| Publication |
Sentence |
Publish Date |
Extraction Date |
Species |
| K Fukuo, T Nakahashi, S Nomura, S Hata, T Suhara, M Shimizu, M Tamatani, S Morimoto, Y Kitamura, T Ogihar. Possible participation of Fas-mediated apoptosis in the mechanism of atherosclerosis. Gerontology. vol 43 Suppl 1. 1997-08-04. PMID:9187937. |
to clarify the mechanisms that trigger apoptosis in atherosclerotic lesions, we examined whether cytokines released from macrophages can modulate fas, a death signal, in cultured human vsmcs. |
1997-08-04 |
2023-08-12 |
human |
| J D McLeod, L S Walker, R V Parry, S G Ward, D M Sanso. Activation of phosphatidylinositol 3-kinase following Fas engagement is not required for Fas-mediated apoptosis in Jurkat T cells. Biochemical Society transactions. vol 25. issue 2. 1997-08-04. PMID:9191285. |
activation of phosphatidylinositol 3-kinase following fas engagement is not required for fas-mediated apoptosis in jurkat t cells. |
1997-08-04 |
2023-08-12 |
Not clear |
| L Behrens, A Bender, M A Johnson, R Hohlfel. Cytotoxic mechanisms in inflammatory myopathies. Co-expression of Fas and protective Bcl-2 in muscle fibres and inflammatory cells. Brain : a journal of neurology. vol 120 ( Pt 6). 1997-08-01. PMID:9217678. |
expression of the fas 'death receptor', fas (cd95/apo-1) renders cells susceptible to programmed cell death ('apoptosis'), whereas bcl-2 protects cells from apoptosis. |
1997-08-01 |
2023-08-12 |
Not clear |
| L Behrens, A Bender, M A Johnson, R Hohlfel. Cytotoxic mechanisms in inflammatory myopathies. Co-expression of Fas and protective Bcl-2 in muscle fibres and inflammatory cells. Brain : a journal of neurology. vol 120 ( Pt 6). 1997-08-01. PMID:9217678. |
our results suggest that, although fas expression confers susceptibility to fas-mediated apoptosis, fas-expressing muscle fibres and inflammatory cells are protected by the anti-apoptotic protein bcl-2. |
1997-08-01 |
2023-08-12 |
Not clear |
| J Wang, N Nonomura, N Ichimaru, H Azuma, M Hatori, Y Kokado, K Matsumiya, T Miki, S Takahara, A Okuyam. Expression of Fas and Fas ligand in renal grafts with acute and chronic rejection in the rat model. Journal of interferon & cytokine research : the official journal of the International Society for Interferon and Cytokine Research. vol 17. issue 6. 1997-07-31. PMID:9198004. |
our data demonstrated that the fas system might play an important role in acute and chronic rejection by causing apoptosis, and the spleen may eliminate the lymphocytes strongly expressing fas ligand after completion of the acute rejection. |
1997-07-31 |
2023-08-12 |
rat |
| C Dong, G L Winters, J E Wilson, B M McManu. Enhanced lymphocyte longevity and absence of proliferation and lymphocyte apoptosis in Quilty effects of human heart allografts. The American journal of pathology. vol 151. issue 1. 1997-07-31. PMID:9212738. |
twenty-six paraffin-embedded endomyocardial biopsy specimens with qe (five qe as and twenty-one qe bs) from twenty-two cardiac allografts were studied by immunohistochemistry for expression of bcl-2, fas antigen, proliferating cell nuclear antigen (pcna), perforin, t cells (uchl-1), macrophages (cd68), and apoptosis by in situ terminal deoxyribonucleotide transferase (tdt)-mediated dutp nick end labeling (tunel). |
1997-07-31 |
2023-08-12 |
human |
| C Dong, G L Winters, J E Wilson, B M McManu. Enhanced lymphocyte longevity and absence of proliferation and lymphocyte apoptosis in Quilty effects of human heart allografts. The American journal of pathology. vol 151. issue 1. 1997-07-31. PMID:9212738. |
1) apoptosis does not occur in lymphocytes in qe where enhanced bcl-2 (apoptosis inhibitor) and fas antigen (apoptosis inducer) are expressed. |
1997-07-31 |
2023-08-12 |
human |
| E J Fuchs, K A McKenna, A Bed. p53-dependent DNA damage-induced apoptosis requires Fas/APO-1-independent activation of CPP32beta. Cancer research. vol 57. issue 13. 1997-07-25. PMID:9205051. |
we investigated whether p53-dependent apoptosis requires activation of cpp32beta (caspase 3), a cysteine protease that has been found to mediate apoptosis in response to ligation of the fas molecule or to granzyme b, a component of ctl lytic granules. |
1997-07-25 |
2023-08-12 |
mouse |
| E J Fuchs, K A McKenna, A Bed. p53-dependent DNA damage-induced apoptosis requires Fas/APO-1-independent activation of CPP32beta. Cancer research. vol 57. issue 13. 1997-07-25. PMID:9205051. |
