| Publication |
Sentence |
Publish Date |
Extraction Date |
Species |
| M Gotoh, Y Yoshikawa, T Otani, T Kato, M Kobayashi, K Kato, M Saito, A Kondo, K Miyak. [Effects of alpha-1-blocking agent in the treatment of detrusor sphincter dyssynergia]. Nihon Hinyokika Gakkai zasshi. The japanese journal of urology. vol 81. issue 12. 1991-04-09. PMID:2292821. |
patients with cva and spinal cord injury with incomplete paralysis showed good response rates in subjective improvement, 69% and 60% respectively. |
1991-04-09 |
2023-08-11 |
Not clear |
| M Gotoh, Y Yoshikawa, T Otani, T Kato, M Kobayashi, K Kato, M Saito, A Kondo, K Miyak. [Effects of alpha-1-blocking agent in the treatment of detrusor sphincter dyssynergia]. Nihon Hinyokika Gakkai zasshi. The japanese journal of urology. vol 81. issue 12. 1991-04-09. PMID:2292821. |
however, those with spinal cord injury with complete paralysis showed a poor response (28% for c, 23% for th and 14% for l). |
1991-04-09 |
2023-08-11 |
Not clear |
| G Urca, R Urc. Neurotoxic effects of excitatory amino acids in the mouse spinal cord: quisqualate and kainate but not N-methyl-D-aspartate induce permanent neural damage. Brain research. vol 529. issue 1-2. 1991-03-19. PMID:1980847. |
the highest concentrations of these agonists produced, in some animals, a massive, non-selective destruction of neurons within the lumbar spinal cord, accompanied by complete paralysis of the hindlimbs. |
1991-03-19 |
2023-08-11 |
mouse |
| R Bakshi, A H Newman, A I Fade. Dynorphin A-(1-17) induces alterations in free fatty acids, excitatory amino acids, and motor function through an opiate-receptor-mediated mechanism. The Journal of neuroscience : the official journal of the Society for Neuroscience. vol 10. issue 12. 1991-02-20. PMID:1980130. |
the endogenous opioid dynorphin a-(1-17) (dyn a) has been implicated as a mediator of tissue damage after traumatic spinal cord injury (tsci) and causes hindlimb paralysis when administered intrathecally. |
1991-02-20 |
2023-08-11 |
Not clear |
| b' S Vlajkovi\\xc4\\x87, B D Jankovi\\xc4\\x8. Experimental epilepsy: electrically and chemically induced convulsions modulate experimental allergic encephalomyelitis and other immune inflammatory reactions in the rat. The International journal of neuroscience. vol 54. issue 1-2. 1991-02-08. PMID:2265961.' |
male wistar rats were exposed to electroconvulsive shock (ecs) and pentylentetrazol (ptz) for a period of 38 consecutive days; 21 days before and 17 days after immunization with guinea pig spinal cord in complete freund's adjuvant (cfa) ecs and ptz completely prevented the appearance of paralysis and decreased incidence and severity of lesions characteristic of experimental allergic encephalomyelitis, when compared to control sham-shocked and saline-treated rats. |
1991-02-08 |
2023-08-11 |
rat |
| V O Gardner, V J Caiozzo, S K Munden, R J Bridge. Excitotoxins can produce protein degradation in the spinal cord. Spine. vol 15. issue 9. 1991-01-29. PMID:1979692. |
overdistraction and derotation of the scoliotic spine during surgery represent potential complications that could lead to spinal cord dysfunction and paralysis. |
1991-01-29 |
2023-08-11 |
rat |
| P K Wong, E Floyd, P F Szure. High susceptibility of FVB/N mice to the paralytic disease induced by ts1, a mutant of Moloney murine leukemia virus TB. Virology. vol 180. issue 1. 1991-01-29. PMID:1984656. |
our studies show that there was enhanced replication of ts1 in all tissues of fvb/n mice and that the virus titer in the spinal cord was more than 10-fold higher in fvb/n than in balb/c mice by 30 days postinoculation, when the clinical signs of paralysis became evident in fvb/n mice. |
1991-01-29 |
2023-08-11 |
mouse |
| S R White, G K Samathanam, R M Bowker, M W Wessendor. Damage to bulbospinal serotonin-, tyrosine hydroxylase-, and TRH-containing axons occurs early in the development of experimental allergic encephalomyelitis in rats. Journal of neuroscience research. vol 27. issue 1. 1991-01-24. PMID:1701489. |
spinal cord axons immunoreactive for serotonin, catecholamines, or a thyrotropin-releasing hormone marker peptide were found to be markedly swollen and distorted by the earliest stage of detectable paralysis during eae development (the flaccid tail stage). |
1991-01-24 |
2023-08-11 |
rat |
| J H Frisbie, E J Aguiler. Chronic pain after spinal cord injury: an expedient diagnostic approach. Paraplegia. vol 28. issue 7. 1991-01-17. PMID:2250989. |
