| Publication |
Sentence |
Publish Date |
Extraction Date |
Species |
| C Castellano, F Pavon. Effects of the selective kappa-opioid receptor agonist U-50,488 on locomotor activity and passive avoidance behaviour in DBA/2 and C57BL/6 mice. Archives internationales de pharmacodynamie et de therapie. vol 288. issue 2. 1987-12-04. PMID:2823735. |
moreover, this activity depression was enhanced in both strains by the administration of haloperidol and muscimol, showing the involvement of dopaminergic and gabaergic mechanisms. |
1987-12-04 |
2023-08-11 |
mouse |
| K Kashihara, K Fukuda, M Sato, S Otsuk. Haloperidol prevents the methamphetamine-induced apomorphine subsensitivity of dopamine metabolism in rat striatum. Neuroscience research. vol 4. issue 5. 1987-12-01. PMID:3670749. |
since gamma-butyrolactone was administered in combination with apomorphine so as to remove complications which might arise from the modification of impulse flow through the striatonigral feedback loop, it is possible that the observed effect of apomorphine upon striatal dopaminergic metabolism and its antagonism with haloperidol may be mediated by autoreceptors of dopaminergic axon terminals in rat striatum. |
1987-12-01 |
2023-08-11 |
rat |
| V Huang, H Figge, R Demlin. Haloperidol complications in burn patients. The Journal of burn care & rehabilitation. vol 8. issue 4. 1987-11-20. PMID:3654715. |
haloperidol causes a relative imbalance of dopaminergic and cholinergic neuronal activity in the basal ganglia with a relative increase in cholinergic activity being responsible for eps. |
1987-11-20 |
2023-08-11 |
Not clear |
| M W Emmett-Oglesby, A Her. Opioid modulation of the discriminative stimulus produced by pentylenetetrazol. Psychopharmacology. vol 92. issue 3. 1987-10-16. PMID:2819916. |
the observation that haloperidol blocked the ptz-like stimulus of mu agonists suggests the possible involvement of dopaminergic mechanisms in the mediation of the effect. |
1987-10-16 |
2023-08-11 |
rat |
| D G Morgan, J S Randall, N A Telford, M N Gordon, Y N Sinha, C E Finch, P K Randal. Genotypic influences on pituitary responsiveness to haloperidol in mice. Psychoneuroendocrinology. vol 12. issue 3. 1987-08-31. PMID:3615750. |
previous studies from this laboratory demonstrated that cba/j mice have impaired striatal dopaminergic supersensitivity in response to subchronic haloperidol administration. |
1987-08-31 |
2023-08-11 |
mouse |
| S Olczak, J W Guzek, B Stempnia. The hypothalamic and neurohypophysial vasopressor and oxytocic activities in stressed rats treated with haloperidol. Experimental and clinical endocrinology. vol 89. issue 2. 1987-08-28. PMID:3609161. |
in rats otherwise not treated, dopaminergic blockade due to haloperidol was followed by an increase of the vasopressin content in the neurohypophysis. |
1987-08-28 |
2023-08-11 |
rat |
| J M Frey, W W Morgan, M K Ticku, R D Huffma. A dietary haloperidol regimen for inducing dopamine receptor supersensitivity in rats. Pharmacology, biochemistry, and behavior. vol 26. issue 4. 1987-07-29. PMID:3602028. |
the induction of dopaminergic supersensitivity in rats by the administration of haloperidol in their diet for 30 days (chal) in three increasing concentrations (7-15 mg/kg/day) was compared to that induced by single daily subcutaneous injections (schal, 0.7 mg/kg) on the basis of biochemical (radioimmunoassay of serum haloperidol levels, 3h-spiroperidol binding) or behavioral (apomorphine stereotypy, spontaneous locomotor activity) parameters. |
1987-07-29 |
2023-08-11 |
rat |
| M Bourin, L Poisson, C Larouss. Piracetam interactions with neuroleptics in psychopharmacological tests. Neuropsychobiology. vol 16. issue 2-3. 1987-07-02. PMID:2884593. |
two psychopharmacological tests which usually predict neuroleptic activity were conducted after joint administration of piracetam and three neuroleptics (haloperidol, fluphenazine and sulpiride) chosen for their different chemical classes and dopaminergic affinities. |
1987-07-02 |
2023-08-11 |
Not clear |
| A Louilot, M Le Moal, H Simo. Presynaptic control of dopamine metabolism in the nucleus accumbens. Lack of effect of buspirone as demonstrated using in vivo voltammetry. Life sciences. vol 40. issue 20. 1987-06-23. PMID:3573992. |
in the present study, presynaptic effects of buspirone on dopaminergic metabolism in the nucleus accumbens were investigated, and they were compared to the effects of the classical neuroleptic, haloperidol. |
1987-06-23 |
2023-08-11 |
Not clear |
| A Louilot, M Le Moal, H Simo. Presynaptic control of dopamine metabolism in the nucleus accumbens. Lack of effect of buspirone as demonstrated using in vivo voltammetry. Life sciences. vol 40. issue 20. 1987-06-23. PMID:3573992. |
dopaminergic terminals were isolated by infusion of tetrodotoxin into the median forebrain bundle in order to evaluate the effects of buspirone and haloperidol on presynaptic receptors. |
1987-06-23 |
2023-08-11 |
Not clear |
| P C Contreras, R Quirion, D R Gehlert, M L Contreras, T L O'Donohu. Autoradiographic distribution of non-dopaminergic binding sites labeled by [3H]haloperidol in rat brain. Neuroscience letters. vol 75. issue 2. 1987-06-16. PMID:3033560. |
