelitis in the cervical spinal cord; radiculitis; neuritis and demyelination in the spinal roots; and inflammation with neurodegeneration in the drg that was concomitant with significant neuronal and satellite glial cell apoptosis. |
2016-01-21 |
2023-08-13 |
Not clear |
| Barbara B Raddatz, Annika Lehmbecker, Arno Kalkuhl, Ulrich Deschl, Wolfgang Baumgärtner, Reiner Ulric. Transcriptional analysis of glial cell differentiation in the postnatal murine spinal cord. International journal of developmental neuroscience : the official journal of the International Society for Developmental Neuroscience. vol 42. 2015-12-30. PMID:25702526. |
transcriptional analysis of glial cell differentiation in the postnatal murine spinal cord. |
2015-12-30 |
2023-08-13 |
mouse |
| A Takeda, Y Atobe, T Kadota, R C Goris, K Funakosh. Axonal regeneration through the fibrous scar in lesioned goldfish spinal cord. Neuroscience. vol 284. 2015-12-22. PMID:25290012. |
within 1 week after hemisection of the spinal cord, the open wound was closed by a fibrous scar that was demarcated from the surrounding nervous tissue by the glia limitans, which was immunoreactive for laminin. |
2015-12-22 |
2023-08-13 |
Not clear |
| Danju Tso, Randall D McKinno. Cell replacement therapy for central nervous system diseases. Neural regeneration research. vol 10. issue 9. 2015-11-25. PMID:26604878. |
the brain and spinal cord can not replace neurons or supporting glia that are lost through traumatic injury or disease. |
2015-11-25 |
2023-08-13 |
Not clear |
| Jenny A K Ekberg, James A St Joh. Olfactory ensheathing cells for spinal cord repair: crucial differences between subpopulations of the glia. Neural regeneration research. vol 10. issue 9. 2015-11-25. PMID:26604892. |
olfactory ensheathing cells for spinal cord repair: crucial differences between subpopulations of the glia. |
2015-11-25 |
2023-08-13 |
Not clear |
| Hans G Novrup, Valerie Bracchi-Ricard, Ditte G Ellman, Jerome Ricard, Anjana Jain, Erik Runko, Lise Lyck, Minna Yli-Karjanmaa, David E Szymkowski, Damien D Pearse, Kate L Lambertsen, John R Bethe. Central but not systemic administration of XPro1595 is therapeutic following moderate spinal cord injury in mice. Journal of neuroinflammation. vol 11. 2015-11-20. PMID:25204558. |
glial cell activation and overproduction of inflammatory mediators in the central nervous system (cns) have been implicated in acute traumatic injuries to the cns, including spinal cord injury (sci). |
2015-11-20 |
2023-08-13 |
mouse |
| Yona Goldshmit, Frisca Frisca, Alexander R Pinto, Alice Pébay, Jean-Kitty K Y Tang, Ashley L Siegel, Jan Kaslin, Peter D Curri. Fgf2 improves functional recovery-decreasing gliosis and increasing radial glia and neural progenitor cells after spinal cord injury. Brain and behavior. vol 4. issue 2. 2015-10-21. PMID:24683512. |
fgf2 improves functional recovery-decreasing gliosis and increasing radial glia and neural progenitor cells after spinal cord injury. |
2015-10-21 |
2023-08-12 |
Not clear |
| Yu Zhang, Zuchao Gu, Guixing Qiu, Yueming Son. Combination of chondroitinase ABC, glial cell line-derived neurotrophic factor and Nogo A antibody delayed-release microspheres promotes the functional recovery of spinal cord injury. The Journal of craniofacial surgery. vol 24. issue 6. 2015-09-16. PMID:24220426. |
combination of chondroitinase abc, glial cell line-derived neurotrophic factor and nogo a antibody delayed-release microspheres promotes the functional recovery of spinal cord injury. |
2015-09-16 |
2023-08-12 |
rat |
