All Relations between Amyotrophic Lateral Sclerosis and spinal cord

Publication Sentence Publish Date Extraction Date Species
A Matsumoto, A Kawashima, S Doi, F Moriwaka, K Tashir. [The spinal somatosensory evoked potentials in amyotrophic lateral sclerosis in relation to the spinal cord conduction velocities]. No to shinkei = Brain and nerve. vol 51. issue 1. 1999-04-21. PMID:10065459. the lumbar-to-cervical conduction velocity (spinal cord conduction velocity, sccv) was electrophysiologically studied in 14 patients with amyotrophic lateral sclerosis (als). 1999-04-21 2023-08-12 human
N K Wong, M J Stron. Nitric oxide synthase expression in cervical spinal cord in sporadic amyotrophic lateral sclerosis. European journal of cell biology. vol 77. issue 4. 1999-04-13. PMID:9930658. nitric oxide synthase expression in cervical spinal cord in sporadic amyotrophic lateral sclerosis. 1999-04-13 2023-08-12 Not clear
N Mitsuma, M Yamamoto, M Li, Y Ito, T Mitsuma, T Mutoh, M Takahashi, G Sobu. Expression of GDNF receptor (RET and GDNFR-alpha) mRNAs in the spinal cord of patients with amyotrophic lateral sclerosis. Brain research. vol 820. issue 1-2. 1999-03-29. PMID:10023033. expression of gdnf receptor (ret and gdnfr-alpha) mrnas in the spinal cord of patients with amyotrophic lateral sclerosis. 1999-03-29 2023-08-12 Not clear
N Mitsuma, M Yamamoto, M Li, Y Ito, T Mitsuma, T Mutoh, M Takahashi, G Sobu. Expression of GDNF receptor (RET and GDNFR-alpha) mRNAs in the spinal cord of patients with amyotrophic lateral sclerosis. Brain research. vol 820. issue 1-2. 1999-03-29. PMID:10023033. the mrna levels of ret and gdnfr-alpha were studied in the spinal cord of patients with amyotrophic lateral sclerosis (als) by reverse transcription followed by polymerase chain reaction (rt-pcr) and in situ hybridization (ish). 1999-03-29 2023-08-12 Not clear
R K Phul, M E Smith, P J Shaw, P G Inc. Expression of nitric oxide synthase in the spinal cord in amyotrophic lateral sclerosis. Journal of the neurological sciences. vol 160 Suppl 1. 1999-03-18. PMID:9851656. expression of nitric oxide synthase in the spinal cord in amyotrophic lateral sclerosis. 1999-03-18 2023-08-12 human
R K Phul, M E Smith, P J Shaw, P G Inc. Expression of nitric oxide synthase in the spinal cord in amyotrophic lateral sclerosis. Journal of the neurological sciences. vol 160 Suppl 1. 1999-03-18. PMID:9851656. the expression of nitric oxide synthase was studied in human postmortem cervical spinal cord from four individuals with amyotrophic lateral sclerosis (als) and four individuals who had died from non-neurological causes. 1999-03-18 2023-08-12 human
S Ono, T Imai, S Munakata, K Takahashi, F Kanda, K Hashimoto, T Yamano, N Shimizu, K Nagao, M Yamauch. Collagen abnormalities in the spinal cord from patients with amyotrophic lateral sclerosis. Journal of the neurological sciences. vol 160. issue 2. 1999-03-03. PMID:9849796. collagen abnormalities in the spinal cord from patients with amyotrophic lateral sclerosis. 1999-03-03 2023-08-12 Not clear
J P Julien, S Couillard-Després, J Meie. Transgenic mice in the study of ALS: the role of neurofilaments. Brain pathology (Zurich, Switzerland). vol 8. issue 4. 1999-01-29. PMID:9804382. amyotrophic lateral sclerosis (als) is an adult-onset neurological disorder of multiple etiologies that affects primarily motor neurons in the brain and spinal cord. 1999-01-29 2023-08-12 mouse
I Milona. Amyotrophic lateral sclerosis: an introduction. Journal of neurology. vol 245 Suppl 2. 1998-12-09. PMID:9747926. amyotrophic lateral sclerosis (als) is a progressive, neurodegenerative disease that results in the degeneration of lower and upper motor neurones in the brain and the spinal cord. 1998-12-09 2023-08-12 Not clear
M E Gurney, R Liu, J S Althaus, E D Hall, D A Becke. Mutant CuZn superoxide dismutase in motor neuron disease. Journal of inherited metabolic disease. vol 21. issue 5. 1998-12-03. PMID:9728338. mutations of the sod1 gene encoding cuzn sod are found in patients with familial amyotrophic lateral sclerosis (fals), a progressive and fatal paralytic disease that is caused by the death of motor neurons in cortex, brainstem and spinal cord. 1998-12-03 2023-08-12 mouse
M Urushitani, S Shimohama, T Kihara, H Sawada, A Akaike, M Ibi, R Inoue, Y Kitamura, T Taniguchi, J Kimur. Mechanism of selective motor neuronal death after exposure of spinal cord to glutamate: involvement of glutamate-induced nitric oxide in motor neuron toxicity and nonmotor neuron protection. Annals of neurology. vol 44. issue 5. 1998-12-02. PMID:9818936. in this study, we analyzed the mechanism of selective motor neuronal death, a characteristic of amyotrophic lateral sclerosis, using embryonic rat spinal cord culture. 1998-12-02 2023-08-12 rat
