All Relations between cerebellum and Ataxia

Publication Sentence Publish Date Extraction Date Species
T A Martin, J G Keating, H P Goodkin, A J Bastian, W T Thac. Throwing while looking through prisms. I. Focal olivocerebellar lesions impair adaptation. Brain : a journal of neurology. vol 119 ( Pt 4). 1996-11-26. PMID:8813282. patients with infarcts in the distribution of the posterior inferior cerebellar artery usually had impaired or absent adaptation but little or no ataxia. 1996-11-26 2023-08-12 human
T A Martin, J G Keating, H P Goodkin, A J Bastian, W T Thac. Throwing while looking through prisms. I. Focal olivocerebellar lesions impair adaptation. Brain : a journal of neurology. vol 119 ( Pt 4). 1996-11-26. PMID:8813282. by contrast, patients with damage in the distribution of the superior cerebellar artery or in cerebellar thalamus usually had ataxia but preserved adaptation. 1996-11-26 2023-08-12 human
L Schöls, O Riess, S Schöls, S Zeck, G Amoiridis, M Langkafel, J T Epplen, H Przunte. Spinocerebellar ataxia type 1: Clinical and neurophysiological characteristics in German kindreds. Acta neurologica Scandinavica. vol 92. issue 6. 1996-11-22. PMID:8750114. sca1 accounts for 10-15% of dominant cerebellar ataxias in german kindreds. 1996-11-22 2023-08-12 Not clear
R Näkki, F R Sharp, S M Saga. FOS expression in the brainstem and cerebellum following phencyclidine and MK801. Journal of neuroscience research. vol 43. issue 2. 1996-10-30. PMID:8820968. these results support our earlier observations that pcp and mk801 have different actions in the cerebellum, although they both cause ataxia and indistinguishable behavioral symptoms. 1996-10-30 2023-08-12 rat
I Lopes-Cendes, P Maciel, S Kish, C Gaspar, Y Robitaille, H B Clark, A H Koeppen, M Nance, L Schut, I Silveira, P Coutinho, J Sequeiros, G A Roulea. Somatic mosaicism in the central nervous system in spinocerebellar ataxia type 1 and Machado-Joseph disease. Annals of neurology. vol 40. issue 2. 1996-10-16. PMID:8773601. spinocerebellar ataxia type 1 and machado-joseph disease are two autosomal dominant cerebellar ataxias caused by expansions of unstable cag repeats in the coding region of the causative genes. 1996-10-16 2023-08-12 Not clear
K Kinoshita, Y Watanabe, H Asai, M Yamamura, Y Matsuok. Anti-ataxic effects of TRH and its analogue, TA-0910, in Rolling mouse Nagoya by metabolic normalization of the ventral tegmental area. British journal of pharmacology. vol 116. issue 8. 1996-10-10. PMID:8719807. these results suggest that the ataxia of the rolling mouse may be due to dysfunction of the cerebellum and vta, and that amelioration by trh and ta-0910 could result from metabolic normalization of the vta. 1996-10-10 2023-08-12 mouse
M Watanabe, K Abe, M Aoki, T Kameya, J Kaneko, M Shoji, M Ikeda, M Shizuka, Y Ikeda, T Iizuka, S Hirai, Y Itoyam. Analysis of CAG trinucleotide expansion associated with Machado-Joseph disease. Journal of the neurological sciences. vol 136. issue 1-2. 1996-10-04. PMID:8815156. there are currently some types of autosomal dominant cerebellar ataxias such as machado-joseph disease (mjd), spinocerebellar ataxia types 1-5 (sca1-5), or hereditary dentatorubropallidoluysian atrophy. 1996-10-04 2023-08-12 Not clear
W Ziegler, K Wesse. Speech timing in ataxic disorders: sentence production and rapid repetitive articulation. Neurology. vol 47. issue 1. 1996-09-12. PMID:8710079. we studied syllabic timing in patients with ataxia (10 with cerebellar atrophy, 6 with friedreich's ataxia) under two conditions: in a "natural" sentence production context and in the context of a rapid syllable repetition task. 1996-09-12 2023-08-12 Not clear
E Buchwald, S Vorstru. [Creutzfeldt-Jakob disease--a human prion disease]. Nordisk medicin. vol 111. issue 6. 1996-09-05. PMID:8700643. the human prion diseases, creutzfeldt-jakob disease, gerstmann-strøaussler-scheinker syndrome and kuru, are neurodegenerative disorders sharing clinical features of rapidly progressive neurodegenerative dementia and cerebellar symptoms of marked ataxia and tremor, resulting in death within one year after onset. 1996-09-05 2023-08-12 human
P J Vig, D Desaiah, S H Subramony, J D Fratki. Developmental changes in cerebellar endothelin-1 receptors in the neurologic mouse lurcher mutant. Research communications in molecular pathology and pharmacology. vol 89. issue 3. 1996-08-20. PMID:8680799. the lurcher mutant is a suggested animal model for human cerebellar ataxias. 1996-08-20 2023-08-12 mouse
