| Publication |
Sentence |
Publish Date |
Extraction Date |
Species |
| Y I Sheline, P W Wang, M H Gado, J G Csernansky, M W Vannie. Hippocampal atrophy in recurrent major depression. Proceedings of the National Academy of Sciences of the United States of America. vol 93. issue 9. 1996-07-01. PMID:8632988. |
these results suggest that depression is associated with hippocampal atrophy, perhaps due to a progressive process mediated by glucocorticoid neurotoxicity. |
1996-07-01 |
2023-08-12 |
human |
| D M Spillane, T W Rosahl, T C Südhof, R C Malenk. Long-term potentiation in mice lacking synapsins. Neuropharmacology. vol 34. issue 11. 1996-05-20. PMID:8606805. |
these results indicate that the synapsins are not necessary for: (1) the induction or expression of two different forms of ltp in the hippocampus, (2) the enhancement in transmitter release elicited by activation of the camp-dependent protein kinase (pka) and (3) the depression of synaptic transmission caused by h-7. |
1996-05-20 |
2023-08-12 |
mouse |
| D A Turner, P G Aitken, G G Somje. Optical mapping of translucence changes in rat hippocampal slices during hypoxia. Neuroscience letters. vol 195. issue 3. 1996-03-21. PMID:8584212. |
measurement of intrinsic optical signals revealed both spatial and temporal patterns of cell volume changes associated with hypoxia and spreading depression in hippocampus. |
1996-03-21 |
2023-08-12 |
rat |
| C Largo, P Cuevas, G G Somjen, R Martín del Río, O Herrera. The effect of depressing glial function in rat brain in situ on ion homeostasis, synaptic transmission, and neuron survival. The Journal of neuroscience : the official journal of the Society for Neuroscience. vol 16. issue 3. 1996-02-28. PMID:8558250. |
repeated sd provoked in hippocampus in the absence of fc caused only mild depression of a-ps. |
1996-02-28 |
2023-08-12 |
rat |
| R Huang, P G Aitken, G G Somje. The extent and mechanism of the loss of function caused by strongly hypotonic solutions in rat hippocampal slices. Brain research. vol 695. issue 2. 1996-02-27. PMID:8556331. |
to investigate whether prolonged severe swelling would cause irreversible injury to neurons, we exposed hippocampal tissue slices to hypotonic solutions (142 mosmol/kg) and followed the recovery of evoked responses for 5 h. orthodromically evoked responses increased during hypotonia, except during recurrent waves of spreading depression (sd). |
1996-02-27 |
2023-08-12 |
rat |
| S R Chebabo, M A Hester, P G Aitken, G G Somje. Hypotonic exposure enhances synaptic transmission and triggers spreading depression in rat hippocampal tissue slices. Brain research. vol 695. issue 2. 1996-02-27. PMID:8556332. |
hypotonic exposure enhances synaptic transmission and triggers spreading depression in rat hippocampal tissue slices. |
1996-02-27 |
2023-08-12 |
rat |
| D S Kerr, W C Abraha. Cooperative interactions among afferents govern the induction of homosynaptic long-term depression in the hippocampus. Proceedings of the National Academy of Sciences of the United States of America. vol 92. issue 25. 1996-01-24. PMID:8524819. |
prolonged periods of low-frequency stimulation have been shown to produce a robust, long-term synaptic depression (ltd) in both hippocampus and visual cortex. |
1996-01-24 |
2023-08-12 |
rat |
| P T Huerta, J E Lisma. Bidirectional synaptic plasticity induced by a single burst during cholinergic theta oscillation in CA1 in vitro. Neuron. vol 15. issue 5. 1995-12-21. PMID:7576649. |
in standard protocols, the frequency of synaptic stimulation determines whether ca1 hippocampal synapses undergo long-term potentiation or depression. |
1995-12-21 |
2023-08-12 |
Not clear |
| C Bernard, H V Whea. Plasticity of AMPA and NMDA receptor-mediated epileptiform activity in a chronic model of temporal lobe epilepsy. Epilepsy research. vol 21. issue 2. 1995-12-06. PMID:7588593. |
thus although it has been suggested that the nmda receptor-mediated synaptic events contribute to the epileptogenic properties of the neocortex and hippocampus, this evoked depression may act as an intrinsic anticonvulsant mechanism. |
1995-12-06 |
2023-08-12 |
human |
| C Bernard, H V Whea. Simultaneous expression of excitatory postsynaptic potential/spike potentiation and excitatory postsynaptic potential/spike depression in the hippocampus. Neuroscience. vol 67. issue 1. 1995-12-04. PMID:7477912. |
simultaneous expression of excitatory postsynaptic potential/spike potentiation and excitatory postsynaptic potential/spike depression in the hippocampus. |
1995-12-04 |
2023-08-12 |
rat |
| C Bernard, H V Whea. Simultaneous expression of excitatory postsynaptic potential/spike potentiation and excitatory postsynaptic potential/spike depression in the hippocampus. Neuroscience. vol 67. issue 1. 1995-12-04. PMID:7477912. |
