All Relations between dopaminergic and dopamine

Publication Sentence Publish Date Extraction Date Species
K A Keefe, J D Salamone, M J Zigmond, E M Stricke. Paradoxical kinesia in parkinsonism is not caused by dopamine release. Studies in an animal model. Archives of neurology. vol 46. issue 10. 1989-11-09. PMID:2508609. these results suggest that the paradoxical kinesia of dopamine-depleted rats is not a consequence of dopamine release from residual dopaminergic fibers. 1989-11-09 2023-08-11 rat
S Ofori, R E Heikkila, W J Nickla. Attenuation by dopamine uptake blockers of the inhibitory effects of 1-methyl-4-phenyl-1,2,3,6-tetrahydropyridine and some of its analogs on NADH-linked metabolism in mouse neostriatal slices. The Journal of pharmacology and experimental therapeutics. vol 251. issue 1. 1989-11-07. PMID:2571719. in the present study we investigated if dopamine uptake blockers, shown previously to protect against tetrahydropyridine-induced dopaminergic neurotoxicity, would attenuate mptp- and 2'me-mptp-promoted lactate formation in neostriatal slices. 1989-11-07 2023-08-11 mouse
G S Robertson, H A Robertso. Evidence that L-dopa-induced rotational behavior is dependent on both striatal and nigral mechanisms. The Journal of neuroscience : the official journal of the Society for Neuroscience. vol 9. issue 9. 1989-11-07. PMID:2795165. this is accompanied by a loss of dopamine in brain regions, such as the corpus striatum, which receives input from dopaminergic neurons in the substantia nigra (sn). 1989-11-07 2023-08-11 rat
M R Kilbourn, M S Haka, G K Mulholland, P S Sherman, T Pisan. Regional brain distribution of [18F]GBR 13119, a dopamine uptake inhibitor, in CD-1 and C57BL/6 mice. European journal of pharmacology. vol 166. issue 2. 1989-11-06. PMID:2529129. we conclude mice are an appropriate model for the in vivo study of the dopamine uptake system, and that [18f]gbr 13119 may be a suitable in vivo marker for degeneration of striatal dopaminergic neurons. 1989-11-06 2023-08-11 mouse
W H Baldridge, A K Ball, R G Mille. Gap junction particle density of horizontal cells in goldfish retinas lesioned with 6-OHDA. The Journal of comparative neurology. vol 287. issue 2. 1989-10-31. PMID:2507595. to determine whether the light-dependent changes in gap junction morphology are due to endogenous dopamine release, horizontal cell gap junctions were studied in goldfish retinas lacking dopaminergic neurons. 1989-10-31 2023-08-11 Not clear
D A Perese, J Ulman, J Viola, S E Ewing, K S Bankiewic. A 6-hydroxydopamine-induced selective parkinsonian rat model. Brain research. vol 494. issue 2. 1989-10-26. PMID:2528389. this resulted in reproducible, quantifiable rotational behavior in response to either amphetamine or apomorphine and a near total depletion of dopamine in the striatum ipsilateral to the lesion with a dorsolateral distribution of supersensitive dopaminergic d2 receptors. 1989-10-26 2023-08-11 human
H Ikegami, S A Spahn, C Prasa. Neuropeptide-dopamine interactions. IV. Effect of thyrotropin-releasing hormone on striatal dopaminergic neurons. Peptides. vol 10. issue 3. 1989-10-20. PMID:2528727. in conclusion, these data suggest that in vivo administration of trh may modulate dopaminergic activities by altering, directly or indirectly, dopamine release and reuptake. 1989-10-20 2023-08-11 Not clear
A Kolta, T A Reade. Modulatory effects of catecholamines on neurons of the rat visual cortex: single-cell iontophoretic studies. Canadian journal of physiology and pharmacology. vol 67. issue 6. 1989-10-20. PMID:2570629. the catecholamines noradrenaline and dopamine have been proposed as neuromodulators of cortical neuron excitability, and such a regulation could be mediated by specific adrenergic and dopaminergic receptors. 1989-10-20 2023-08-11 rat
W D Kerns, E Arena, D G Morga. Role of dopaminergic and adrenergic receptors in the pathogenesis of arterial lesions induced by fenoldopam mesylate and dopamine in the rat. The American journal of pathology. vol 135. issue 2. 1989-10-20. PMID:2571297. role of dopaminergic and adrenergic receptors in the pathogenesis of arterial lesions induced by fenoldopam mesylate and dopamine in the rat. 1989-10-20 2023-08-11 rat
W D Kerns, E Arena, D G Morga. Role of dopaminergic and adrenergic receptors in the pathogenesis of arterial lesions induced by fenoldopam mesylate and dopamine in the rat. The American journal of pathology. vol 135. issue 2. 1989-10-20. PMID:2571297. dopamine, an alpha- and beta-adrenoceptor and dopaminergic agonist, is used clinically, principally as a pressor agent. 1989-10-20 2023-08-11 rat
