| Publication |
Sentence |
Publish Date |
Extraction Date |
Species |
| L C Foote, T J Schneider, G M Fischer, J K Wang, B Rasmussen, K A Campbell, D H Lynch, S T Ju, A Marshak-Rothstein, T L Rothstei. Intracellular signaling for inducible antigen receptor-mediated Fas resistance in B cells. Journal of immunology (Baltimore, Md. : 1950). vol 157. issue 5. 1996-11-22. PMID:8757305. |
susceptibility to apoptosis is counteracted by ag receptor binding that produces a state of resistance to fas engagement in otherwise sensitive targets. |
1996-11-22 |
2023-08-12 |
Not clear |
| L C Foote, T J Schneider, G M Fischer, J K Wang, B Rasmussen, K A Campbell, D H Lynch, S T Ju, A Marshak-Rothstein, T L Rothstei. Intracellular signaling for inducible antigen receptor-mediated Fas resistance in B cells. Journal of immunology (Baltimore, Md. : 1950). vol 157. issue 5. 1996-11-22. PMID:8757305. |
in the present study, protection from th1-mediated apoptosis was found to be induced by protein kinase c and calcium signals, which in combination mimicked the level of fas resistance produced by surface ig engagement. |
1996-11-22 |
2023-08-12 |
Not clear |
| F Novelli, P Bernabei, L Ozmen, L Rigamonti, A Allione, S Pestka, G Garotta, G Forn. Switching on of the proliferation or apoptosis of activated human T lymphocytes by IFN-gamma is correlated with the differential expression of the alpha- and beta-chains of its receptor. Journal of immunology (Baltimore, Md. : 1950). vol 157. issue 5. 1996-11-22. PMID:8757312. |
to find out how physiologically secreted ifn-gamma controls either the proliferation or the apoptosis of human t lymphocytes, the kinetics of expression of the alpha- and beta-chains of its receptor (ifn-gamma r) were sequentially followed on t lymphocytes first activated with pha and then cultured in the presence of il-2, and related to the kinetics of expression of fas, bcl-2, and il-2r p55 chain. |
1996-11-22 |
2023-08-12 |
human |
| F Novelli, P Bernabei, L Ozmen, L Rigamonti, A Allione, S Pestka, G Garotta, G Forn. Switching on of the proliferation or apoptosis of activated human T lymphocytes by IFN-gamma is correlated with the differential expression of the alpha- and beta-chains of its receptor. Journal of immunology (Baltimore, Md. : 1950). vol 157. issue 5. 1996-11-22. PMID:8757312. |
when these t lymphoblasts were restimulated by pha, okt3, or staphylococcus enterotoxin beta-pokeweed mitogen, both chains up-modulated and most cells underwent apoptosis in a way apparently independent of bcl-2, but not of fas. |
1996-11-22 |
2023-08-12 |
human |
| K Yasutomo, H Nagasawa, H Hisaeda, R A Good, Y Kuroda, K Himen. Fas system-mediated apoptosis suppresses lymphopoiesis. Journal of immunology (Baltimore, Md. : 1950). vol 157. issue 5. 1996-11-22. PMID:8757318. |
fas system-mediated apoptosis suppresses lymphopoiesis. |
1996-11-22 |
2023-08-12 |
mouse |
| K Yasutomo, H Nagasawa, H Hisaeda, R A Good, Y Kuroda, K Himen. Fas system-mediated apoptosis suppresses lymphopoiesis. Journal of immunology (Baltimore, Md. : 1950). vol 157. issue 5. 1996-11-22. PMID:8757318. |
in this study, we investigated the effects and regulatory influences attributable to fas ligand overexpression on lymphocyte development to clarify the role of fas system-mediated apoptosis in lymphopoiesis in vivo. |
1996-11-22 |
2023-08-12 |
mouse |
| H Asahara, T Hasumuna, T Kobata, H Yagita, K Okumura, H Inoue, S Gay, T Sumida, K Nishiok. Expression of Fas antigen and Fas ligand in the rheumatoid synovial tissue. Clinical immunology and immunopathology. vol 81. issue 1. 1996-11-20. PMID:8808638. |
to understand the role of apoptosis through fas/fas ligand (fas-l) interaction in the pathogenesis of rheumatoid arthritis (ra), we examined the expression of fas antigen, fas-l, and apoptosis in synovial tissue obtained from eight patients with ra and five patients with osteoarthritis (oa). |
1996-11-20 |
2023-08-12 |
Not clear |
| H Asahara, T Hasumuna, T Kobata, H Yagita, K Okumura, H Inoue, S Gay, T Sumida, K Nishiok. Expression of Fas antigen and Fas ligand in the rheumatoid synovial tissue. Clinical immunology and immunopathology. vol 81. issue 1. 1996-11-20. PMID:8808638. |
immunohistochemical staining and the dna nick end labeling (tunel) method were combined and revealed that approximately 10 to 30% of fas antigen-expressing cells in the ra synovium showed dna fragmentation characteristic for apoptosis. |
1996-11-20 |
2023-08-12 |
Not clear |
| G H Wong, R L Kaspar, G Zweiger, C Carlson, S E Fong, N Ehsani, G Veha. Strategies for manipulating apoptosis for cancer therapy with tumor necrosis factor and lymphotoxin. Journal of cellular biochemistry. vol 60. issue 1. 1996-11-20. PMID:8825416. |
