All Relations between mog and oligodendrocyte

Publication Sentence Publish Date Extraction Date Species
Y Zhou, X Jia, H Yang, C Chen, X Sun, L Peng, A G Kermode, W Qi. Myelin oligodendrocyte glycoprotein antibody-associated demyelination: comparison between onset phenotypes. European journal of neurology. vol 26. issue 1. 2019-06-03. PMID:30153357. the aim of this study was to analyse the clinical and prognostic features of myelin oligodendrocyte glycoprotein (mog) antibody-associated demyelination with different onset phenotypes. 2019-06-03 2023-08-13 Not clear
Agatha Schlüter, Juan Sandoval, Stéphane Fourcade, Angel Díaz-Lagares, Montserrat Ruiz, Patrizia Casaccia, Manel Esteller, Aurora Pujo. Epigenomic signature of adrenoleukodystrophy predicts compromised oligodendrocyte differentiation. Brain pathology (Zurich, Switzerland). vol 28. issue 6. 2019-05-28. PMID:29476661. we identified a common methylomic signature between the two phenotypes, comprising (i) hypermethylation of genes harboring the h3k27me3 mark at promoter regions, (ii) hypermethylation of genes with major roles in oligodendrocyte differentiation such as mbp, cnp, mog and plp1 and (iii) hypomethylation of immune-associated genes such as ifitm1 and cd59. 2019-05-28 2023-08-13 Not clear
Josef Maxwell Gutman, Mark Kupersmith, Steven Galetta, Ilya Kiste. Anti-myelin oligodendrocyte glycoprotein (MOG) antibodies in patients with optic neuritis and seizures. Journal of the neurological sciences. vol 387. 2019-05-28. PMID:29571858. anti-myelin oligodendrocyte glycoprotein (mog) antibodies in patients with optic neuritis and seizures. 2019-05-28 2023-08-13 Not clear
Josef Maxwell Gutman, Mark Kupersmith, Steven Galetta, Ilya Kiste. Anti-myelin oligodendrocyte glycoprotein (MOG) antibodies in patients with optic neuritis and seizures. Journal of the neurological sciences. vol 387. 2019-05-28. PMID:29571858. we describe four patients who experienced optic neuritis (on) and seizures and were found to have antibodies to myelin oligodendrocyte glycoprotein (mog) in serum. 2019-05-28 2023-08-13 Not clear
S Selek, M Esrefoglu, I Meral, H Bulut, H G Caglar, G Sonuc, C Yildiz, E S Teloglu, N Dogan, B Yuce, E Tiftik, N Bayindi. Effects of Oenothera biennis L. and Hypericum perforatum L. extracts on some central nervous system myelin proteins, brain histopathology and oxidative stress in mice with experimental autoimmune encephalomyelitis. Biotechnic & histochemistry : official publication of the Biological Stain Commission. vol 94. issue 2. 2019-05-23. PMID:30957550. we investigated the effects of oenothera biennis l. and hypericum perforatum l. extracts on brain tissue histopathology, myelin oligodendrocyte glycoprotein (mog), myelin basic protein (mbp), total antioxidant status (tas), total oxidant status (tos) and oxidative stress index (osi) in mice with experimental autoimmune encephalomyelitis (eae). 2019-05-23 2023-08-13 mouse
Joshua S Stoolman, Patrick C Duncker, Amanda K Huber, David A Giles, Jesse M Washnock-Schmid, Athena M Soulika, Benjamin M Sega. An IFNγ/CXCL2 regulatory pathway determines lesion localization during EAE. Journal of neuroinflammation. vol 15. issue 1. 2019-05-20. PMID:30012158. myelin oligodendrocyte glycoprotein (mog)-reactive t-helper (th)1 cells induce conventional experimental autoimmune encephalomyelitis (ceae), characterized by ascending paralysis and monocyte-predominant spinal cord infiltrates, in c57bl/6 wildtype (wt) hosts. 2019-05-20 2023-08-13 Not clear
Ruo-Xuan Sui, Qiang Miao, Jing Wang, Qing Wang, Li-Juan Song, Jing-Wen Yu, Liang Cao, Wei Xiao, Bao-Guo Xiao, Cun-Gen M. Protective and therapeutic role of Bilobalide in cuprizone-induced demyelination. International immunopharmacology. vol 66. 2019-05-14. PMID:30445309. we for the first time found that cpz caused the splenic atrophy and induced the formation of myelin oligodendrocyte glycoprotein (mog) antibody, which was attenuated by bilobalide. 2019-05-14 2023-08-13 Not clear
