| Publication |
Sentence |
Publish Date |
Extraction Date |
Species |
| W J Logan, S I Hari. Specific uptake of norepinephrine and dopamine by homogenates of rat cerebral cortex after locus ceruleus lesion. The Journal of neuroscience : the official journal of the Society for Neuroscience. vol 2. issue 3. 1982-05-27. PMID:7062118. |
these results suggest that the specific uptake of norepinephrine and dopamine is a valid marker of noradrenergic innervation of the parietal cortex and that dopamine seems to be accumulated primarily by norepinephrine terminals in this region. |
1982-05-27 |
2023-08-12 |
rat |
| W J Logan, S I Hari. Specific uptake of norepinephrine and dopamine by homogenates of rat cerebral cortex after locus ceruleus lesion. The Journal of neuroscience : the official journal of the Society for Neuroscience. vol 2. issue 3. 1982-05-27. PMID:7062118. |
two and 8 weeks after locus ceruleus lesion, a dmi-insensitive, benztropine-sensitive dopamine uptake becomes apparent in ipsilateral cortical preparations, this may represent proliferation or increased function of dopaminergic cortical projections, compensating for noradrenergic denervation. |
1982-05-27 |
2023-08-12 |
rat |
| J L Elghozi, K H Le Quan-Bui, J T Earnhardt, P Meyer, M A Devync. In vivo dopamine release from the anterior hypothalamus of the rat. European journal of pharmacology. vol 73. issue 2-3. 1982-02-22. PMID:7308291. |
finally, ventral noradrenergic bundle lesion on the same side of the superfusion site considerably enhanced dopamine release which may indicate an inhibitory control of dopamine release by noradrenergic neurons. |
1982-02-22 |
2023-08-12 |
rat |
| B J Clar. Dopamine receptors and the cardiovascular system. Postgraduate medical journal. vol 57 Suppl 1. 1982-01-09. PMID:6117848. |
compounds which selectively stimulate vascular and neuronal dopamine receptors, decrease vascular resistance in the mesenteric, renal and coronary vascular beds, inhibit sympathetic ganglionic transmission and decrease transmitter efflux from certain noradrenergic neurones may prove effective in the treatment of cardiovascular disease and serve, in addition, as useful pharmacological tools. |
1982-01-09 |
2023-08-12 |
Not clear |
| E K Perry, B E Tomlinson, G Blessed, R H Perry, A J Cross, T J Cro. Neuropathological and biochemical observations on the noradrenergic system in Alzheimer's disease. Journal of the neurological sciences. vol 51. issue 2. 1981-11-22. PMID:7276979. |
abnormalities of the noradrenergic system in alzheimer's disease have been investigated by comparing quantitative histological and biochemical components of this transmitter system (locus coeruleus neuron counts and cortical dopamine beta-hydroxylase (dbh) activity, respectively) in a series of demented and non-demented cases. |
1981-11-22 |
2023-08-12 |
Not clear |
| M J Bannon, E B Bunney, R H Rot. Mesocortical dopamine neurons: rapid transmitter turnover compared to other brain catecholamine systems. Brain research. vol 218. issue 1-2. 1981-11-18. PMID:7272743. |
prefrontal cortical dopamine was unaffected by dorsal noradrenergic bundle lesions. |
1981-11-18 |
2023-08-12 |
rat |
| R Deskin, F J Seidler, W L Whitmore, T A Slotki. Development of alpha-noradrenergic and Dopaminergic receptor systems depends on maturation of their presynaptic nerve terminals in the rat brain. Journal of neurochemistry. vol 36. issue 5. 1981-08-20. PMID:6264034. |
uptakes of [(3)h]dopamine or [(3)h]norepinephrine into dopaminergic and noradrenergic nerve terminals were low during the 1st week postpartum and increased rapidly during the 2nd week. |
1981-08-20 |
2023-08-12 |
rat |
| I Nicoletti, P Filipponi, L Fedeli, R Palumbo, P Santori, F Santeusanio, P Brunett. Progesterone positive feedback on gonadotropin release in estrogen-primed postmenopausal women: central nervous system and pituitary as possible sites of action. The Journal of clinical endocrinology and metabolism. vol 53. issue 1. 1981-08-20. PMID:6787064. |
to investigate the site and mode of action of progesterone in inducing gonadotropin release, the effects of catecholamine-depleting (methyldopa) or dopamine agonist (bromocriptine) drugs on progesterone positive feedback and the gonadotropin response to a centrally acting noradrenergic drug (clonidine) were evaluated in estrogen-primed postmenopausal women. |
1981-08-20 |
2023-08-12 |
human |
| I Nicoletti, P Filipponi, L Fedeli, R Palumbo, P Santori, F Santeusanio, P Brunett. Progesterone positive feedback on gonadotropin release in estrogen-primed postmenopausal women: central nervous system and pituitary as possible sites of action. The Journal of clinical endocrinology and metabolism. vol 53. issue 1. 1981-08-20. PMID:6787064. |
the dissociated responses of lh and prl in bromocriptine-pretreated subjects and the significant reduction of the lh rise after progesterone in methyldopa-pretreated women seem to invalidate the hypothesis that a fall in endogenous dopamine is responsible for progesterone positive feedback and suggest that neural noradrenergic mechanisms are involved in progesterone-induced gonadotropin release. |
1981-08-20 |
2023-08-12 |
human |
| A J Lees, R Banniste. The use of lisuride in the treatment of multiple system atrophy with autonomic failure (Shy-Drager syndrome). Journal of neurology, neurosurgery, and psychiatry. vol 44. issue 4. 1981-08-20. PMID:7241162. |