although the p55-tnf/lt receptor is structurally related to the fas receptor, they can each signal apoptosis by distinct pathways. |
1996-11-20 |
2023-08-12 |
Not clear |
| P Wattre, V Bert, D Hobe. [Apoptosis and human viral infections]. Annales de biologie clinique. vol 54. issue 5. 1996-11-18. PMID:8869358. |
activated lymphocytes responding to viral infections express high levels of fas and are susceptible to apoptosis. |
1996-11-18 |
2023-08-12 |
human |
| P Wattre, V Bert, D Hobe. [Apoptosis and human viral infections]. Annales de biologie clinique. vol 54. issue 5. 1996-11-18. PMID:8869358. |
inversly, cytomegalovirus, hepatitis c virus and myxoviruses up-regulate fas antigen prior to undergoing apoptosis. |
1996-11-18 |
2023-08-12 |
human |
| M X Wu, Z Ao, M Hegen, C Morimoto, S F Schlossma. Requirement of Fas(CD95), CD45, and CD11a/CD18 in monocyte-dependent apoptosis of human T cells. Journal of immunology (Baltimore, Md. : 1950). vol 157. issue 2. 1996-11-07. PMID:8752920. |
requirement of fas(cd95), cd45, and cd11a/cd18 in monocyte-dependent apoptosis of human t cells. |
1996-11-07 |
2023-08-12 |
human |
| M X Wu, Z Ao, M Hegen, C Morimoto, S F Schlossma. Requirement of Fas(CD95), CD45, and CD11a/CD18 in monocyte-dependent apoptosis of human T cells. Journal of immunology (Baltimore, Md. : 1950). vol 157. issue 2. 1996-11-07. PMID:8752920. |
moreover, this mo-dependent t cell apoptosis could be blocked by a fas-ig fusion protein, as well as by a nonlytic mab against fas molecule. |
1996-11-07 |
2023-08-12 |
human |
| M Walchner, C Leib-Mösch, G Messer, P Kin. [Endogenous retroviral sequences as a factor in the pathogenesis of systemic lupus erythematosus]. Der Hautarzt; Zeitschrift fur Dermatologie, Venerologie, und verwandte Gebiete. vol 47. issue 7. 1996-11-07. PMID:8926164. |
in mrl-lpr/lpr mice with sle-like disease the insertion of a mobile retroviral element, the early transposon (etn), into the second intron of the fas gene leads to reduced apoptosis, accumulation of lymphocytes and earlier mortality. |
1996-11-07 |
2023-08-12 |
mouse |
| J Wu, J Wilson, J He, L Xiang, P H Schur, J D Mount. Fas ligand mutation in a patient with systemic lupus erythematosus and lymphoproliferative disease. The Journal of clinical investigation. vol 98. issue 5. 1996-11-04. PMID:8787672. |
the apoptosis genes, fas and fas ligand (fasl), are candidate contributory genes in human sle, as mutations of these genes result in autoimmunity in several murine models of this disease. |
1996-11-04 |
2023-08-12 |
human |
| C Redondo, I Flores, A Gonzalez, S Nagata, A C Carrera, I Merida, C Martinez-. Linomide prevents the lethal effect of anti-Fas antibody and reduces Fas-mediated ceramide production in mouse hepatocytes. The Journal of clinical investigation. vol 98. issue 5. 1996-11-04. PMID:8787688. |
heart, lung, and ovary showed no signs of apoptosis promoted by fas ligation. |
1996-11-04 |
2023-08-12 |
mouse |
| M M Keane, S A Ettenberg, G A Lowrey, E K Russell, S Lipkowit. Fas expression and function in normal and malignant breast cell lines. Cancer research. vol 56. issue 20. 1996-11-04. PMID:8841000. |
fas mrna increased slightly in both cell lines that became sensitive and in the cell line that remained resistant to fas-mediated apoptosis upon ifn-gamma treatment. |
1996-11-04 |
2023-08-12 |
human |
| M M Keane, S A Ettenberg, G A Lowrey, E K Russell, S Lipkowit. Fas expression and function in normal and malignant breast cell lines. Cancer research. vol 56. issue 20. 1996-11-04. PMID:8841000. |
together, these data suggest that down-regulation of fas and its pathway may be a step in tumor progression and that modulation of fas expression may provide an approach to inducing apoptosis in breast cancer cells. |
1996-11-04 |
2023-08-12 |
human |
| J A DiGiuseppe, P LeBeau, J Augenbraun, M J Borowit. Multiparameter flow-cytometric analysis of bcl-2 and Fas expression in normal and neoplastic hematopoiesis. American journal of clinical pathology. vol 106. issue 3. 1996-10-29. PMID:8816592. |
proteins that modulate apoptosis include bcl-2, which inhibits apoptosis, and fas (cd95, also known as apo-1), which induces apoptosis in susceptible cells bound by fas ligand (fasl). |
1996-10-29 |
2023-08-12 |
Not clear |
| H Hebestreit, S Yousefi, I Balatti, M Weber, R Crameri, D Simon, K Hartung, A Schapowal, K Blaser, H U Simo. Expression and function of the Fas receptor on human blood and tissue eosinophils. European journal of immunology. vol 26. issue 8. 1996-10-24. PMID:8765020. |
these data suggest that fas ligand/fas interactions are involved in the regulation of eosinophil apoptosis and that defects in this system could contribute to the accumulation of these cells in allergic and asthmatic diseases. |
1996-10-24 |
2023-08-12 |
human |