| Publication |
Sentence |
Publish Date |
Extraction Date |
Species |
| M S Buchsbau. Frontal lobes, basal ganglia, temporal lobes--three sites for schizophrenia? Schizophrenia bulletin. vol 16. issue 3. 1991-03-22. PMID:2287928. |
frontal lobes, basal ganglia, temporal lobes--three sites for schizophrenia? |
1991-03-22 |
2023-08-11 |
Not clear |
| M S Buchsbau. Frontal lobes, basal ganglia, temporal lobes--three sites for schizophrenia? Schizophrenia bulletin. vol 16. issue 3. 1991-03-22. PMID:2287928. |
this special issue of the schizophrenia bulletin focuses on three brain areas hypothesized to play a role in the etiology of schizophrenia--the frontal lobes, the basal ganglia, and the temporal lobes. |
1991-03-22 |
2023-08-11 |
Not clear |
| M S Buchsbau. The frontal lobes, basal ganglia, and temporal lobes as sites for schizophrenia. Schizophrenia bulletin. vol 16. issue 3. 1991-03-22. PMID:2287929. |
the frontal lobes, basal ganglia, and temporal lobes as sites for schizophrenia. |
1991-03-22 |
2023-08-11 |
Not clear |
| M S Buchsbau. The frontal lobes, basal ganglia, and temporal lobes as sites for schizophrenia. Schizophrenia bulletin. vol 16. issue 3. 1991-03-22. PMID:2287929. |
positron emission tomography studies with fluorodeoxyglucose in patients with schizophrenia are reviewed and findings in the frontal lobes, basal ganglia, and temporal lobes summarized from more than 20 published studies. |
1991-03-22 |
2023-08-11 |
Not clear |
| A Pfefferbaum, K O Lim, M Rosenbloom, R B Zipursk. Brain magnetic resonance imaging: approaches for investigating schizophrenia. Schizophrenia bulletin. vol 16. issue 3. 1991-03-22. PMID:2287935. |
techniques for evaluating frontal lobes, temporal lobes, and basal ganglia integrity are reviewed, and recent observations on these brain regions in patients with schizophrenia are described. |
1991-03-22 |
2023-08-11 |
Not clear |
| R Sandyk, S R Ka. The relationship of negative schizophrenia to parkinsonism. The International journal of neuroscience. vol 55. issue 1. 1991-01-28. PMID:2147920. |
as such, negative symptoms may be a manifestation of disease of the basal ganglia and constitute the core pathology in schizophrenia. |
1991-01-28 |
2023-08-11 |
Not clear |
| M Carlsson, A Carlsso. Interactions between glutamatergic and monoaminergic systems within the basal ganglia--implications for schizophrenia and Parkinson's disease. Trends in neurosciences. vol 13. issue 7. 1990-08-23. PMID:1695402. |
interactions between glutamatergic and monoaminergic systems within the basal ganglia--implications for schizophrenia and parkinson's disease. |
1990-08-23 |
2023-08-11 |
mouse |
| A Fernandez-Bouzas, B Angrist, P Hemdal, L A Adler, J Rotrose. Basal ganglia calcification in schizophrenia. Biological psychiatry. vol 27. issue 6. 1990-05-24. PMID:2322632. |
basal ganglia calcification in schizophrenia. |
1990-05-24 |
2023-08-11 |
Not clear |
| M F Casanova, C M Prasad, I Waldman, B Illowsky, B Stein, D R Weinberger, J B Kleinma. No difference in basal ganglia mineralization between schizophrenic and nonschizophrenic patients: a quantitative computerized tomographic study. Biological psychiatry. vol 27. issue 2. 1990-02-22. PMID:2294977. |
in addition, several postmortem studies suggest that increased mineralization (especially iron) of the basal ganglia is a possible clinicopathological correlate of schizophrenia. |
1990-02-22 |
2023-08-11 |
human |
| M S Buchsbaum, R J Haie. Functional and anatomical brain imaging: impact on schizophrenia research. Schizophrenia bulletin. vol 13. issue 1. 1987-07-30. PMID:3496658. |
