| Publication |
Sentence |
Publish Date |
Extraction Date |
Species |
| Sara González, Marta Valenti, Rosario de Miguel, Filomena Fezza, Javier Fernández-Ruiz, Vincenzo Di Marzo, José A Ramo. Changes in endocannabinoid contents in reward-related brain regions of alcohol-exposed rats, and their possible relevance to alcohol relapse. British journal of pharmacology. vol 143. issue 4. 2005-02-01. PMID:15371286. |
based on these data, we examined whether pharmacological activation/blockade of endocannabinoid transmission might influence ethanol intake in rats allowed to relapse to alcohol consumption after subsequent periods of alcoholization and alcohol deprivation. |
2005-02-01 |
2023-08-12 |
rat |
| Sara González, Marta Valenti, Rosario de Miguel, Filomena Fezza, Javier Fernández-Ruiz, Vincenzo Di Marzo, José A Ramo. Changes in endocannabinoid contents in reward-related brain regions of alcohol-exposed rats, and their possible relevance to alcohol relapse. British journal of pharmacology. vol 143. issue 4. 2005-02-01. PMID:15371286. |
treatment with either delta(9)-tetrahydrocannabinol or cp55,940, two cannabinoid agonists, reduced both total liquid and ethanol intake but did not affect ethanol preference. |
2005-02-01 |
2023-08-12 |
rat |
| Sara González, Marta Valenti, Rosario de Miguel, Filomena Fezza, Javier Fernández-Ruiz, Vincenzo Di Marzo, José A Ramo. Changes in endocannabinoid contents in reward-related brain regions of alcohol-exposed rats, and their possible relevance to alcohol relapse. British journal of pharmacology. vol 143. issue 4. 2005-02-01. PMID:15371286. |
treatment with sr141716, a selective cannabinoid cb(1) receptor antagonist, also produced a significant reduction in both total liquid and ethanol intake without affecting ethanol preference. |
2005-02-01 |
2023-08-12 |
rat |
| Sara González, Marta Valenti, Rosario de Miguel, Filomena Fezza, Javier Fernández-Ruiz, Vincenzo Di Marzo, José A Ramo. Changes in endocannabinoid contents in reward-related brain regions of alcohol-exposed rats, and their possible relevance to alcohol relapse. British journal of pharmacology. vol 143. issue 4. 2005-02-01. PMID:15371286. |
treatment with cannabinoid agonists or a selective cb(1) receptor antagonist resulted in a reduction of ethanol intake by rats allowed to relapse to alcohol consumption after periods of alcoholization and alcohol deprivation, but these effects did not appear to be due to changes in neurobiological substrates currently involved in alcohol reinforcement/relapse. |
2005-02-01 |
2023-08-12 |
rat |
| Peer K Lilleng, Lars Ivar Mehlum, Liliana Bachs, Inge Moril. Deaths after intravenous misuse of transdermal fentanyl. Journal of forensic sciences. vol 49. issue 6. 2005-01-21. PMID:15568716. |
in the first case, the toxicological analysis revealed fentanyl (2.7 ng/ml), morphine (31.4 ng/ml), and ethanol (1.1 g/l) in postmortem blood and amphetamine, cannabinoids, morphine, and ethanol (1.4 g/l) in postmortem urine. |
2005-01-21 |
2023-08-12 |
Not clear |
| Audra L Stinchcomb, Satyanarayana Valiveti, Dana C Hammell, Diana R Ramse. Human skin permeation of Delta8-tetrahydrocannabinol, cannabidiol and cannabinol. The Journal of pharmacy and pharmacology. vol 56. issue 3. 2004-07-20. PMID:15025853. |
combinations of these cannabinoids with ethanol will be further studied in transdermal patch formulations in vitro and in vivo, as significant flux levels of all the drugs were obtained. |
2004-07-20 |
2023-08-12 |
human |