in this study, using kainic acid lesioned hippocampus, we have shown that tetanic stimulation produced excitatory postsynaptic potential/spike potentiation of the control synaptic response and excitatory postsynaptic potential/spike depression of the isolated n-methyl-d-aspartate receptor-mediated responses. |
1995-12-04 |
2023-08-12 |
rat |
| J A Davies, S J Annels, B G Dickie, Y Ellis, N J Knot. A comparison between the stimulated and paroxysmal release of endogenous amino acids from rat cerebellar, striatal and hippocampal slices: a manifestation of spreading depression? Journal of the neurological sciences. vol 131. issue 1. 1995-11-20. PMID:7561953. |
a comparison between the stimulated and paroxysmal release of endogenous amino acids from rat cerebellar, striatal and hippocampal slices: a manifestation of spreading depression? |
1995-11-20 |
2023-08-12 |
rat |
| J A Davies, S J Annels, B G Dickie, Y Ellis, N J Knot. A comparison between the stimulated and paroxysmal release of endogenous amino acids from rat cerebellar, striatal and hippocampal slices: a manifestation of spreading depression? Journal of the neurological sciences. vol 131. issue 1. 1995-11-20. PMID:7561953. |
we have observed a transient spontaneous release of amino acids in slice preparations obtained from rat cerebellum, striatum and hippocampus; this phenomenon has similar properties to stimulus-evoked spreading depression. |
1995-11-20 |
2023-08-12 |
rat |
| P Igelmun. Modulation of synaptic transmission at low temperatures by hibernation-related changes in ionic microenvironment in hippocampal slices of golden hamsters. Cryobiology. vol 32. issue 4. 1995-10-02. PMID:7656568. |
in conclusion, changes in the ionic microenvironment occurring during entrance into hibernation presumably result in depression of synaptic transmission at low temperatures in the hamster hippocampus. |
1995-10-02 |
2023-08-12 |
Not clear |
| M Bijak, M Pap. The effect of chronic treatment with imipramine on the responsiveness of hippocampal CA1 neurons to phenylephrine and serotonin in a chronic mild stress model of depression. European neuropsychopharmacology : the journal of the European College of Neuropsychopharmacology. vol 5. issue 1. 1995-08-24. PMID:7613100. |
these results suggest that the imipramine-induced functional changes in alpha 1-noradrenergic and serotonergic receptors in the hippocampus are not involved in the anti-anhedonic effect of chronic imipramine administration in the cms model of depression. |
1995-08-24 |
2023-08-12 |
rat |
| D J Doolette, D I Ker. Hyperexcitability in CA1 of the rat hippocampal slice following hypoxia or adenosine. Brain research. vol 677. issue 1. 1995-08-17. PMID:7606456. |
participation of adenosine receptors in the depression of synaptic transmission during hypoxia, and the production of multiple populations spikes in the pyramidal neurons following hypoxia, has been investigated in the ca1 area of the rat hippocampal slice. |
1995-08-17 |
2023-08-12 |
rat |
| R A Pearce, S D Grunder, L D Fauche. Different mechanisms for use-dependent depression of two GABAA-mediated IPSCs in rat hippocampus. The Journal of physiology. vol 484 ( Pt 2). 1995-08-10. PMID:7602535. |
different mechanisms for use-dependent depression of two gabaa-mediated ipscs in rat hippocampus. |
1995-08-10 |
2023-08-12 |
rat |
| R A Pearce, S D Grunder, L D Fauche. Different mechanisms for use-dependent depression of two GABAA-mediated IPSCs in rat hippocampus. The Journal of physiology. vol 484 ( Pt 2). 1995-08-10. PMID:7602535. |
the mechanisms involved in the use-dependent depression of gabaa,fast and gabaa,slow, two gabaa-mediated ipscs in the rat hippocampal slice preparation, were investigated by observing the effects of paired-pulse depression and of baclofen and cgp 35348 on monosynaptic inhibitory currents recorded from ca1 pyramidal neurons. |
1995-08-10 |
2023-08-12 |
rat |
| X O Zhu, N McNaughto. Effects of long-term administration of antidepressants on septal driving of hippocampal RSA. The International journal of neuroscience. vol 79. issue 1-2. 1995-06-14. PMID:7744554. |
since both acute and chronic administration of imipramine but not phenelzine also produce similar effects to anxiolytics on the frequency of reticular-elicited hippocampal rsa, we suggest that (1) imipramine has a separate anxiolytic action, in addition to its antidepressant action; and (2) phenelzine may have no central anxiolytic action despite its capacity to relieve somatic symptoms in atypical depression. |
1995-06-14 |
2023-08-12 |
rat |
| N Kawahara, C A Ruetzler, I Klatz. Protective effect of spreading depression against neuronal damage following cardiac arrest cerebral ischaemia. Neurological research. vol 17. issue 1. 1995-06-13. PMID:7746347. |
effect of spreading depression (sd) on survival of ca1 hippocampal neurons was studied in sprague-dawley rats subjected to cardiac arrest cerebral ischaemia (caci). |
1995-06-13 |
2023-08-12 |
rat |