| Publication |
Sentence |
Publish Date |
Extraction Date |
Species |
| K Ohshima, T Onai, S Okada, M Umahara, M Sato, Y Nakamura, M Mori, I Kobayashi, T Saka. Medialbasal hypothalamic deafferentation modulates feeding response to insulin in rats. Physiology & behavior. vol 53. issue 5. 1993-07-12. PMID:8511201. |
insulin is essential in developing and maintaining obesity, but the role of insulin in food intake in hypothalamic obesity is still unclear. |
1993-07-12 |
2023-08-12 |
rat |
| K Ohshima, T Onai, S Okada, M Umahara, M Sato, Y Nakamura, M Mori, I Kobayashi, T Saka. Medialbasal hypothalamic deafferentation modulates feeding response to insulin in rats. Physiology & behavior. vol 53. issue 5. 1993-07-12. PMID:8511201. |
the present study demonstrated that exogenous insulin increased food intake dose relatedly in mbh deafferented diabetic rats without developing hypoglycemia. |
1993-07-12 |
2023-08-12 |
rat |
| K Ohshima, T Onai, S Okada, M Umahara, M Sato, Y Nakamura, M Mori, I Kobayashi, T Saka. Medialbasal hypothalamic deafferentation modulates feeding response to insulin in rats. Physiology & behavior. vol 53. issue 5. 1993-07-12. PMID:8511201. |
in contrast, in the mbh deafferented diabetic rats, insulin increased food intake in sow-related manner concomitant with a greater increased body weight gain than the sham-operated diabetic rats. |
1993-07-12 |
2023-08-12 |
rat |
| K Ohshima, T Onai, S Okada, M Umahara, M Sato, Y Nakamura, M Mori, I Kobayashi, T Saka. Medialbasal hypothalamic deafferentation modulates feeding response to insulin in rats. Physiology & behavior. vol 53. issue 5. 1993-07-12. PMID:8511201. |
these data indicate that insulin action on food intake mediated through the central nervous system is modulated by mbh deafferentation. |
1993-07-12 |
2023-08-12 |
rat |
| T D Fahey, K Hoffman, W Colvin, G Laute. The effects of intermittent liquid meal feeding on selected hormones and substrates during intense weight training. International journal of sport nutrition. vol 3. issue 1. 1993-06-29. PMID:8499939. |
serum insulin increased from 12.2 +/- 1.2 and 11.2 +/- 1.3 before feeding to 37.2 +/- 4.8 and 45.0 +/- 5.0 mu.ml-1 during exercise in mw and m, respectively, and remained elevated for 120 min. |
1993-06-29 |
2023-08-12 |
human |
| T D Fahey, K Hoffman, W Colvin, G Laute. The effects of intermittent liquid meal feeding on selected hormones and substrates during intense weight training. International journal of sport nutrition. vol 3. issue 1. 1993-06-29. PMID:8499939. |
perceived exertion during exercise was 8.5 +/- 0.16 for mw and 8.3 +/- 0.18 for w. feeding a liquid meal before and during weight training exercise can increase serum insulin and maintain blood glucose for a prolonged period. |
1993-06-29 |
2023-08-12 |
human |
| G B Smyth, D W Young, S H Dura. Maturation of insulin and glucose responses to normal feeding in foals. Australian veterinary journal. vol 70. issue 4. 1993-06-17. PMID:8494520. |
maturation of insulin and glucose responses to normal feeding in foals. |
1993-06-17 |
2023-08-12 |
Not clear |
| M Egawa, S Inoue, S Satoh, Y Takamura, K Nagai, H Nakagaw. Acute and chronic effects of VMH lesions on circadian rhythms in food intake and metabolites. Brain research bulletin. vol 31. issue 3-4. 1993-06-11. PMID:8490728. |
in the static phase, 12 weeks after surgery, the difference of food intake between light and dark cycle appeared in vmh-lesioned rats, but the loss of rhythmicity for serum glucose, insulin, and triglyceride remained. |
1993-06-11 |
2023-08-12 |
rat |
| M Egawa, S Inoue, S Satoh, Y Takamura, K Nagai, H Nakagaw. Acute and chronic effects of VMH lesions on circadian rhythms in food intake and metabolites. Brain research bulletin. vol 31. issue 3-4. 1993-06-11. PMID:8490728. |
vmh lesions thus abolished circadian rhythmicity in serum insulin, glucose, and triglyceride for a long period; however, the disturbed rhythmicity of food intake was reversible. |
1993-06-11 |
2023-08-12 |
rat |
| G A Bra. The nutrient balance hypothesis: peptides, sympathetic activity, and food intake. Annals of the New York Academy of Sciences. vol 676. 1993-06-10. PMID:8489134. |
galanin, neuropeptide y, opioid peptides, growth hormone releasing hormone and desacetyl-msh increase food intake whereas insulin, glucagon, cholecystokinin, anorectin, corticotropin releasing hormone, neurotensin, bombesin, enterostatin, cyclo-his-pro and thyrotropin-releasing hormone reduce food intake. |
1993-06-10 |
2023-08-12 |
Not clear |
| G A Bra. The nutrient balance hypothesis: peptides, sympathetic activity, and food intake. Annals of the New York Academy of Sciences. vol 676. 1993-06-10. PMID:8489134. |