the distribution of non-dopaminergic binding sites labeled by haloperidol was clearly quite different from that labeled by dopaminergic ligands. |
1987-06-16 |
2023-08-11 |
rat |
| R M Quock, F J Kouchich, T K Ishii, D G Lang. Microwave facilitation of domperidone antagonism of apomorphine-induced stereotypic climbing in mice. Bioelectromagnetics. vol 8. issue 1. 1987-06-11. PMID:3579999. |
this drug effect was competitively inhibited by systemic pretreatment with the centrally acting dopaminergic antagonist haloperidol but not by microwave irradiation (2.45 ghz, 20 mw/cm2, cw, 10 min) nor by systemic pretreatment with domperidone, a dopaminergic antagonist that only poorly penetrates the blood-brain barrier (bbb). |
1987-06-11 |
2023-08-11 |
mouse |
| H Takahashi, J Tanaka, S Tsuda, Y Shiras. Contribution of monoaminergic nervous system in potentiation of 2-sec-butylphenyl N-methylcarbamate (BPMC) toxicity by malathion in male mice. Fundamental and applied toxicology : official journal of the Society of Toxicology. vol 8. issue 3. 1987-06-10. PMID:3569712. |
the acute lethality of bpmc was decreased by muscarinic blockers (atropine, methylatropine, or trihexyphenidyl) or a monoamine oxidase inhibior (pargyline) and increased by a monoamine depleter (reserpine) or a dopaminergic blocker (haloperidol). |
1987-06-10 |
2023-08-11 |
mouse |
| N P Shugalev, A D Shiriae. [EEG analysis of the effect of stimulation of GABAergic structures of the caudate nucleus on the development of the effect of haloperidol]. Zhurnal vysshei nervnoi deiatelnosti imeni I P Pavlova. vol 37. issue 1. 1987-06-08. PMID:3577401. |
functional significance of gaba-structures of the caudate nucleus was studied by eeg analysis of the influence of stimulation of these structures on the development of the effect of systemic introduction of the blockader of dopaminergic receptors haloperidol. |
1987-06-08 |
2023-08-11 |
Not clear |
| J M Van Ree, N Ramse. The dopamine hypothesis of opiate reward challenged. European journal of pharmacology. vol 134. issue 2. 1987-05-29. PMID:3032655. |
haloperidol was administered either systemically or locally into different brain areas with dopaminergic terminals in rats allowed to initiate and maintain intravenous heroin self-administration. |
1987-05-29 |
2023-08-11 |
rat |
| Y Kataoka, H Ohta, M Fujiwara, R Oishi, S Uek. Noradrenergic involvement in catalepsy induced by delta 9-tetrahydrocannabinol. Neuropharmacology. vol 26. issue 1. 1987-05-07. PMID:3031535. |
these results indicate that noradrenergic neurons have an important role in the manifestation of catalepsy induced by thc, whereas dopaminergic neurons are important in catalepsy induced by haloperidol. |
1987-05-07 |
2023-08-11 |
rat |
| L S Lin, W T Chiu, C J Shih, M T Li. Involvement of both opiate and catecholaminergic receptors of ventromedial hypothalamus in the locomotor stimulant action of thyrotropin-releasing hormone. Journal of neural transmission. vol 68. issue 3-4. 1987-04-27. PMID:3031208. |
the locomotor stimulant action provoked by trh was antagonized by pretreatment of ventromedial hypothalamus with either an alpha-adrenergic receptor antagonist (yohimbine), a dopaminergic receptor antagonist (haloperidol) or an opiate receptor antagonist (naloxone), but not with a beta-adrenergic receptor antagonist (propranolol). |
1987-04-27 |
2023-08-11 |
rat |
| G Gallagher, A Brown, S Szab. Effect of dopamine-related drugs on duodenal ulcer induced by cysteamine or propionitrile: prevention and aggravation may not be mediated by gastrointestinal secretory changes in the rat. The Journal of pharmacology and experimental therapeutics. vol 240. issue 3. 1987-04-27. PMID:3559980. |
aggravation of cysteamine-induced duodenal ulcer was seen especially after (-)-butaclamol, (-)-sulpiride, haloperidol and, less effectively, after other dopaminergic antagonists. |
1987-04-27 |
2023-08-11 |
rat |
| b' I P Ashmarin, N Iu Syracheva, T I Vlasova, V N Kalikhevich, A A Kamenski\\xc4\\xa. [Taftsin correction of pharmacologically induced behavioral disorders in white rats]. Biulleten\' eksperimental\'noi biologii i meditsiny. vol 103. issue 2. 1987-04-17. PMID:3814799.' |
at the same time tuftsin had no effect when applied after the treatment by dopaminergic drugs (dtc, haloperidol, apomorphine). |
1987-04-17 |
2023-08-11 |
rat |
| E Jungmann, G Germann, I Austin, B Mack, D Storp-Wenke, W Fassbinder, U Schwedes, K H Usadel, A Encke, K Schöfflin. The role of adrenal medulla in endogenous dopaminergic inhibition of aldosterone secretion. Research in experimental medicine. Zeitschrift fur die gesamte experimentelle Medizin einschliesslich experimenteller Chirurgie. vol 186. issue 6. 1987-04-13. PMID:3547525. |
to examine potential sources of the dopamine thus inhibitorily acting in the adrenal zona glomerulosa, the responsiveness of aldosterone, plasma renin activity, prolactin, and plasma catecholamines to haloperidol, a dopaminergic antagonist, was studied in rats 6 weeks after unilateral adrenalectomy (group b), 6 weeks after unilateral adrenal demedullation followed by contralateral adrenalectomy 5 days later (group c), and in controls without any pretreatment (group a). |
1987-04-13 |
2023-08-11 |
rat |