All Relations between feeding and insulin

Publication Sentence Publish Date Extraction Date Species
A Inui, M Okita, T Inoue, N Sakatani, M Oya, H Morioka, T Ogawa, N Mizuno, S Bab. Mechanism of actions of cholecystokinin octapeptide on food intake and insulin and pancreatic polypeptide release in the dog. Peptides. vol 9. issue 5. 1989-05-26. PMID:3072534. cck-8 was less effective in increasing insulin and pp concentrations than in reducing feeding. 1989-05-26 2023-08-11 dog
S B Reiser, H F Weiser, V Schusdziarra, J R Siewer. Effect of pacing on small intestinal motor activity and hormonal response in dogs. Digestive diseases and sciences. vol 34. issue 4. 1989-05-19. PMID:2649321. our study showed that pacing of the loop results in a significant reduction of the motility of the loop in the overnight fasted state (up to 39%), during loop feeding (up to 55%), and oral feeding (up to 39%); a similar reduction of the motility of the remaining small intestine (up to 43%); and a significant postprandial increase of insulin (9.0 microu/ml) and decrease of glucagon (94 pg/ml). 1989-05-19 2023-08-11 Not clear
C Y Guezennec, P Satabin, F Duforez, D Merino, F Peronnet, J Kozie. Oxidation of corn starch, glucose, and fructose ingested before exercise. Medicine and science in sports and exercise. vol 21. issue 1. 1989-05-11. PMID:2494417. plasma glucose and insulin concentrations significantly increased in response to g and s feeding. 1989-05-11 2023-08-11 human
N H Rubin, G Alinder, W J Rietveld, P L Rayford, J C Thompso. Restricted feeding schedules alter the circadian rhythms of serum insulin and gastric inhibitory polypeptide. Regulatory peptides. vol 23. issue 3. 1989-05-10. PMID:3070645. restricted feeding schedules alter the circadian rhythms of serum insulin and gastric inhibitory polypeptide. 1989-05-10 2023-08-11 rat
N H Rubin, G Alinder, W J Rietveld, P L Rayford, J C Thompso. Restricted feeding schedules alter the circadian rhythms of serum insulin and gastric inhibitory polypeptide. Regulatory peptides. vol 23. issue 3. 1989-05-10. PMID:3070645. insulin and gastric inhibitory polypeptide (gip) have a circadian rhythm of secretion that is altered by various feeding schedules. 1989-05-10 2023-08-11 rat
N H Rubin, G Alinder, W J Rietveld, P L Rayford, J C Thompso. Restricted feeding schedules alter the circadian rhythms of serum insulin and gastric inhibitory polypeptide. Regulatory peptides. vol 23. issue 3. 1989-05-10. PMID:3070645. circadian rhythms occurred for insulin with all feeding schedules and for gip with all schedules except fasted rats. 1989-05-10 2023-08-11 rat
N H Rubin, G Alinder, W J Rietveld, P L Rayford, J C Thompso. Restricted feeding schedules alter the circadian rhythms of serum insulin and gastric inhibitory polypeptide. Regulatory peptides. vol 23. issue 3. 1989-05-10. PMID:3070645. this finding implies an endogenous insulin rhythm, whereas food intake controls gip secretion. 1989-05-10 2023-08-11 rat
C E Barnett, D R Talbert, D A Vanderweel. Phentolamine stimulates insulin release and glucose clearance and suppresses food ingestion. Physiology & behavior. vol 44. issue 4-5. 1989-05-05. PMID:2853389. the final experiments subdivided the 1-hr feeding period into three 20-min segments and, in separate animals, food intake or plasma insulin and glucose changes were assessed. 1989-05-05 2023-08-11 human
C E Barnett, D R Talbert, D A Vanderweel. Phentolamine stimulates insulin release and glucose clearance and suppresses food ingestion. Physiology & behavior. vol 44. issue 4-5. 1989-05-05. PMID:2853389. pta-injected subjects showed an immediate modest enhancement of insulin release during the first 20-min feeding segment following injection, compared to controls, while blood glucose levels decreased but never differed reliably between groups. 1989-05-05 2023-08-11 human
W J Kuenzel, J McMurtr. Neuropeptide Y: brain localization and central effects on plasma insulin levels in chicks. Physiology & behavior. vol 44. issue 4-5. 1989-05-05. PMID:3070587. since the immunocytochemical study showed that npy was localized in brain structures known to alter food intake and the compound is a member of the pancreatic polypeptide family, a second study was designed to determine if the neuropeptide altered plasma concentrations of insulin, glucagon and glucose following intracerebroventricular administration. 1989-05-05 2023-08-11 Not clear