insulin reduces food intake in animals fed a high carbohydrate diet, but not when fed a high fat diet. |
1993-06-10 |
2023-08-12 |
Not clear |
| J Rouru, U Pesonen, K Isaksson, R Huupponen, M Koul. Effect of chronic treatment with TFMPP, a 5-HT1 receptor agonist, on food intake, weight gain, plasma insulin and neuropeptide mRNA expression in obese Zucker rats. European journal of pharmacology. vol 234. issue 2-3. 1993-06-03. PMID:8482326. |
effect of chronic treatment with tfmpp, a 5-ht1 receptor agonist, on food intake, weight gain, plasma insulin and neuropeptide mrna expression in obese zucker rats. |
1993-06-03 |
2023-08-12 |
rat |
| P Holtenius, G Olsson, C Björkma. Periparturient concentrations of insulin glucagon and ketone bodies in dairy cows fed two different levels of nutrition and varying concentrate/roughage ratios. Zentralblatt fur Veterinarmedizin. Reihe A. vol 40. issue 2. 1993-05-25. PMID:8480460. |
before calving the feeding regimen had a very strong influence on the basal insulin level. |
1993-05-25 |
2023-08-12 |
Not clear |
| U H Malabu, S J Cotton, Y T Kruszynska, G William. Acute hyperinsulinemia increases neuropeptide Y concentrations in the hypothalamic arcuate nucleus of fasted rats. Life sciences. vol 52. issue 17. 1993-05-03. PMID:8464342. |
neuropeptide y, a major hypothalamic peptide, stimulates feeding, insulin secretion and weight gain when injected intrahypothalamically. |
1993-05-03 |
2023-08-12 |
rat |
| M Wozniak, B Rydzewski, S P Baker, M K Raizad. The cellular and physiological actions of insulin in the central nervous system. Neurochemistry international. vol 22. issue 1. 1993-04-06. PMID:8443563. |
recognized physiological effects of insulin in the central nervous system (cns) include regulation of food intake, control of glucose uptake and trophic actions on neuronal and glial cells. |
1993-04-06 |
2023-08-12 |
Not clear |
| R B Lyon, D M Vinc. Nutrition management of insulin-dependent diabetes mellitus in adults: review by the diabetes care and education dietetic practice group. Journal of the American Dietetic Association. vol 93. issue 3. 1993-04-01. PMID:8267691. |
treatment requires the use of exogenous insulin coordinated with food intake and physical activity. |
1993-04-01 |
2023-08-12 |
Not clear |
| A Inui, M Okita, M Miura, Y Hirosue, N Mizuno, S Baba, M Kasug. Plasma and cerebroventricular fluid levels of pancreatic polypeptide in the dog: effects of feeding, insulin-induced hypoglycemia, and physical exercise. Endocrinology. vol 132. issue 3. 1993-04-01. PMID:8440184. |
we conclude that 1) pp and insulin in csf do not appear to play a major role in the short term regulation of food intake and acute changes in energy metabolism; and 2) pp, probably after entering the brain, may modulate brain function in such physiological situations as strenuous exercise. |
1993-04-01 |
2023-08-12 |
dog |
| M R Lebowitz, S A Blumentha. The molar ratio of insulin to C-peptide. An aid to the diagnosis of hypoglycemia due to surreptitious (or inadvertent) insulin administration. Archives of internal medicine. vol 153. issue 5. 1993-03-24. PMID:8439228. |
hence, the molar ratio of insulin to c-peptide in peripheral venous blood (icpr) should be less than 1.0 during fasting and feeding, unless exogenous insulin is introduced into the systemic circulation. |
1993-03-24 |
2023-08-12 |
Not clear |
| L H Storlien, N D Oakes, D A Pan, M Kusunoki, A B Jenkin. Syndromes of insulin resistance in the rat. Inducement by diet and amelioration with benfluorex. Diabetes. vol 42. issue 3. 1993-03-16. PMID:8432416. |
compared with starch feeding, fructose and fat feeding significantly impaired insulin action at the whole-body level (-46% and -41%, respectively, both p < 0.001), as well as in individual skeletal muscles. |
1993-03-16 |
2023-08-12 |
human |
| M B Davidson, D Garve. Studies on mechanisms of hepatic insulin resistance in cafeteria-fed rats. The American journal of physiology. vol 264. issue 1 Pt 1. 1993-03-08. PMID:8430783. |
after 3 days of cafeteria feeding the rats were obese, manifested a small but significant decrease in fasting glucose levels, and showed no change in fasting insulin levels, basal hepatic glucose production (hgp), insulin binding to hepatic membranes, and glucose utilization during a euglycemic hyperinsulinemic clamp, but the rats did demonstrate an increased glucose disappearance rate associated with an enhanced insulin response to intra-arterial glucose and hepatic insulin resistance during the clamp. |
1993-03-08 |
2023-08-12 |
rat |