| Sergio Ortiz, José M Oliva, Sandra Pérez-Rial, Tomás Palomo, Jorge Manzanare. Chronic ethanol consumption regulates cannabinoid CB1 receptor gene expression in selected regions of rat brain. Alcohol and alcoholism (Oxford, Oxfordshire). vol 39. issue 2. 2004-06-28. PMID:14998822. |
chronic ethanol consumption regulates cannabinoid cb1 receptor gene expression in selected regions of rat brain. |
2004-06-28 |
2023-08-12 |
rat |
| Sergio Ortiz, José M Oliva, Sandra Pérez-Rial, Tomás Palomo, Jorge Manzanare. Chronic ethanol consumption regulates cannabinoid CB1 receptor gene expression in selected regions of rat brain. Alcohol and alcoholism (Oxford, Oxfordshire). vol 39. issue 2. 2004-06-28. PMID:14998822. |
the aim of this study was to examine the effects of chronic ethanol consumption in cannabinoid cb(1) receptor gene expression in wistar rats. |
2004-06-28 |
2023-08-12 |
rat |
| Javier Fernández-Solari, Camila Scorticati, Claudia Mohn, Andrea De Laurentiis, Silvia Billi, Ana Franchi, Samuel M McCann, Valeria Rettor. Alcohol inhibits luteinizing hormone-releasing hormone release by activating the endocannabinoid system. Proceedings of the National Academy of Sciences of the United States of America. vol 101. issue 9. 2004-06-17. PMID:14981261. |
we hypothesized that ethanol (etoh) might act through the endocannabinoid system to inhibit luteinizing hormone-releasing hormone (lhrh) release. |
2004-06-17 |
2023-08-12 |
Not clear |
| Mickaël Naassila, Olivier Pierrefiche, Catherine Ledent, Martine Daous. Decreased alcohol self-administration and increased alcohol sensitivity and withdrawal in CB1 receptor knockout mice. Neuropharmacology. vol 46. issue 2. 2004-05-28. PMID:14680762. |
recent advances in the understanding of the neurobiological basis of alcohol dependence suggest that the endocannabinoid system may play a key role in the reinforcing effects of ethanol. |
2004-05-28 |
2023-08-12 |
mouse |
| Balapal S Basavarajappa, Mariko Saito, Thomas B Cooper, Basalingappa L Hungun. Chronic ethanol inhibits the anandamide transport and increases extracellular anandamide levels in cerebellar granule neurons. European journal of pharmacology. vol 466. issue 1-2. 2004-03-08. PMID:12679143. |
anandamide transport processes are independent of cannabinoid cb1 receptor, as cannabinoid cb1 receptor knockout mice exhibited time-dependent anandamide transport and cannabinoid cb1 receptor antagonists did not alter the effects of chronic ethanol on anandamide transport. |
2004-03-08 |
2023-08-12 |
mouse |
| Frank Musshoff, Dirk W Lachenmeier, Burkhard Made. Methadone substitution: medicolegal problems in Germany. Forensic science international. vol 133. issue 1-2. 2003-07-29. PMID:12742698. |
in most of the cases, up to five additional drugs were also being taken: benzodiazepines (61%), ethanol (40%), morphine (39%), cannabinoids (35%), cocaine (28%), anti-depressants (3%), and amphetamines (2%). |
2003-07-29 |
2023-08-12 |
Not clear |
| Ildiko Racz, Andras Bilkei-Gorzo, Zsuzsanna E Toth, Kerstin Michel, Miklós Palkovits, Andreas Zimme. A critical role for the cannabinoid CB1 receptors in alcohol dependence and stress-stimulated ethanol drinking. The Journal of neuroscience : the official journal of the Society for Neuroscience. vol 23. issue 6. 2003-04-17. PMID:12657705. |
a critical role for the cannabinoid cb1 receptors in alcohol dependence and stress-stimulated ethanol drinking. |
2003-04-17 |
2023-08-12 |
mouse |