the reliability of distinguishing central, musculoskeletal, and syringomyelic pain by two points of history: (1) pain quality and (2) pain location relative to the level of paralysis in spinal cord injury patients was tested by (1) physical examination, and (2) by radiographic imaging. |
1991-01-17 |
2023-08-11 |
Not clear |
| Y Hiraizumi, E Fujimaki, T Tachikaw. Long-term morphology of spastic or flaccid muscles in spinal cord-transected rabbits. Clinical orthopaedics and related research. issue 260. 1990-12-14. PMID:2225635. |
rabbits were prepared by spinal cord transection at t10 (spastic paralysis) or by spinal cord removal below l7 (flaccid paralysis). |
1990-12-14 |
2023-08-11 |
Not clear |
| R Y Ki. Extradural spinal cord compression from metastatic tumor. Alabama medicine : journal of the Medical Association of the State of Alabama. vol 60. issue 1-2. 1990-12-07. PMID:2239623. |
early diagnosis is critical since onset of spinal cord injury may be sudden, often progressing to irreversible paralysis in a period of hours. |
1990-12-07 |
2023-08-11 |
Not clear |
| D Green, M Y Lee, A C Lim, J S Chmiel, M Vetter, T Pang, D Chen, L Fenton, G M Yarkony, P R Meye. Prevention of thromboembolism after spinal cord injury using low-molecular-weight heparin. Annals of internal medicine. vol 113. issue 8. 1990-10-18. PMID:2169216. |
to examine the safety and effectiveness of a low-molecular-weight heparin in the prevention of thromboembolism in patients with recent spinal cord injury and complete motor paralysis. |
1990-10-18 |
2023-08-11 |
Not clear |
| S Helweg-Larsen, B Rasmusson, P S Sørense. Recovery of gait after radiotherapy in paralytic patients with metastatic epidural spinal cord compression. Neurology. vol 40. issue 8. 1990-09-10. PMID:2381531. |
the response to radiation therapy seems to depend on the rate of loss of spinal cord function rather than the length of total paralysis. |
1990-09-10 |
2023-08-11 |
Not clear |
| D S Heffez, J Aryanpur, G M Hutchins, J M Freema. The paralysis associated with myelomeningocele: clinical and experimental data implicating a preventable spinal cord injury. Neurosurgery. vol 26. issue 6. 1990-08-08. PMID:2362676. |
the paralysis associated with myelomeningocele: clinical and experimental data implicating a preventable spinal cord injury. |
1990-08-08 |
2023-08-11 |
rat |
| D S Heffez, J Aryanpur, G M Hutchins, J M Freema. The paralysis associated with myelomeningocele: clinical and experimental data implicating a preventable spinal cord injury. Neurosurgery. vol 26. issue 6. 1990-08-08. PMID:2362676. |
we suspected that paralysis may be due in part to a spinal cord injury caused by exposure of the neural tube to the amniotic fluid. |
1990-08-08 |
2023-08-11 |
rat |
| D S Heffez, J Aryanpur, G M Hutchins, J M Freema. The paralysis associated with myelomeningocele: clinical and experimental data implicating a preventable spinal cord injury. Neurosurgery. vol 26. issue 6. 1990-08-08. PMID:2362676. |
we therefore propose a "two-hit" hypothesis to explain the paralysis seen in children with myelomeningocele: congenital myelodysplasia complicated by an intrauterine spinal cord injury. |
1990-08-08 |
2023-08-11 |
rat |
| D S Heffez, J Aryanpur, G M Hutchins, J M Freema. The paralysis associated with myelomeningocele: clinical and experimental data implicating a preventable spinal cord injury. Neurosurgery. vol 26. issue 6. 1990-08-08. PMID:2362676. |
intrauterine protection of the exposed spinal cord might prevent some or all of the paralysis. |
1990-08-08 |
2023-08-11 |
rat |
| M Matsuoka, H Igisu, J Lin, N Inou. Effects of acrylamide and N,N'-methylene-bis-acrylamide on creatine kinase activity. Brain research. vol 507. issue 2. 1990-06-20. PMID:2337777. |
in vivo, only acrylamide (50 mg/kg b. wt./day, 8 days) caused paralysis of hind limbs and suppressed ck activity in cerebrum, cerebellum, spinal cord and muscle in rats. |
1990-06-20 |
2023-08-11 |
rat |
| H Takahashi, T Yamamuro, H Okumura, R Kasai, K Tad. Bone blood flow after spinal paralysis in the rat. Journal of orthopaedic research : official publication of the Orthopaedic Research Society. vol 8. issue 3. 1990-05-21. PMID:2324858. |
the goal of this study was to investigate the acute and chronic effects of paralysis induced by spinal cord section or sciatic neurotomy on bone blood flow in the rat. |
1990-05-21 |
2023-08-11 |
rat |
| A Otter, W F Blakemor. Observation on the presence of Listeria monocytogenes in axons. Acta microbiologica Hungarica. vol 36. issue 2-3. 1990-05-09. PMID:2517159. |
paralysis of the injected leg occurs 7-12 days later and examination of the spinal cord reveals lesions very similar to those found in the brains of affected sheep. |
1990-05-09 |
2023-08-11 |
mouse |