| Publication |
Sentence |
Publish Date |
Extraction Date |
Species |
| L Ahtee, S Kaakkol. Effect of mecamylamine on the fate of dopamine in striatal and mesolimbic areas of rat brain; interaction with morphine and haloperidol. British journal of pharmacology. vol 62. issue 2. 1978-04-17. PMID:564219. |
5 the results indicate that mecamylamine inhibits the release of dopamine both from the striatal and mesolimbic dopaminergic neurones. |
1978-04-17 |
2023-08-11 |
rat |
| J L Gordon, H J Olverma. 5-Hydroxytryptamine and dopamine transport by rat and human blood platelets. British journal of pharmacology. vol 62. issue 2. 1978-04-17. PMID:623937. |
consequently although platelets may be valid models of transport in 5-hydroxytryptaminergic neurones, they should not be regarded as models for the dopamine transport mechanism found in dopaminergic neurones. |
1978-04-17 |
2023-08-11 |
human |
| D Tsang, S La. Effect of monoamine receptor agonists and antagonists on cyclic AMP accumulation in human cerebral cortex slices. Canadian journal of physiology and pharmacology. vol 55. issue 6. 1978-03-29. PMID:23211. |
in human cerebral cortex slices noradrenaline, isoproterenol (a beta-adrenergic agonist), dopamine, apomorphine (a dopaminergic agonist), and serotonin stimulated cyclic amp formation: noradrenaline greater than or equal to isoproterenol greater than dopamine = apomorphine = serotonin. |
1978-03-29 |
2023-08-11 |
human |
| D Tsang, S La. Effect of monoamine receptor agonists and antagonists on cyclic AMP accumulation in human cerebral cortex slices. Canadian journal of physiology and pharmacology. vol 55. issue 6. 1978-03-29. PMID:23211. |
pimozide (a selective dopaminergic antagonist) inhibited the increase of cyclic amp formation induced by dopamine or apomorphine but not that induced by noradrenaline, isoproterenol, or serotonin. |
1978-03-29 |
2023-08-11 |
human |
| G Langer, E J Sacha. Dopaminergic factors in human prolactin regulation: effects of neuroleptics and dopamine. Psychoneuroendocrinology. vol 2. issue 4. 1978-03-29. PMID:601174. |
dopaminergic factors in human prolactin regulation: effects of neuroleptics and dopamine. |
1978-03-29 |
2023-08-11 |
human |
| A Carlsso. Antipsychotic drugs, neurotransmitters, and schizophrenia. The American journal of psychiatry. vol 135. issue 2. 1978-03-21. PMID:23684. |
the fact that dopaminergic hyperfunction induced by amphetamines or dopa may lead to a disturbance mimicking paranoid schizophrenia lends further support for a key role of dopamine in mental functions. |
1978-03-21 |
2023-08-11 |
Not clear |
| A Carlsso. Antipsychotic drugs, neurotransmitters, and schizophrenia. The American journal of psychiatry. vol 135. issue 2. 1978-03-21. PMID:23684. |
although a primary disturbance in dopamine function in schizophrenia cannot be ruled out, the intimate relationship between dopaminergic and other neuronal systems must be emphasized. |
1978-03-21 |
2023-08-11 |
Not clear |
| G J Trachte, A M Lefe. Influence of dopamine on liver dynamics in hemorrhagic shock. Circulatory shock. vol 4. issue 4. 1978-03-21. PMID:598004. |
the increase in hepatic artery and portal vein flows appeared to result from stimulation of dopaminergic receptors since the dopamine dose was in the dopaminergic range and because haloperidol, a dopamine blocker, abolished the improved hemodynamic effects of dopamine. |
1978-03-21 |
2023-08-11 |
Not clear |
| A Morelli, F Narducci, G Camardella, M A Pelli, A Floridi, G G Nenc. Dopamine uptake by platelets in hepatic encephalopathy; evidence for a possible depletion of brain dopamine. Acta hepato-gastroenterologica. vol 24. issue 6. 1978-03-10. PMID:341631. |
a defective dopamine uptake by the he platelets suggests that a similar biochemical derangement is, also, present in the nerve cells of the dopaminergic system. |
1978-03-10 |
2023-08-11 |
Not clear |
| O S Steinsland, J P Hiebl. Dopaminergic inhibition of adrenergic neurotransmission as a model for studies on dopamine receptor mechanisms. Science (New York, N.Y.). vol 199. issue 4327. 1978-02-23. PMID:22933. |
the calculated affinities of these drugs for the dopaminergic receptor correlate closely with both the pharmacological potencies of these drugs in vivo and their reported potencies as inhibitors of [3h]haloperidol binding to "dopamine receptors" in brain homogenates. |
1978-02-23 |
2023-08-11 |
rabbit |
| T J Crowley, M M Hoehn, C O Rutledge, M A Stallings, R K Heaton, S Sundell, D Stilso. Dopamine excretion and vulnerability to drug-induced Parkinsonism. Schizophrenic patients. Archives of general psychiatry. vol 35. issue 1. 1978-02-23. PMID:619844. |