W D Kerns, E Arena, D G Morga. Role of dopaminergic and adrenergic receptors in the pathogenesis of arterial lesions induced by fenoldopam mesylate and dopamine in the rat. The American journal of pathology. vol 135. issue 2. 1989-10-20. PMID:2571297. thus, the induction of splanchnic arterial lesions in the rat by dopamine and fm is caused by stimulation of, and interaction between, alpha-adrenoceptors and dopaminergic da1 receptors. 1989-10-20 2023-08-11 rat
A J Stoessl, M T Martin-Iverson, T M Barth, C T Dourish, S D Iverse. Effects of ageing on the behavioural responses to dopamine agonists: decreased yawning and locomotion, but increased stereotypy. Brain research. vol 495. issue 1. 1989-10-19. PMID:2570627. these findings suggest that (1) the dopamine receptors mediating yawning and stereotypy have different anatomical locations (2) ageing is associated with decreased responsiveness to stimulation of dopamine autoreceptors, consequent upon the loss of dopaminergic nerve terminals, and (3) while the functional response to selective stimulation of postsynaptic d2 receptors decreases with age, the postsynaptic response to a mixed d1/d2 agonist increases. 1989-10-19 2023-08-11 rat
P A Broderic. Characterizing stearate probes in vitro for the electrochemical detection of dopamine and serotonin. Brain research. vol 495. issue 1. 1989-10-19. PMID:2776030. the methodology is relevant to the interpretation of electrochemical signals for dopamine and serotonin in vivo in neuroanatomical substrates, richly innervated by dopaminergic and serotonergic neuronal circuitry. 1989-10-19 2023-08-11 Not clear
B Moghaddam, B S Bunne. Differential effect of cocaine on extracellular dopamine levels in rat medial prefrontal cortex and nucleus accumbens: comparison to amphetamine. Synapse (New York, N.Y.). vol 4. issue 2. 1989-10-19. PMID:2781466. based on the relatively small increase of extracellular dopamine levels in medial prefrontal cortex by cocaine, it is postulated that dopaminergic innervation of other structures besides medial prefrontal cortex may be involved in maintenance of cocaine self-administration. 1989-10-19 2023-08-11 rat
E Chung, M T Dvorozniak, M H Van Woert, H C L. Regional distribution of calcium/calmodulin-dependent phosphatase activity of calcineurin in rat brain. Research communications in chemical pathology and pharmacology. vol 64. issue 3. 1989-10-13. PMID:2551001. within the striatum, calcineurin activity does not appear to be present in dopaminergic terminals, since lesions of the nigro-striatal dopaminergic pathway (which reduce striatal dopamine levels by 97%) had no effect on calcineurin activity. 1989-10-13 2023-08-11 rat
R K Carder, D Jackson, H J Morris, R D Lund, M J Zigmon. Dopamine released from mesencephalic transplants restores modulation of striatal acetylcholine release after neonatal 6-hydroxydopamine: an in vitro analysis. Experimental neurology. vol 105. issue 3. 1989-10-10. PMID:2569987. we wished to determine whether dopamine released from the transplant also reinstated dopaminergic inhibition of striatal acetylcholine release. 1989-10-10 2023-08-11 rat
H Haikala, T Leikola-Pelh. Nicotine antagonizes the effects of reserpine on the striatal metabolism of dopamine, 5-hydroxytryptamine and noradrenaline in hypothermic but not in normothermic mice. Naunyn-Schmiedeberg's archives of pharmacology. vol 339. issue 5. 1989-09-29. PMID:2770886. the changes in striatal metabolism of dopamine, noradrenaline and 5-hydroxytryptamine (5-ht) we nicotine had temperature dependent effects on the dopamine metabolism which indicates a block of dopaminergic neurons as suggested in our earlier studies. 1989-09-29 2023-08-11 mouse
S Rose, M Nomoto, E Kelly, G Kilpatrick, P Jenner, C D Marsde. Increased caudate dopamine turnover may contribute to the recovery of motor function in marmosets treated with the dopaminergic neurotoxin MPTP. Neuroscience letters. vol 101. issue 3. 1989-09-25. PMID:2505199. increased caudate dopamine turnover may contribute to the recovery of motor function in marmosets treated with the dopaminergic neurotoxin mptp. 1989-09-25 2023-08-11 Not clear
B Dean, D L Copolo. Dopamine uptake by platelets is selective, temperature dependent and not influenced by the dopamine-D1 or dopamine-D2 receptor. Life sciences. vol 45. issue 5. 1989-09-25. PMID:2570336. the human platelet, which takes up and releases dopamine, has been proposed as a peripheral model for the study of dopaminergic neurons in the central nervous system (cns). 1989-09-25 2023-08-11 human
M F Suaud-Chagny, M Buda, F G Gono. Pharmacology of electrically evoked dopamine release studied in the rat olfactory tubercle by in vivo electrochemistry. European journal of pharmacology. vol 164. issue 2. 1989-09-20. PMID:2788097. dopamine (da) release was evoked by electrical stimulation of the dopaminergic pathway. 1989-09-20 2023-08-11 rat