destruction of post-synaptic dopamine receptors and damage to central noradrenergic systems may offer an explanation for the lack of therapeutic effect of lisuride. |
1981-08-20 |
2023-08-12 |
Not clear |
| H Hallberg, O Almgren, J Engel, J Jonaso. Effects of propranolol on the locomotor stimulation induced by activation of postsynaptic catecholamine receptors. Psychopharmacology. vol 72. issue 3. 1981-06-23. PMID:6261288. |
the present study was undertaken in order to clarify the possible involvement of a central beta-adrenoceptor mediated action on the stimulation of locomotor activity by the dopamine agonist apomorphine and the noradrenergic agonist clonidine. |
1981-06-23 |
2023-08-12 |
mouse |
| Y Nomur. The locomotor effect of clonidine and its interaction with alpha-flupenthixol or haloperidol in the developing rat. Naunyn-Schmiedeberg's archives of pharmacology. vol 313. issue 1. 1981-05-21. PMID:7207635. |
it is suggested that intact function of both alpha-adrenoceptors and dopamine receptors is involved in the control of locomotor activity in developing rats and that there is the complex interaction of noradrenergic and dopaminergic system in the control of locomotor activity. |
1981-05-21 |
2023-08-12 |
rat |
| M Sar, W E Stump. Central noradrenergic neurones concentrate 3H-oestradiol. Nature. vol 289. issue 5797. 1981-04-13. PMID:7464918. |
recent studies with antiserum to dopamine-beta-hydroxylase (dbh), the enzyme that converts dopamine to noradrenaline, provided evidence for the existence of noradrenergic cells. |
1981-04-13 |
2023-08-12 |
rat |
| B R Cooper, T J Hester, R A Maxwel. Behavioral and biochemical effects of the antidepressant bupropion (Wellbutrin): evidence for selective blockade of dopamine uptake in vivo. The Journal of pharmacology and experimental therapeutics. vol 215. issue 1. 1981-03-27. PMID:6778989. |
when bupropion or desmethylimipramine was given before intracisternal injections of 6-hydroxydopamine, bupropion produced a dose-related selective antagonism of the destruction of dopamine neurons, while under the same conditions, desmethylimipramine produced a dose-related selective antagonism of the destruction of noradrenergic neurons. |
1981-03-27 |
2023-08-12 |
rat |
| S P Kalra, J W Simpkins, P S Kalr. Effects of pentobarbital on hypothalamic catecholamines and LRH activities. Acta endocrinologica. vol 95. issue 1. 1981-03-17. PMID:7006289. |
pnt administration rapidly reduced the noradrenergic (ne) turnover in the mbh and poa, whereas dopamine (da) turnover was unaltered. |
1981-03-17 |
2023-08-12 |
rat |
| P Songkittiguna, H Majewski, M J Ran. Inhibition by hydralazine of the conversion of dopamine to noradrenaline in rat atria in vitro and in vivo. Clinical and experimental pharmacology & physiology. vol 7. issue 5. 1981-03-17. PMID:7449192. |
hydralazine treatment may result in the appearance of dopamine as a significant co-transmitter in noradrenergic nerves, and this may contribute to the antihypertensive effect of hydralazine. |
1981-03-17 |
2023-08-12 |
rat |
| G Sperk, I Galhaup, E Shlögl, H Hörtnagl, O Hornykiewic. A sensitive and reliable assay for dopamine beta-hydroxylase in tissue. Journal of neurochemistry. vol 35. issue 4. 1981-03-17. PMID:7452299. |
(2) because of removal of endogenous catecholamines, dopamine, the main physiological substrate of dbh in noradrenergic neurons, can be used for the assay. |
1981-03-17 |
2023-08-12 |
rat |
| D L Rowe-Murphy, A van Tienhoven, N R Scott, P E Hillman, C L Wood, A L Johnson, W S Schwar. Effects of 6-hydroxydopamine and pimozide on temperature maintenance by the chicken. The American journal of physiology. vol 239. issue 3. 1981-01-26. PMID:7435601. |
intraventricular implants of pimozide in adult white leghorn hens were used to block dopamine (da) receptors, and 6-hydroxydopamine (6-ohda) was injected intraventricularly to destroy the noradrenergic system locally. |
1981-01-26 |
2023-08-12 |
chicken |
| J S Fink, G P Smit. Relationships between selective denervation of dopamine terminal fields in the anterior forebrain and behavioral responses to amphetamine and apomorphine. Brain research. vol 201. issue 1. 1980-12-18. PMID:7191345. |
to investigate the relationship between denervation of dopamine (da) terminal fields in the anterior forebrain and the behavioral responses to amphetamine (1.5 mg/kg) and apomorphine (1 mg/kg), we injected 6-hydroxydopamine (6-ohda) bilaterally into the anterolateral hypothalamus (alh) or into specific mesolimbic and anterior striatal terminal fields after pretreatment with desmethylimipramine to protect noradrenergic axons and terminals from 6-ohda toxicity. |
1980-12-18 |
2023-08-12 |
Not clear |
| S Z Langer, I Cavero, R Massingha. Recent developments in noradrenergic neurotransmission and its relevance to the mechanism of action of certain antihypertensive agents. Hypertension (Dallas, Tex. : 1979). vol 2. issue 4. 1980-10-27. PMID:6105128. |
a third type of catecholamine receptor found at the noradrenergic nerve ending is the inhibitory dopamine (da) receptor, which might be of significance in the development of new antihypertensive agents. |
1980-10-27 |
2023-08-12 |
Not clear |