| Publication |
Sentence |
Publish Date |
Extraction Date |
Species |
| C Krieger, D Jacques, A Parent, R Quirio. [3H]phorbol 12,13-dibutyrate/PKC binding in thoracic spinal cord: no change in amyotrophic lateral sclerosis. Neuroreport. vol 6. issue 10. 1996-01-02. PMID:7488729. |
[3h]phorbol 12,13-dibutyrate/pkc binding in thoracic spinal cord: no change in amyotrophic lateral sclerosis. |
1996-01-02 |
2023-08-12 |
human |
| R M Duberley, I P Johnson, P Anand, M Swash, J Martin, P N Leigh, S Zema. Ciliary neurotrophic factor receptor expression in spinal cord and motor cortex in amyotrophic lateral sclerosis. Journal of the neurological sciences. vol 129 Suppl. 1995-11-24. PMID:7595599. |
ciliary neurotrophic factor receptor expression in spinal cord and motor cortex in amyotrophic lateral sclerosis. |
1995-11-24 |
2023-08-12 |
Not clear |
| R M Duberley, I P Johnson, P Anand, M Swash, J Martin, P N Leigh, S Zema. Ciliary neurotrophic factor receptor expression in spinal cord and motor cortex in amyotrophic lateral sclerosis. Journal of the neurological sciences. vol 129 Suppl. 1995-11-24. PMID:7595599. |
ciliary neurotrophic factor (cntf) is known to rescue motor neurones in animal models of injury and neurodegeneration and we have recently described regional changes of cntf protein levels in spinal cord from patients with sporadic motor neurone disease of the amyotrophic lateral sclerosis (als) type. |
1995-11-24 |
2023-08-12 |
Not clear |
| R A Lanius, H B Paddon, M Mezei, R Wagey, C Krieger, S L Pelech, C A Sha. A role for amplified protein kinase C activity in the pathogenesis of amyotrophic lateral sclerosis. Journal of neurochemistry. vol 65. issue 2. 1995-08-24. PMID:7616256. |
amyotrophic lateral sclerosis (als) is a human neurodegenerative disorder of unknown origin that is characterized by progressive degeneration of corticospinal tracts and anterior horn cells in the brainstem and spinal cord. |
1995-08-24 |
2023-08-12 |
human |
| L Virgo, J de Belleroch. Induction of the immediate early gene c-jun in human spinal cord in amyotrophic lateral sclerosis with concomitant loss of NMDA receptor NR-1 and glycine transporter mRNA. Brain research. vol 676. issue 1. 1995-08-03. PMID:7796170. |
induction of the immediate early gene c-jun in human spinal cord in amyotrophic lateral sclerosis with concomitant loss of nmda receptor nr-1 and glycine transporter mrna. |
1995-08-03 |
2023-08-12 |
human |
| M Bonduell. [Charcot. Dates. Legend and reality]. Histoire des sciences medicales. vol 28. issue 4. 1995-05-25. PMID:11640481. |
taking advantage of histology together with a mastery of the anatomical-clinical method permitted him to achieve considerable progress with a few years (associating locomotor ataxia with lesions of the posterior roots and columns; acute and chronic progressive muscular atrophy with those of the anterior horn: separating multiple sclerosis from parkinson's disease) which culminated in the masterful description of amyotrophic lateral sclerosis (1874), the pathological anatomy and physiology of the spinal cord (1873) and cerebral localisations of motor areas (1875). |
1995-05-25 |
2023-08-12 |
Not clear |
| J S Parboosingh, G A Rouleau, V Meninger, D McKenna-Yasek, R H Brown, D A Figlewic. Absence of mutations in the Mn superoxide dismutase or catalase genes in familial amyotrophic lateral sclerosis. Neuromuscular disorders : NMD. vol 5. issue 1. 1995-05-22. PMID:7719145. |
familial amyotrophic lateral sclerosis (fals) is an autosomal dominant, adult onset, neurological disorder caused by the degeneration of motor neurons of the cortex, brainstem and spinal cord. |
1995-05-22 |
2023-08-12 |
Not clear |
| M Uchino, Y Ando, Y Tanaka, T Nakamura, E Uyama, S Mita, T Murakami, M And. Decrease in Cu/Zn- and Mn-superoxide dismutase activities in brain and spinal cord of patients with amyotrophic lateral sclerosis. Journal of the neurological sciences. vol 127. issue 1. 1995-04-28. PMID:7699393. |
decrease in cu/zn- and mn-superoxide dismutase activities in brain and spinal cord of patients with amyotrophic lateral sclerosis. |
1995-04-28 |
2023-08-12 |
Not clear |
| A Adem, J Ekblom, P G Gillberg, S S Jossan, A Höög, B Winblad, S M Aquilonius, L H Wang, V Sar. Insulin-like growth factor-1 receptors in human spinal cord: changes in amyotrophic lateral sclerosis. Journal of neural transmission. General section. vol 97. issue 1. 1995-04-17. PMID:7888151. |
insulin-like growth factor-1 receptors in human spinal cord: changes in amyotrophic lateral sclerosis. |
1995-04-17 |
2023-08-12 |
human |
| M R Witt, O Gredal, K Dekermendjian, M Undén, M Nielse. Calcium homeostasis in fibroblasts from patients with amyotrophic lateral sclerosis. Journal of the neurological sciences. vol 126. issue 2. 1995-03-14. PMID:7853028. |
amyotrophic lateral sclerosis (als) is a chronic neurodegenerative disorder of the motor system in the cns characterized by motor neuron death in the spinal cord, brain stem and cortex. |
1995-03-14 |
2023-08-12 |
human |
