| Publication |
Sentence |
Publish Date |
Extraction Date |
Species |
| Haiyin Wu, Brian O'Neill, Dawn D Han, Keerthi Thirtamara-Rajamani, Yanlin Wang, Howard H G. Restoration of cocaine stimulation and reward by reintroducing wild type dopamine transporter in adult knock-in mice with a cocaine-insensitive dopamine transporter. Neuropharmacology. vol 86. 2015-07-14. PMID:24835281. |
in summary, cocaine induced locomotion and reward can be restored in fully developed dat-ci mice, and cocaine inhibition of dat expressed in dopaminergic neurons originated from the ventral midbrain mediates cocaine reward and stimulation. |
2015-07-14 |
2023-08-13 |
mouse |
| Stephanie Kullmann, Martin Heni, Katarzyna Linder, Stephan Zipfel, Hans-Ulrich Häring, Ralf Veit, Andreas Fritsche, Hubert Preiss. Resting-state functional connectivity of the human hypothalamus. Human brain mapping. vol 35. issue 12. 2015-07-13. PMID:25131690. |
our results indicate that the mh and lh network are tapped into different parts of the dopaminergic circuitry of the brain, potentially modulating food reward based on the functional connections to the ventral and dorsal striatum, respectively. |
2015-07-13 |
2023-08-13 |
human |
| Howard L Fields, Elyssa B Margoli. Understanding opioid reward. Trends in neurosciences. vol 38. issue 4. 2015-07-10. PMID:25637939. |
systemic opioid reward requires mop receptor function in the midbrain ventral tegmental area (vta) which contains dopaminergic neurons. |
2015-07-10 |
2023-08-13 |
Not clear |
| Howard L Fields, Elyssa B Margoli. Understanding opioid reward. Trends in neurosciences. vol 38. issue 4. 2015-07-10. PMID:25637939. |
vta dopaminergic neurons are implicated in various aspects of reward including reward prediction error, working memory, and incentive salience. |
2015-07-10 |
2023-08-13 |
Not clear |
| Kenneth Blum, Marlene Oscar-Berman, Zsolt Demetrovics, Debmalya Barh, Mark S Gol. Genetic Addiction Risk Score (GARS): molecular neurogenetic evidence for predisposition to Reward Deficiency Syndrome (RDS). Molecular neurobiology. vol 50. issue 3. 2015-07-08. PMID:24878765. |
we have published extensively on the neurogenetics of brain reward systems with reference to the genes related to dopaminergic function in particular. |
2015-07-08 |
2023-08-13 |
Not clear |
| Thore Apitz, Nico Bunzec. Early effects of reward anticipation are modulated by dopaminergic stimulation. PloS one. vol 9. issue 10. 2015-06-10. PMID:25285436. |
while the mesolimbic system, including dopaminergic midbrain, ventral striatum and prefrontal cortex have long been associated with reward processing, recent studies also indicate a prominent role of early visual brain regions. |
2015-06-10 |
2023-08-13 |
human |
| Miklós Sárvári, Levente Deli, Pál Kocsis, László Márk, Gábor Maász, Erik Hrabovszky, Imre Kalló, Dávid Gajári, Csaba Vastagh, Balázs Sümegi, Károly Tihanyi, Zsolt Liposit. Estradiol and isotype-selective estrogen receptor agonists modulate the mesocortical dopaminergic system in gonadectomized female rats. Brain research. vol 1583. 2015-06-05. PMID:24976584. |
the mesocortical dopaminergic pathway projecting from the ventral tegmental area (vta) to the prefrontal cortex (pfc) contributes to the processing of reward signals. |
2015-06-05 |
2023-08-13 |
rat |
| Nathalie Wrobel, Katja Wiech, Katarina Forkmann, Christoph Ritter, Ulrike Binge. Haloperidol blocks dorsal striatum activity but not analgesia in a placebo paradigm. Cortex; a journal devoted to the study of the nervous system and behavior. vol 57. 2015-06-04. PMID:24796219. |
our study therefore suggests that dopaminergic neurotransmission might not be causally involved in placebo analgesia but is related to phenomena associated with placebo analgesia such as reward processing and learning. |
2015-06-04 |
2023-08-13 |
human |
| Daniel G Dillon, Ian G Dobbins, Diego A Pizzagall. Weak reward source memory in depression reflects blunted activation of VTA/SN and parahippocampus. Social cognitive and affective neuroscience. vol 9. issue 10. 2015-06-03. PMID:24078019. |
reward responses in the medial temporal lobes and dopaminergic midbrain boost episodic memory formation in healthy adults, and weak memory for emotionally positive material in depression suggests this mechanism may be dysfunctional in major depressive disorder (mdd). |
2015-06-03 |
2023-08-12 |
human |
| Daniel G Dillon, Ian G Dobbins, Diego A Pizzagall. Weak reward source memory in depression reflects blunted activation of VTA/SN and parahippocampus. Social cognitive and affective neuroscience. vol 9. issue 10. 2015-06-03. PMID:24078019. |
consistent with predictions, controls also showed a stronger encoding response to reward tokens vs zero tokens in the right parahippocampus and dopaminergic midbrain, whereas the mdd group showed the opposite pattern-stronger responses to zero vs reward tokens-in these regions. |
2015-06-03 |
2023-08-12 |
human |
| Daniel G Dillon, Ian G Dobbins, Diego A Pizzagall. Weak reward source memory in depression reflects blunted activation of VTA/SN and parahippocampus. Social cognitive and affective neuroscience. vol 9. issue 10. 2015-06-03. PMID:24078019. |