Kazuto Katsuse, Masanori Kurihara, Yusuke Sugiyama, Satoshi Kodama, Miwako Takahashi, Toshimitsu Momose, Masato Yumoto, Kimihiko Kaneko, Toshiyuki Takahashi, Akatsuki Kubota, Toshihiro Hayashi, Tatsushi Tod. Aphasic status epilepticus preceding tumefactive left hemisphere lesion in anti-MOG antibody associated disease. Multiple sclerosis and related disorders. vol 27. 2019-04-17. PMID:30347340. anti-myelin oligodendrocyte glycoprotein (mog) antibodies have recently been associated with epilepsy with flair hyperintense cortical lesions on mri. 2019-04-17 2023-08-13 Not clear
R Deschamps, A Gueguen, A Lecler, K Lecouturier, C Lamirel, C Bensa, R Marignier, C Vignal, O Gou. Acute idiopathic optic neuritis: not always benign. European journal of neurology. vol 25. issue 11. 2019-04-16. PMID:30004610. we compared this cohort with patients with on related to myelin oligodendrocyte glycoprotein (mog) or on related to aquaporin-4 (aqp4) antibodies. 2019-04-16 2023-08-13 Not clear
Priyanka Khare, Dilip K Challa, Siva Charan Devanaboyina, Ramraj Velmurugan, Samuel Hughes, Benjamin M Greenberg, Raimund J Ober, E Sally War. Myelin oligodendrocyte glycoprotein-specific antibodies from multiple sclerosis patients exacerbate disease in a humanized mouse model. Journal of autoimmunity. vol 86. 2019-04-08. PMID:28964723. myelin oligodendrocyte glycoprotein (mog) is exposed on the outer surface of the myelin sheath, and as such, represents a possible target antigen for antibodies in multiple sclerosis (ms) and other demyelinating diseases. 2019-04-08 2023-08-13 mouse
Yara Dadalti Fragoso, Nise Alessandra C Sousa, Soniza Vieira Alves-Leon, Ronaldo Maciel Dias, Maria Lucia V Pimentel, Sidney Gomes, Marcus Vinicius M Goncalves, Carla Vieira Stella, Carlos Bernardo Tauil, Andrea Anacleto, Caroline Vieira Spessotto, Eber Castro Correa, Audred C Biondo Eboni, Alfredo Damasceno, Benito Damasceno, João Gabriel D Farinhas, Rhea Sylvia de Souza Mota, Eduardo G Almeida Nogueira, Valeria Coelho S R Pereira, Claudio Scorcine, Tamar Bacon, Ilya Kiste. Clinical characteristics of 153 Brazilian patients with neuromyelitis optica spectrum disorder (NMOSD). Multiple sclerosis and related disorders. vol 27. 2019-04-04. PMID:30504040. some patients are seropositive for antibodies against aquaporin-4 (aqp4), others are positive for anti-myelin oligodendrocyte glycoprotein (mog), while a few are negative for both biomarkers. the disease is complex, and only now are specific therapeutic clinical trials being carried out. 2019-04-04 2023-08-13 Not clear
Estefania Ugarte-Berzal, Nele Berghmans, Lise Boon, Erik Martens, Jennifer Vandooren, Bénédicte Cauwe, Greet Thijs, Paul Proost, Jo Van Damme, Ghislain Opdenakke. Gelatinase B/matrix metalloproteinase-9 is a phase-specific effector molecule, independent from Fas, in experimental autoimmune encephalomyelitis. PloS one. vol 13. issue 10. 2019-03-13. PMID:30273366. we provoked eae with myelin oligodendrocyte glycoprotein (mog) peptide and compared eae development in four genotypes (wild-type (wt), single knockout mmp-9-/-, faslpr, and mmp-9-/-/faslpr) and monitored leukocytes, cytokines and chemokines as immunological parameters. 2019-03-13 2023-08-13 mouse