decreased activity in the basal ganglia, which can be reversed with neuroleptic treatment, is also seen in schizophrenia. |
1987-07-30 |
2023-08-11 |
Not clear |
| M S Buchsbaum, J C Wu, L E DeLisi, H H Holcomb, E Hazlett, K Cooper-Langston, R Kessle. Positron emission tomography studies of basal ganglia and somatosensory cortex neuroleptic drug effects: differences between normal controls and schizophrenic patients. Biological psychiatry. vol 22. issue 4. 1987-05-26. PMID:2882788. |
glucose metabolic rate in the basal ganglia, thalamus, and somatosensory cortex was examined in eight patients with schizophrenia before and after receiving neuroleptic medication. |
1987-05-26 |
2023-08-11 |
Not clear |
| M S Buchsbaum, J C Wu, L E DeLisi, H H Holcomb, E Hazlett, K Cooper-Langston, R Kessle. Positron emission tomography studies of basal ganglia and somatosensory cortex neuroleptic drug effects: differences between normal controls and schizophrenic patients. Biological psychiatry. vol 22. issue 4. 1987-05-26. PMID:2882788. |
metabolic rates in the basal ganglia tended to be low in patients with schizophrenia in comparison to 24 age- and sex-matched controls. |
1987-05-26 |
2023-08-11 |
Not clear |
| B Bogerts, E Meertz, R Schönfeldt-Bausc. Basal ganglia and limbic system pathology in schizophrenia. A morphometric study of brain volume and shrinkage. Archives of general psychiatry. vol 42. issue 8. 1985-08-21. PMID:4015323. |
basal ganglia and limbic system pathology in schizophrenia. |
1985-08-21 |
2023-08-11 |
Not clear |
| A Francis, H Freema. Psychiatric abnormality and brain calcification over four generations. The Journal of nervous and mental disease. vol 172. issue 3. 1984-04-12. PMID:6142086. |
an association between basal ganglia calcification and psychiatric abnormality, mainly schizophrenia, is described in three generations of a family; schizophrenia is also recorded in the previous generation. |
1984-04-12 |
2023-08-12 |
Not clear |
| S Roffler-Tarlov, A M Graybie. Weaver mutation has differential effects on the dopamine-containing innervation of the limbic and nonlimbic striatum. Nature. vol 307. issue 5946. 1984-02-23. PMID:6690983. |
the original distinction between nigrostriatal and mesolimbic systems nevertheless was pivotal in suggesting that the basal ganglia are related to limbic as well as to sensorimotor functions, and remains of interest because dopaminergic mechanisms may be implicated not only in the aetiology of sensorimotor impairments such as those of parkinson's disease, but also in neuropsychiatric disorders such as schizophrenia. |
1984-02-23 |
2023-08-12 |
mouse |
| A Arregui, A V Mackay, E G Spokes, L L Iverse. Reduced activity of angiotensin-converting enzyme in basal ganglia in early onset schizophrenia. Psychological medicine. vol 10. issue 2. 1980-08-25. PMID:6247736. |
reduced activity of angiotensin-converting enzyme in basal ganglia in early onset schizophrenia. |
1980-08-25 |
2023-08-12 |
Not clear |
| T I Lidsky, P M Weinhold, F M Levin. Implications of basal ganglionic dysfunction for schizophrenia. Biological psychiatry. vol 14. issue 1. 1979-05-24. PMID:420906. |
this paper is addressed to presenting evidence that the basal ganglia are involved in mediating schizophrenia. |
1979-05-24 |
2023-08-11 |
Not clear |
| T I Lidsky, P M Weinhold, F M Levin. Implications of basal ganglionic dysfunction for schizophrenia. Biological psychiatry. vol 14. issue 1. 1979-05-24. PMID:420906. |
taken together, these data are in accord with the hypothesis that some dysfunction involving the basal ganglia is a major factor in schizophrenia. |
1979-05-24 |
2023-08-11 |
Not clear |