this study of schizophrenic patients, and our previous research in parkinson's disease, suggest that urinary dopamine excretion may reflect dopaminergic function of the extrapyramidal motor system in both conditions. |
1978-02-23 |
2023-08-11 |
Not clear |
| A Carlsso. Does dopamine play a role in schizophrenia? Psychological medicine. vol 7. issue 4. 1978-02-18. PMID:22890. |
dopaminergic hyperfunction, induced by amphetamines or dopa, may lead to a disturbance mimicking paranoid schizophrenia, lending further support for a key role of dopamine in mental functions. |
1978-02-18 |
2023-08-11 |
Not clear |
| A Carlsso. Does dopamine play a role in schizophrenia? Psychological medicine. vol 7. issue 4. 1978-02-18. PMID:22890. |
while a primary disturbance in dopamine function in schizophrenia cannot be ruled out, the intimate relationship between dopaminergic and other neuronal systems must be emphasized. |
1978-02-18 |
2023-08-11 |
Not clear |
| S Yehuda, M Kah. D-Amphetamine thermal effects, metabolic rate and motor activity in rats. The International journal of neuroscience. vol 7. issue 4. 1978-02-18. PMID:591193. |
the effect of d-amphetamine-induced hypothermia among rats maintained at a cold ambient temperature is related to its ability to release dopamine in the dopaminergic neurons in the mesolimbic pathway. |
1978-02-18 |
2023-08-11 |
rat |
| S Consolo, H Ladinsky, S Bianchi, D Ghezz. Apparent lack of a dopaminergic-cholinergic link in the rat nucleus accumbens septi-tuberculum olfactorium. Brain research. vol 135. issue 2. 1978-01-27. PMID:922475. |
the nucleus accumbens septi and tuberculum olfactorium (nas-to), which from part of the mesolimbic dopaminergic system, and the striatum, which is part of the nigrostriatal dopamingeric system, contain high levels of both dopamine (da) and acetylcholine and resemble each other in some other biochemical properties. |
1978-01-27 |
2023-08-11 |
rat |
| P Bedard, J D Parkes, C D Marsde. Nomifensine in Parkinson's disease. British journal of clinical pharmacology. vol 4Suppl 2. 1977-12-29. PMID:334223. |
loss of dopaminergic nigrostriatal neurones characterizes parkinson's disease, and it is probably for this reason that indirect dopamine (da) agonists, whose actions depend on intact presynaptic mechanisms, are less active. |
1977-12-29 |
2023-08-11 |
Not clear |
| A Nieoullon, A Cheramy, J Glowinsk. Interdependence of the nigrostriatal dopaminergic systems on the two sides of the brain in the cat. Science (New York, N.Y.). vol 198. issue 4315. 1977-11-30. PMID:910137. |
the release of [3h]dopamine also decreased in the left caudate nucleus and increased in the right structure when dopamine was applied to the left substantia nigra which reduces the activity of the left dopaminergic pathway. |
1977-11-30 |
2023-08-11 |
cat |
| N K Dadkar, A N Dohadwalla, B K Bhattachary. Possible role of dopamine in central effects of cocaine as measured by apomorphine gnawing test in mice. Psychopharmacology. vol 52. issue 2. 1977-09-22. PMID:407594. |
these findings would suggest that dopaminergic mechanism plays a significant role in the potentiation of apomorphine gnawing activity by cocaine and also support the view that inhibition of dopamine uptake is responsible for the stimulatory action of cocaine. |
1977-09-22 |
2023-08-11 |
mouse |
| S E Vizi, A Rónai, L Hársing, J Knol. Inhibitory effect of dopamine on acetylcholine release from caudate nucleus. Polish journal of pharmacology and pharmacy. vol 29. issue 3. 1977-09-22. PMID:887499. |
it has been shown that dopamine inhibits the release of acetylcholine (ach) from nerve terminals of caudate cholinergic interneurons, and the imbalance between dopaminergic and cholinergic system by 6-hydroxydopamine pretreatment leads to an increased ach release. |
1977-09-22 |
2023-08-11 |
Not clear |
| S E Vizi, A Rónai, L Hársing, J Knol. Inhibitory effect of dopamine on acetylcholine release from caudate nucleus. Polish journal of pharmacology and pharmacy. vol 29. issue 3. 1977-09-22. PMID:887499. |
the parkinson-syndrome induced by drugs which depress dopaminergic function either by reducing the output of dopamine, such as reserpine, or by antagonizing its action on da receptors, as for instance haloperidol or chlorpromazine, can be attributed to the augmented release of ach from caudate cholinergic nerve terminals and a consequent increase of cholinergic outflow from caudate nucleus. |
1977-09-22 |
2023-08-11 |
Not clear |