D A Vanderweele, E Haraczkiewicz, J R Vassell. Tolbutamide affects food ingestion in a manner consistent with its glycemic effects in the rat. Physiology & behavior. vol 44. issue 4-5. 1989-05-05. PMID:3070588. the sulphonylurea tolbutamide possesses the ability to stimulate insulin release, produce hypoglycemia and increase food intake; however, no study has investigated the effects of moderate doses which do not produce frank hypoglycemia. 1989-05-05 2023-08-11 rat
D A Vanderweele, E Haraczkiewicz, J R Vassell. Tolbutamide affects food ingestion in a manner consistent with its glycemic effects in the rat. Physiology & behavior. vol 44. issue 4-5. 1989-05-05. PMID:3070588. thus, the present studies suggest that when endogenous insulin levels are modestly raised by tolbutamide, such that only moderate reductions of circulating glucose were observed, decreases in food intake occur. 1989-05-05 2023-08-11 rat
L H Storlien, A W Thorburn, G A Smythe, A B Jenkins, D J Chisholm, E W Kraege. Effect of d-fenfluramine on basal glucose turnover and fat-feeding-induced insulin resistance in rats. Diabetes. vol 38. issue 4. 1989-05-04. PMID:2647557. we report that chronic d-fenfluramine (5 mg.kg-1.day-1) ameliorates insulin resistance induced by high-fat feeding. 1989-05-04 2023-08-11 rat
L H Storlien, A W Thorburn, G A Smythe, A B Jenkins, D J Chisholm, E W Kraege. Effect of d-fenfluramine on basal glucose turnover and fat-feeding-induced insulin resistance in rats. Diabetes. vol 38. issue 4. 1989-05-04. PMID:2647557. impaired suppression of hepatic glucose output by insulin, caused by fat feeding, was totally reversed by d-fenfluramine. 1989-05-04 2023-08-11 rat
D A Czec. Effect of insulin and 2-deoxy-D-glucose on feeding and plasma glucose levels in the spiny mouse. Physiology & behavior. vol 43. issue 6. 1989-05-04. PMID:3070582. adult male spiny mice (acomys cahirinus) were challenged with 2-deoxy-d-glucose (2-dg) or regular insulin, and food intake or plasma glucose concentration was measured. 1989-05-04 2023-08-11 mouse
D A Czec. Effect of insulin and 2-deoxy-D-glucose on feeding and plasma glucose levels in the spiny mouse. Physiology & behavior. vol 43. issue 6. 1989-05-04. PMID:3070582. in contrast, regular insulin injections (1-50 u/kg) stimulated a modest, but significant increase in feeding, which was apparent within 2 hr at a low dose of 1 u/kg. 1989-05-04 2023-08-11 mouse
G D Means, J M Burn. Glucagon auto-immunization fails to stimulate food intake or growth in young rabbits. Comparative biochemistry and physiology. A, Comparative physiology. vol 91. issue 3. 1989-05-02. PMID:2906842. no differences between the controls and auto-immunized group were found at any time for weekly weight gain, food intake, blood glucose or insulin concentrations. 1989-05-02 2023-08-11 Not clear
C Larue-Achagiotis, M Goubern, M C Laur. Concomitant food intake and adipose tissue responses under chronic insulin infusion in rats. Physiology & behavior. vol 44. issue 1. 1989-04-28. PMID:3070583. concomitant food intake and adipose tissue responses under chronic insulin infusion in rats. 1989-04-28 2023-08-11 rat
C Larue-Achagiotis, M Goubern, M C Laur. Concomitant food intake and adipose tissue responses under chronic insulin infusion in rats. Physiology & behavior. vol 44. issue 1. 1989-04-28. PMID:3070583. body weight (bw), food intake (fi), and activity of white adipose tissue (wat) and brown adipose tissue (bat) were studied in adult male rats under chronic insulin infusion. 1989-04-28 2023-08-11 rat
J Licinio-Paixa. Hyperinsulinemia; a mediator of decreased food intake and weight loss in anorexia nervosa and major depression. Medical hypotheses. vol 28. issue 2. 1989-04-21. PMID:2648126. while it should be kept in mind that the etiology of those diseases is still unclear, it seems reasonable to propose that the maintenance of anorectic behavior in the eating disorders as well as the decreased food intake of major depression, leading to continued weight loss seen in both conditions, are either caused or mediated by insulin in levels which are elevated but insufficient to cause hypoglycemia. 1989-04-21 2023-08-11 Not clear