| Yan Zhu, Tomoko Matsumoto, Takashi Nagasawa, Fabienne Mackay, Fujio Murakam. Chemokine Signaling Controls Integrity of Radial Glial Scaffold in Developing Spinal Cord and Consequential Proper Position of Boundary Cap Cells. The Journal of neuroscience : the official journal of the Society for Neuroscience. vol 35. issue 24. 2015-09-01. PMID:26085643. |
we show that cxcr4, a receptor of chemokine cxcl12, is expressed in spinal cord radial glia. |
2015-09-01 |
2023-08-13 |
mouse |
| Yan Zhu, Tomoko Matsumoto, Takashi Nagasawa, Fabienne Mackay, Fujio Murakam. Chemokine Signaling Controls Integrity of Radial Glial Scaffold in Developing Spinal Cord and Consequential Proper Position of Boundary Cap Cells. The Journal of neuroscience : the official journal of the Society for Neuroscience. vol 35. issue 24. 2015-09-01. PMID:26085643. |
conditional knock-out of cxcr4 in radial glia caused disrupted radial glial scaffold with gaps at the pial endfeet layer and consequentially led to an invasion of boundary cap (bc) cells into the spinal cord. |
2015-09-01 |
2023-08-13 |
mouse |
| Chih-Yen Wang, Shang-Hsun Yang, Shun-Fen Tzen. MicroRNA-145 as one negative regulator of astrogliosis. Glia. vol 63. issue 2. 2015-08-19. PMID:25139829. |
astrogliosis occurs at the lesion site within days to weeks after spinal cord injury (sci) and involves the proliferation and hypertrophy of astrocytes, leading to glia scar formation. |
2015-08-19 |
2023-08-13 |
rat |
| H A Petrosyan, V Alessi, V Singh, A S Hunanyan, J M Levine, V L Arvania. Transduction efficiency of neurons and glial cells by AAV-1, -5, -9, -rh10 and -hu11 serotypes in rat spinal cord following contusion injury. Gene therapy. vol 21. issue 12. 2015-07-27. PMID:25119378. |
thus, aav-rh10 carries promising potential as a gene therapy vector, particularly if both the neuronal and glial cell populations in damaged spinal cord are targeted. |
2015-07-27 |
2023-08-13 |
human |
| Elizabeth Amy Old, Anna K Clark, Marzia Malcangi. The role of glia in the spinal cord in neuropathic and inflammatory pain. Handbook of experimental pharmacology. vol 227. 2015-07-27. PMID:25846618. |
the role of glia in the spinal cord in neuropathic and inflammatory pain. |
2015-07-27 |
2023-08-13 |
Not clear |
| Huaqing Liu, Mila Angert, Tasuku Nishihara, Igor Shubayev, Jennifer Dolkas, Veronica I Shubaye. Spinal Glia Division Contributes to Conditioning Lesion-Induced Axon Regeneration Into the Injured Spinal Cord: Potential Role of Cyclic AMP-Induced Tissue Inhibitor of Metalloproteinase-1. Journal of neuropathology and experimental neurology. vol 74. issue 6. 2015-07-27. PMID:25933384. |
spinal glia division contributes to conditioning lesion-induced axon regeneration into the injured spinal cord: potential role of cyclic amp-induced tissue inhibitor of metalloproteinase-1. |
2015-07-27 |
2023-08-13 |
Not clear |
| Huaqing Liu, Mila Angert, Tasuku Nishihara, Igor Shubayev, Jennifer Dolkas, Veronica I Shubaye. Spinal Glia Division Contributes to Conditioning Lesion-Induced Axon Regeneration Into the Injured Spinal Cord: Potential Role of Cyclic AMP-Induced Tissue Inhibitor of Metalloproteinase-1. Journal of neuropathology and experimental neurology. vol 74. issue 6. 2015-07-27. PMID:25933384. |
here, using bromodeoxyuridine, mitomycin (mitosis inhibitor), and cholera toxin b tracer, we demonstrate that snc-induced division of spinal glia is related to the spinal induction of tissue inhibitor of metalloproteinase-1 and contributes to central sensory axon growth into the damaged spinal cord. |
2015-07-27 |
2023-08-13 |
Not clear |