we next examined whether the fas system is required for p53-dependent apoptosis and whether stimuli that induce activation of cpp32beta induce apoptosis in p53-deficient cells. |
1997-07-25 |
2023-08-12 |
mouse |
| E J Fuchs, K A McKenna, A Bed. p53-dependent DNA damage-induced apoptosis requires Fas/APO-1-independent activation of CPP32beta. Cancer research. vol 57. issue 13. 1997-07-25. PMID:9205051. |
thymocytes or activated t cells from fas-deficient mice were resistant to apoptosis induced by ligation of fas or cd3, respectively, but remained normally susceptible to irradiation. |
1997-07-25 |
2023-08-12 |
mouse |
| E J Fuchs, K A McKenna, A Bed. p53-dependent DNA damage-induced apoptosis requires Fas/APO-1-independent activation of CPP32beta. Cancer research. vol 57. issue 13. 1997-07-25. PMID:9205051. |
thymocytes from p53-deficient mice, although resistant to dna damage, remained sensitive to cpp32beta-mediated apoptosis induced by ligation of fas or cd3, or by exposure to cytotoxic t cells. |
1997-07-25 |
2023-08-12 |
mouse |
| J M Puck, M C Snelle. ALPS: an autoimmune human lymphoproliferative syndrome associated with abnormal lymphocyte apoptosis. Seminars in immunology. vol 9. issue 1. 1997-07-24. PMID:9106310. |
the cell surface receptor fas is an important mediator of lymphocyte apoptosis; defective fas expression causes accumulation of lymphocytes and autoimmune disease in mice. |
1997-07-24 |
2023-08-12 |
mouse |
| J M Puck, M C Snelle. ALPS: an autoimmune human lymphoproliferative syndrome associated with abnormal lymphocyte apoptosis. Seminars in immunology. vol 9. issue 1. 1997-07-24. PMID:9106310. |
apoptosis defects due to mutations of fas have also been found in a rare human autoimmune lymphoproliferative syndrome (alps). |
1997-07-24 |
2023-08-12 |
mouse |
| J M Puck, M C Snelle. ALPS: an autoimmune human lymphoproliferative syndrome associated with abnormal lymphocyte apoptosis. Seminars in immunology. vol 9. issue 1. 1997-07-24. PMID:9106310. |
all nine exhibited impaired lymphocyte apoptosis in vitro, and eight had heterozygous fas gene mutations. |
1997-07-24 |
2023-08-12 |
mouse |
| M Irmler, M Thome, M Hahne, P Schneider, K Hofmann, V Steiner, J L Bodmer, M Schröter, K Burns, C Mattmann, D Rimoldi, L E French, J Tschop. Inhibition of death receptor signals by cellular FLIP. Nature. vol 388. issue 6638. 1997-07-24. PMID:9217161. |
the widely expressed protein fas is a member of the tumour necrosis factor receptor family which can trigger apoptosis. |
1997-07-24 |
2023-08-12 |
human |
| M Irmler, M Thome, M Hahne, P Schneider, K Hofmann, V Steiner, J L Bodmer, M Schröter, K Burns, C Mattmann, D Rimoldi, L E French, J Tschop. Inhibition of death receptor signals by cellular FLIP. Nature. vol 388. issue 6638. 1997-07-24. PMID:9217161. |
flip(l) is expressed during the early stage of t-cell activation, but disappears when t cells become susceptible to fas ligand-mediated apoptosis. |
1997-07-24 |
2023-08-12 |
human |
| E Mita, N Hayash. [Apoptosis in human diseases: role of Fas system in liver cell injury by viral hepatitis]. Rinsho byori. The Japanese journal of clinical pathology. vol 45. issue 5. 1997-07-22. PMID:9170976. |
[apoptosis in human diseases: role of fas system in liver cell injury by viral hepatitis]. |
1997-07-22 |
2023-08-12 |
human |
| E Mita, N Hayash. [Apoptosis in human diseases: role of Fas system in liver cell injury by viral hepatitis]. Rinsho byori. The Japanese journal of clinical pathology. vol 45. issue 5. 1997-07-22. PMID:9170976. |
therefore, we examined the implication of the fas system-mediated apoptosis in the liver cell injury. |
1997-07-22 |
2023-08-12 |
human |
| M C Ruiz-Ruiz, M Izquierdo, G de Murcia, A López-Riva. Activation of protein kinase C attenuates early signals in Fas-mediated apoptosis. European journal of immunology. vol 27. issue 6. 1997-07-22. PMID:9209497. |
activation of protein kinase c (pkc) has been reported to inhibit fas (apo-1, cd95)-mediated apoptosis in different cellular systems. |
1997-07-22 |
2023-08-12 |
human |
| M C Ruiz-Ruiz, M Izquierdo, G de Murcia, A López-Riva. Activation of protein kinase C attenuates early signals in Fas-mediated apoptosis. European journal of immunology. vol 27. issue 6. 1997-07-22. PMID:9209497. |
human jurkat leukemic t cells express the fas antigen in the cell membrane and undergo apoptosis upon cross-linking by anti-fas monoclonal antibodies (mab). |
1997-07-22 |
2023-08-12 |
human |