| P A Sillevis Smitt, T P Mulder, H W Verspaget, H G Blaauwgeers, D Troost, J M de Jon. Metallothionein in amyotrophic lateral sclerosis. Biological signals. vol 3. issue 4. 1995-02-28. PMID:7834014. |
amyotrophic lateral sclerosis (als) is a neurodegenerative disorder characterized by loss of motor neurons in the spinal cord, brainstem and motor cortex. |
1995-02-28 |
2023-08-12 |
Not clear |
| T N Nagaraja, M Gourie-Devi, A Nalini, T R Raj. Neurofilament phosphorylation is enhanced in cultured chick spinal cord neurons exposed to cerebrospinal fluid from amyotrophic lateral sclerosis patients. Acta neuropathologica. vol 88. issue 4. 1995-02-27. PMID:7839827. |
neurofilament phosphorylation is enhanced in cultured chick spinal cord neurons exposed to cerebrospinal fluid from amyotrophic lateral sclerosis patients. |
1995-02-27 |
2023-08-12 |
chicken |
| S Terao, G Sobue, T Yasuda, T Kachi, T Mitsum. [The corticospinal tract lesion of amyotrophic lateral sclerosis--magnetic resonance imaging of the spinal cord]. Rinsho shinkeigaku = Clinical neurology. vol 34. issue 9. 1995-02-14. PMID:7820958. |
[the corticospinal tract lesion of amyotrophic lateral sclerosis--magnetic resonance imaging of the spinal cord]. |
1995-02-14 |
2023-08-12 |
Not clear |
| P J Shaw, R M Chinnery, P G Inc. Non-NMDA receptors in motor neuron disease (MND): a quantitative autoradiographic study in spinal cord and motor cortex using [3H]CNQX and [3H]kainate. Brain research. vol 655. issue 1-2. 1995-02-06. PMID:7812772. |
the changes in [3h]kainate binding were observed only in the amyotrophic lateral sclerosis (als) subgroup of mnd, while the changes in [3h]cnqx binding in the spinal cord were more marked in als compared to progressive muscular atrophy (pma) cases. |
1995-02-06 |
2023-08-12 |
Not clear |
| D A Figlewicz, M G McInnis, J Goto, J L Haines, A C Warren, A Krizus, N Khodr, R H Brown, D McKenna-Yasek, S E Antonaraki. Identification of flanking markers for the familial amyotrophic lateral sclerosis gene ALS1 on chromosome 21. Journal of the neurological sciences. vol 124 Suppl. 1995-02-02. PMID:7807155. |
amyotrophic lateral sclerosis (als) is a progressive, adult-onset, neurodegenerative disorder characterized by the death of large motor neurons from the cerebral cortex, brainstem, and spinal cord. |
1995-02-02 |
2023-08-12 |
Not clear |
| Y Yoshiyama, T Yamada, K Asanuma, T Asah. Apoptosis related antigen, Le(Y) and nick-end labeling are positive in spinal motor neurons in amyotrophic lateral sclerosis. Acta neuropathologica. vol 88. issue 3. 1995-01-31. PMID:7810291. |
immunohistochemistry using an antibody for le(y), as well as nick-end labeling for the detection of dna breaks, was done on cervical spinal cord sections from ten patients with amyotrophic lateral sclerosis (als) and nine patients who had died from other causes. |
1995-01-31 |
2023-08-12 |
Not clear |
| T Umahara, A Hirano, S Kato, N Shibata, S H Ye. Demonstration of neurofibrillary tangles and neuropil thread-like structures in spinal cord white matter in parkinsonism-dementia complex on Guam and in Guamanian amyotrophic lateral sclerosis. Acta neuropathologica. vol 88. issue 2. 1995-01-04. PMID:7985499. |
demonstration of neurofibrillary tangles and neuropil thread-like structures in spinal cord white matter in parkinsonism-dementia complex on guam and in guamanian amyotrophic lateral sclerosis. |
1995-01-04 |
2023-08-12 |
Not clear |
| T Umahara, A Hirano, S Kato, N Shibata, S H Ye. Demonstration of neurofibrillary tangles and neuropil thread-like structures in spinal cord white matter in parkinsonism-dementia complex on Guam and in Guamanian amyotrophic lateral sclerosis. Acta neuropathologica. vol 88. issue 2. 1995-01-04. PMID:7985499. |
this report concerns the demonstration and distribution of neurofibrillary tangles (nfts), immunoreactive neuropil thread-like structures and dots in the spinal cord gray and white matter of six patients with parkinsonism-dementia complex on guam and five patients with guamanian amyotrophic lateral sclerosis (als). |
1995-01-04 |
2023-08-12 |
Not clear |
| M Tomiyama, K Kannari, J Nunomura, Y Oyama, K Takebe, M Matsunag. Quantitative autoradiographic distribution of glutamate receptors in the cervical segment of the spinal cord of the wobbler mouse. Brain research. vol 650. issue 2. 1994-12-29. PMID:7953705. |
the reduction of glutamate content has been observed in the spinal cord of the wobbler mouse, a purported model of amyotrophic lateral sclerosis (als). |
1994-12-29 |
2023-08-12 |
mouse |
| M E Alexianu, A H Mohamed, R G Smith, L V Colom, S H Appe. Apoptotic cell death of a hybrid motoneuron cell line induced by immunoglobulins from patients with amyotrophic lateral sclerosis. Journal of neurochemistry. vol 63. issue 6. 1994-12-23. PMID:7964760. |
previous experiments have documented that immunoglobulins from patients with amyotrophic lateral sclerosis (als) can kill motoneuron-neuroblastoma hybrid cells [ventral spinal cord 4.1 (vsc 4.1)] by a calcium-dependent process. |
1994-12-23 |
2023-08-12 |
human |