Reginald C Adiele, Chiedukam A Adiel. Metabolic defects in multiple sclerosis. Mitochondrion. vol 44. 2019-03-08. PMID:29246870. bioenergetics, oxygen and ion metabolism dominate the metabolic and biochemical pathways that maintain neuronal viability and impulse transmission which directly or indirectly point to mitochondrial integrity and adenosine triphosphate (atp) availability indicating the involvement of mitochondria in the pathogenesis of ms. loss of myelin proteins including myelin basic protein (mbp), proteolipid protein (plp), myelin associated glycoprotein (mag), myelin oligodendrocyte glycoproetin (mog), 2, 3,-cyclic nucleotide phosphodiestarase (cnpase); microglia and microphage activation, oligodendrocyte apoptosis as well as expression of inducible nitric oxide synthase (i-nos) and myeloperoxidase activities have been implicated in a subset of balo's type and relapsing remitting ms (rrms) lesions indicating the involvement of metabolic defects and oxidative stress in ms. 2019-03-08 2023-08-13 Not clear
Monika Bradl, Markus Reindl, Hans Lassman. Mechanisms for lesion localization in neuromyelitis optica spectrum disorders. Current opinion in neurology. vol 31. issue 3. 2019-03-05. PMID:29465432. neuromyelitis optica spectrum disorders (nmosd) are severe inflammatory diseases of the central nervous system (cns), with the presence of aquaporin 4 (aqp4)-specific serum antibodies in the vast majority of patients, and with the presence of myelin oligodendrocyte glycoprotein (mog)-specific antibodies in approximately 40% of all aqp4-antibody negative nmosd patients. 2019-03-05 2023-08-13 Not clear
Bahadır Konuskan, Mirac Yildirim, Rahsan Gocmen, Tuncay Derya Okur, Ipek Polat, Huseyin Kilic, Sema Saltik, Zeynep Ozturk, Kivilcim Gucuyener, Sakir Altunbasak, Tamer Celik, Gulsen Kose, Arzu Yilmaz, Mustafa Komur, Hulya Kayilioglu, Banu Anla. Retrospective analysis of children with myelin oligodendrocyte glycoprotein antibody-related disorders. Multiple sclerosis and related disorders. vol 26. 2019-02-28. PMID:30212767. knowledge has been expanding on myelin oligodendrocyte glycoprotein (mog) antibody-associated central nervous system disorders. 2019-02-28 2023-08-13 Not clear
Viktoria Papp, Annika R Langkilde, Morten Blinkenberg, Karen Schreiber, Poul Erik Hyldgaard Jensen, Finn Sellebjer. Clinical utility of anti-MOG antibody testing in a Danish cohort. Multiple sclerosis and related disorders. vol 26. 2019-02-28. PMID:30227311. anti-myelin oligodendrocyte glycoprotein (mog) antibody (ab) can be found in different immune-mediated inflammatory cns disorders. 2019-02-28 2023-08-13 Not clear
Mercedes Giralt, Amalia Molinero, Juan Hidalg. Active Induction of Experimental Autoimmune Encephalomyelitis (EAE) with MOG Methods in molecular biology (Clifton, N.J.). vol 1791. 2019-02-18. PMID:30006713. mice are immunized with an emulsion of myelin oligodendrocyte glycoprotein peptide 35-55 (mog 2019-02-18 2023-08-13 mouse
Satoshi Kitagawa, Takashi Osada, Kimihiko Kaneko, Toshiyuki Takahashi, Norihiro Suzuki, Jin Nakahar. [Clinical analysis of opticospinal multiple sclerosis (OSMS) presentation detecting anti-myelin oligodendrocyte glycoprotein (MOG) antibody]. Rinsho shinkeigaku = Clinical neurology. vol 58. issue 12. 2019-01-30. PMID:30487359. [clinical analysis of opticospinal multiple sclerosis (osms) presentation detecting anti-myelin oligodendrocyte glycoprotein (mog) antibody]. 2019-01-30 2023-08-13 Not clear
Hiroshi Adachi, Yukiko Ide, Toshiyuki Takahashi, Yukihiro Yoneda, Yasufumi Kageyam. [Cerebral cortical encephalitis with anti-myelin oligodendrocyte glycoprotein (MOG) antibody]. Rinsho shinkeigaku = Clinical neurology. vol 58. issue 12. 2019-01-30. PMID:30487364. [cerebral cortical encephalitis with anti-myelin oligodendrocyte glycoprotein (mog) antibody]. 2019-01-30 2023-08-13 Not clear
Hiroshi Adachi, Yukiko Ide, Toshiyuki Takahashi, Yukihiro Yoneda, Yasufumi Kageyam. [Cerebral cortical encephalitis with anti-myelin oligodendrocyte glycoprotein (MOG) antibody]. Rinsho shinkeigaku = Clinical neurology. vol 58. issue 12. 2019-01-30. PMID:30487364. serum anti-myelin oligodendrocyte glycoprotein (mog) antibody was positive, while anti-aquaporin-4 antibody, anti-n-methyl-d-aspartate (nmda) receptor antibody, and other autoimmune antibodies were all negative. 2019-01-30 2023-08-13 Not clear