| Ildiko Racz, Andras Bilkei-Gorzo, Zsuzsanna E Toth, Kerstin Michel, Miklós Palkovits, Andreas Zimme. A critical role for the cannabinoid CB1 receptors in alcohol dependence and stress-stimulated ethanol drinking. The Journal of neuroscience : the official journal of the Society for Neuroscience. vol 23. issue 6. 2003-04-17. PMID:12657705. |
here we show that ethanol withdrawal symptoms were completely absent in cannabinoid cb1 receptor-deficient mice, although acute effects of ethanol and ethanol tolerance and preference were basically normal. |
2003-04-17 |
2023-08-12 |
mouse |
| Cory S Freedland, Christopher T Whitlow, Hilary R Smith, Linda J Porrin. Functional consequences of the acute administration of the cannabinoid receptor antagonist, SR141716A, in cannabinoid-naive and -tolerant animals: a quantitative 2-[14C]deoxyglucose study. Brain research. vol 962. issue 1-2. 2003-04-01. PMID:12543468. |
administration of the cannabinoid antagonist, sr141716a, markedly reduces intake of sucrose solutions, food pellets, and ethanol. |
2003-04-01 |
2023-08-12 |
rat |
| Lei Wang, Jie Liu, Judith Harvey-White, Andreas Zimmer, George Kuno. Endocannabinoid signaling via cannabinoid receptor 1 is involved in ethanol preference and its age-dependent decline in mice. Proceedings of the National Academy of Sciences of the United States of America. vol 100. issue 3. 2003-03-18. PMID:12538878. |
endocannabinoid signaling via cannabinoid receptor 1 is involved in ethanol preference and its age-dependent decline in mice. |
2003-03-18 |
2023-08-12 |
mouse |
| Lei Wang, Jie Liu, Judith Harvey-White, Andreas Zimmer, George Kuno. Endocannabinoid signaling via cannabinoid receptor 1 is involved in ethanol preference and its age-dependent decline in mice. Proceedings of the National Academy of Sciences of the United States of America. vol 100. issue 3. 2003-03-18. PMID:12538878. |
cannabinoids and ethanol can activate the same reward pathways, which could suggest endocannabinoid involvement in the rewarding effects of ethanol. |
2003-03-18 |
2023-08-12 |
mouse |
| Roberto Ciccocioppo, Lorena Antonelli, Michela Biondini, Marina Perfumi, Pierluigi Pompei, Maurizio Mass. Memory impairment following combined exposure to delta(9)-tetrahydrocannabinol and ethanol in rats. European journal of pharmacology. vol 449. issue 3. 2003-02-13. PMID:12167466. |
since both ethanol and cannabinoids are known to impair learning and memory, the present study investigated in rats the effects of combined exposure to ethanol and delta(9)-tetrahydrocannabinol (thc) in a memory task, the object recognition test. |
2003-02-13 |
2023-08-12 |
rat |
| Sara González, Maria Grazia Cascio, Javier Fernández-Ruiz, Filomena Fezza, Vincenzo Di Marzo, José A Ramo. Changes in endocannabinoid contents in the brain of rats chronically exposed to nicotine, ethanol or cocaine. Brain research. vol 954. issue 1. 2003-01-24. PMID:12393235. |
changes in endocannabinoid contents in the brain of rats chronically exposed to nicotine, ethanol or cocaine. |
2003-01-24 |
2023-08-12 |
rat |
| Sara González, Maria Grazia Cascio, Javier Fernández-Ruiz, Filomena Fezza, Vincenzo Di Marzo, José A Ramo. Changes in endocannabinoid contents in the brain of rats chronically exposed to nicotine, ethanol or cocaine. Brain research. vol 954. issue 1. 2003-01-24. PMID:12393235. |
in the present study, we have extended this objective by examining changes in the brain contents of arachidonoylethanolamide (aea) and 2-arachidonoyl-glycerol (2-ag), the endogenous ligands for cannabinoid receptors, in animals chronically exposed to cocaine, nicotine or ethanol. |
2003-01-24 |
2023-08-12 |
rat |