J C Aschoff, N A Kailer, K Walte. [Physostigmine in treatment of cerebellar ataxia]. Der Nervenarzt. vol 67. issue 4. 1996-08-19. PMID:8684510. cerebellar ataxias are still a challenging problem for neurologists, and to this day there exists no medical, physiotherapeutic, psychot-erapeutic or surgical therapy which constantly leads to a reduction of ataxic symptoms. 1996-08-19 2023-08-12 Not clear
A Berio, A Piazz. [Marinesco-Sjögren syndrome with chronic progressive ophthalmoplegia caused by presumed defective oxidative phosphorylation]. La Pediatria medica e chirurgica : Medical and surgical pediatrics. vol 18. issue 1. 1996-08-19. PMID:8685033. a case of marinesco-sjögren syndrome (ataxia, cataracts, retarded somatic and mental maturation, spasticity) with myopathy, hyperlactacidaemia, hypopyruvicaemia, chronic progressive external ophthalmoplegia was reported and relationships with the cerebellar ataxias of unknown aetiology discussed. 1996-08-19 2023-08-12 Not clear
S H Subramony, J D Fratkin, B V Manyam, R D Currie. Dominantly inherited cerebello-olivary atrophy is not due to a mutation at the spinocerebellar ataxia-I, Machado-Joseph disease, or Dentato-Rubro-Pallido-Luysian atrophy locus. Movement disorders : official journal of the Movement Disorder Society. vol 11. issue 2. 1996-08-16. PMID:8684388. we describe a family with dominantly inherited ataxia of late adult onset with relatively "pure" cerebellar signs. 1996-08-16 2023-08-12 Not clear
I D Dukes, L H Philipso. K+ channels: generating excitement in pancreatic beta-cells. Diabetes. vol 45. issue 7. 1996-08-06. PMID:8666132. mutations in k+ channel genes underlie diseases as diverse as persistent hyperinsulinemia of infancy, cardiac long qt syndrome, cerebellar degeneration, and certain ataxias. 1996-08-06 2023-08-12 Not clear
H Maruyama, H Kawakami, S Nakamur. Reevaluation of the exact CAG repeat length in hereditary cerebellar ataxias using highly denaturing conditions and long PCR. Human genetics. vol 97. issue 5. 1996-07-26. PMID:8655136. reevaluation of the exact cag repeat length in hereditary cerebellar ataxias using highly denaturing conditions and long pcr. 1996-07-26 2023-08-12 Not clear
A Lezin, G Cancel, G Stevanin, D Smadja, J C Vernant, A Dürr, J Martial, G G Buisson, R Bellance, H Chneiweiss, Y Agid, A Bric. Autosomal dominant cerebellar ataxia type I in Martinique (French West Indies): genetic analysis of three unrelated SCA2 families. Human genetics. vol 97. issue 5. 1996-07-26. PMID:8655151. autosomal dominant cerebellar ataxias (adcas) are a group of neurodegenerative disorders that are clinically and genetically heterogeneous. 1996-07-26 2023-08-12 Not clear
K Inoue, T Hanihara, Y Yamada, K Kosaka, T Katsuragi, K Iwabuch. Clinical and genetic evaluation of Japanese autosomal dominant cerebellar ataxias; is Machado-Joseph disease common in the Japanese? Journal of neurology, neurosurgery, and psychiatry. vol 60. issue 6. 1996-07-19. PMID:8648347. clinical and genetic evaluation of japanese autosomal dominant cerebellar ataxias; is machado-joseph disease common in the japanese? 1996-07-19 2023-08-12 Not clear
G H Elta, C A Caldwell, T T Nostran. Esophageal dysphagia as the sole symptom in type I Chiari malformation. Digestive diseases and sciences. vol 41. issue 3. 1996-06-11. PMID:8617124. the most common symptoms include headache, syncope, disordered eye movement, sensory loss, weakness, and cerebellar features such as ataxia. 1996-06-11 2023-08-12 Not clear
I Torres-Aleman, V Barrios, A Lledo, J Bercian. The insulin-like growth factor I system in cerebellar degeneration. Annals of neurology. vol 39. issue 3. 1996-05-09. PMID:8602752. since igf-i is present in the olivocerebellar circuitry, two types of late-onset cerebellar ataxias (olivopontocerebellar and idiopathic cerebellar cortical atrophy) were chosen to test this hypothesis. 1996-05-09 2023-08-12 Not clear
I Torres-Aleman, V Barrios, A Lledo, J Bercian. The insulin-like growth factor I system in cerebellar degeneration. Annals of neurology. vol 39. issue 3. 1996-05-09. PMID:8602752. patients suffering from other neurological diseases with cerebellar dysfunction and ataxia not involving the olivocerebellar pathway also had low igf-i levels, while igfbps and insulin levels were normal. 1996-05-09 2023-08-12 Not clear