differential activation of the dopaminergic midbrain by reward vs zero tokens was positively correlated with the reward source memory advantage in controls, but not depressed participants. |
2015-06-03 |
2023-08-12 |
human |
| Louise P Kirsch, Kelvin Dawson, Emily S Cros. Dance experience sculpts aesthetic perception and related brain circuits. Annals of the New York Academy of Sciences. vol 1337. 2015-06-01. PMID:25773627. |
moreover, brain regions involved in mediating the aesthetic response shift from subcortical regions associated with dopaminergic reward processing to posterior temporal regions involved in processing multisensory integration, emotion, and biological motion. |
2015-06-01 |
2023-08-13 |
human |
| Kathrin Koch, Oana Georgiana Rus, Tim Jonas Reeß, Claudia Schachtzabel, Gerd Wagner, C Christoph Schultz, Christian Sorg, Ralf G M Schlösse. Functional connectivity and grey matter volume of the striatum in schizophrenia. The British journal of psychiatry : the journal of mental science. vol 205. issue 3. 2015-05-29. PMID:25012683. |
alterations in the dopaminergic reward system, predominantly the striatum, constitute core characteristics of schizophrenia. |
2015-05-29 |
2023-08-13 |
Not clear |
| Esther Aarts, Abraham A M Nusselein, Peter Smittenaar, Rick C Helmich, Bastiaan R Bloem, Roshan Cool. Greater striatal responses to medication in Parkinson׳s disease are associated with better task-switching but worse reward performance. Neuropsychologia. vol 62. 2015-05-19. PMID:24912070. |
dopaminergic medication in parkinson's disease has been proposed to improve cognitive processing by modulating the severely depleted dorsal striatum, while impairing reward processing by modulating the relatively intact ventral striatum. |
2015-05-19 |
2023-08-13 |
Not clear |
| Juan Pérez-Fernández, Marcus Stephenson-Jones, Shreyas M Suryanarayana, Brita Robertson, Sten Grillne. Evolutionarily conserved organization of the dopaminergic system in lamprey: SNc/VTA afferent and efferent connectivity and D2 receptor expression. The Journal of comparative neurology. vol 522. issue 17. 2015-05-19. PMID:24942187. |
the dopaminergic system influences motor behavior, signals reward and novelty, and is an essential component of the basal ganglia in all vertebrates including the lamprey, one of the phylogenetically oldest vertebrates. |
2015-05-19 |
2023-08-13 |
Not clear |
| Thomas Goschke, Annette Bolt. Emotional modulation of control dilemmas: the role of positive affect, reward, and dopamine in cognitive stability and flexibility. Neuropsychologia. vol 62. 2015-05-19. PMID:25068705. |
finally, we critically review evidence for the popular hypothesis that effects of positive affect may be mediated by dopaminergic modulations of neural processing in prefrontal and striatal brain circuits, and we refine this "dopamine hypothesis of positive affect" by specifying distinct mechanisms by which dopamine may mediate effects of positive affect and reward on cognitive control. |
2015-05-19 |
2023-08-13 |
Not clear |
| Chun-Ling Wei, Zhi-Qiang Liu, Yi-Hui Liu, Wei Re. [High-frequency stimulation on cell soma induces potentiation of intrinsic excitability in VTA dopaminergic neurons]. Sheng li xue bao : [Acta physiologica Sinica]. vol 65. issue 1. 2015-05-16. PMID:23426514. |
the plasticity of vta dopaminergic neurons directly influences actions of other regions of the reward system. |
2015-05-16 |
2023-08-12 |
Not clear |
| Juan-Carlos Bustamante, Alfonso Barrós-Loscertales, Víctor Costumero, Paola Fuentes-Claramonte, Patricia Rosell-Negre, Noelia Ventura-Campos, Juan-José Llopis, César Ávil. Abstinence duration modulates striatal functioning during monetary reward processing in cocaine patients. Addiction biology. vol 19. issue 5. 2015-05-11. PMID:23445167. |
pre-clinical and clinical studies in cocaine addiction highlight alterations in the striatal dopaminergic reward system that subserve maintenance of cocaine use. |
2015-05-11 |
2023-08-12 |
Not clear |
| Gordon B Feld, Luciana Besedovsky, Kosuke Kaida, Thomas F Münte, Jan Bor. Dopamine D2-like receptor activation wipes out preferential consolidation of high over low reward memories during human sleep. Journal of cognitive neuroscience. vol 26. issue 10. 2015-05-11. PMID:24669794. |
however, it is unclear whether preferential consolidation of memories associated with reward requires the reactivation of dopaminergic circuitry known to mediate reward effects at encoding. |
2015-05-11 |
2023-08-12 |
human |
| Gordon B Feld, Luciana Besedovsky, Kosuke Kaida, Thomas F Münte, Jan Bor. Dopamine D2-like receptor activation wipes out preferential consolidation of high over low reward memories during human sleep. Journal of cognitive neuroscience. vol 26. issue 10. 2015-05-11. PMID:24669794. |
these results are consistent with the concept that preferential consolidation of reward-associated memories relies on hippocampus-driven reactivation within the dopaminergic reward system during sleep, whereby during sleep reward contingencies are fed back to the hippocampus to strengthen specific memories, possibly, through dopaminergic facilitation of long-term potentiation. |
2015-05-11 |
2023-08-12 |
human |