W A Pedersen, W Fu, J N Keller, W R Markesbery, S Appel, R G Smith, E Kasarskis, M P Mattso. Protein modification by the lipid peroxidation product 4-hydroxynonenal in the spinal cords of amyotrophic lateral sclerosis patients. Annals of neurology. vol 44. issue 5. 1998-12-02. PMID:9818940. we report increased modification of proteins by 4-hydroxynonenal (hne), a product of membrane lipid peroxidation, in the lumbar spinal cord of sporadic amyotrophic lateral sclerosis (als) patients versus that of neurologically normal controls. 1998-12-02 2023-08-12 Not clear
M Nagai, K Abe, K Okamoto, Y Itoyam. Identification of alternative splicing forms of GLT-1 mRNA in the spinal cord of amyotrophic lateral sclerosis patients. Neuroscience letters. vol 244. issue 3. 1998-11-18. PMID:9593515. identification of alternative splicing forms of glt-1 mrna in the spinal cord of amyotrophic lateral sclerosis patients. 1998-11-18 2023-08-12 human
Y Kawamoto, S Nakamura, I Akiguchi, J Kimur. Immunohistochemical localization of brain-derived neurotrophic factor in the spinal cords of amyotrophic lateral sclerosis and non-amyotrophic lateral sclerosis patients. Journal of neuropathology and experimental neurology. vol 57. issue 9. 1998-09-25. PMID:9737545. to localize and assess bdnf in the human spinal cord with particular reference to amyotrophic lateral sclerosis (als), we immunohistochemically studied spinal cords from 8 als and 13 non-als patients. 1998-09-25 2023-08-12 human
L J Martin, N A Al-Abdulla, A M Brambrink, J R Kirsch, F E Sieber, C Portera-Caillia. Neurodegeneration in excitotoxicity, global cerebral ischemia, and target deprivation: A perspective on the contributions of apoptosis and necrosis. Brain research bulletin. vol 46. issue 4. 1998-09-24. PMID:9671259. in the human brain and spinal cord, neurons degenerate after acute insults (e.g., stroke, cardiac arrest, trauma) and during progressive, adult-onset diseases [e.g., amyotrophic lateral sclerosis, alzheimer's disease]. 1998-09-24 2023-08-12 human
M J Strong, M M Sopper, J P Crow, W L Strong, J S Beckma. Nitration of the low molecular weight neurofilament is equivalent in sporadic amyotrophic lateral sclerosis and control cervical spinal cord. Biochemical and biophysical research communications. vol 248. issue 1. 1998-08-06. PMID:9675103. nitration of the low molecular weight neurofilament is equivalent in sporadic amyotrophic lateral sclerosis and control cervical spinal cord. 1998-08-06 2023-08-12 mouse
N P Bajaj, S T Al-Sarraj, V Anderson, M Kibble, N Leigh, C C Mille. Cyclin-dependent kinase-5 is associated with lipofuscin in motor neurones in amyotrophic lateral sclerosis. Neuroscience letters. vol 245. issue 1. 1998-07-14. PMID:9596352. we have studied the distribution of cyclin dependent kinase-5 (cdk-5) within spinal cord in sporadic and two superoxide dismutase type 1 (sod1) familial cases of amyotrophic lateral sclerosis (als). 1998-07-14 2023-08-12 Not clear
R J Singh, H Karoui, M R Gunther, J S Beckman, R P Mason, B Kalyanarama. Reexamination of the mechanism of hydroxyl radical adducts formed from the reaction between familial amyotrophic lateral sclerosis-associated Cu,Zn superoxide dismutase mutants and H2O2. Proceedings of the National Academy of Sciences of the United States of America. vol 95. issue 12. 1998-07-09. PMID:9618471. amyotrophic lateral sclerosis (als) involves the progressive degeneration of motor neurons in the spinal cord and motor cortex. 1998-07-09 2023-08-12 Not clear
T Canton, J Pratt, J M Stutzmann, A Imperato, A Boirea. Glutamate uptake is decreased tardively in the spinal cord of FALS mice. Neuroreport. vol 9. issue 5. 1998-07-01. PMID:9579663. this study examined high affinity na+-dependent uptake of glutamate in synaptosomal preparations from spinal cord in mice that express a dominant mutation of human copper/zinc superoxide dismutase (sod1) and represent an animal model of amyotrophic lateral sclerosis (als). 1998-07-01 2023-08-12 mouse
T L Parkes, A J Elia, D Dickinson, A J Hilliker, J P Phillips, G L Bouliann. Extension of Drosophila lifespan by overexpression of human SOD1 in motorneurons. Nature genetics. vol 19. issue 2. 1998-07-01. PMID:9620775. the association between mutations in the gene encoding the oxygen radical metabolizing enzyme cuzn superoxide dismutase (sod1) and loss of motorneurons in the brain and spinal cord that occurs in the life-shortening paralytic disease, familial amyotrophic lateral sclerosis (fals; ref. 1998-07-01 2023-08-12 human