All Relations between Obesity Hypoventilation Syndrome and osa

Publication Sentence Publish Date Extraction Date Species
Lucio Casali, Pierluigi Carratù, Matteo Sofi. Clinical variability of respiratory pulmonary hypertension: implications for diagnosis and management. Multidisciplinary respiratory medicine. vol 8. issue 1. 2013-12-16. PMID:24280232. the recognition of out of proportion respiratory ph in several chronic respiratory diseases which include copd, idiopathic pulmonary fibrosis (ipf), combined pulmonary fibrosis and emphysema, obstructive sleep apnea (osa), obesity hypoventilation syndrome (ohs) may be important for a comprehensive clinical classification of severe respiratory ph, as well as, for the inclusion of these patients in randomized clinical trials on ph targeted therapy. 2013-12-16 2023-08-12 Not clear
Poul Jennum, Rikke Ibsen, Jakob Kjellber. Morbidity prior to a diagnosis of sleep-disordered breathing: a controlled national study. Journal of clinical sleep medicine : JCSM : official publication of the American Academy of Sleep Medicine. vol 9. issue 2. 2013-08-15. PMID:23372461. most studies have focused on cardiovascular diseases (cvds) after a diagnosis of obstructive sleep apnea (osa) or obesity hypoventilation syndrome (ohs); however, the overall morbidity prior to an sdb diagnosis has not been evaluated. 2013-08-15 2023-08-12 Not clear
Hideaki Nakayam. [Sleep-disordered breathing]. Nihon rinsho. Japanese journal of clinical medicine. vol 71. issue 2. 2013-07-30. PMID:23631208. and obesity hypoventilation syndrome (ohs) is defined as chronic daytime hypercapnia in combination with obesity and osa. 2013-07-30 2023-08-12 Not clear
Timothy I Morgenthaler, R Nisha Aurora, Terry Brown, Rochelle Zak, Cathy Alessi, Brian Boehlecke, Andrew L Chesson, Leah Friedman, Vishesh Kapur, Rama Maganti, Judith Owens, Jeffrey Pancer, Todd J Swic. Practice parameters for the use of autotitrating continuous positive airway pressure devices for titrating pressures and treating adult patients with obstructive sleep apnea syndrome: an update for 2007. An American Academy of Sleep Medicine report. Sleep. vol 31. issue 1. 2008-03-06. PMID:18220088. current recommendations follow: (1) apap devices are not recommended to diagnose osa; (2) patients with congestive heart failure, patients with significant lung disease such as chronic obstructive pulmonary disease; patients expected to have nocturnal arterial oxyhemoglobin desaturation due to conditions other than osa (e.g., obesity hypoventilation syndrome); patients who do not snore (either naturally or as a result of palate surgery); and patients who have central sleep apnea syndromes are not currently candidates for apap titration or treatment; (3) apap devices are not currently recommended for split-night titration; (4) certain apap devices may be used during attended titration with polysomnography to identify a single pressure for use with standard cpap for treatment of moderate to severe osa; (5) certain apap devices may be initiated and used in the self-adjusting mode for unattended treatment of patients with moderate to severe osa without significant comorbidities (chf, copd, central sleep apnea syndromes, or hypoventilation syndromes); (6) certain apap devices may be used in an unattended way to determine a fixed cpap treatment pressure for patients with moderate to severe osa without significant comorbidities (chf, copd, central sleep apnea syndromes, or hypoventilation syndromes); (7) patients being treated with fixed cpap on the basis of apap titration or being treated with apap must have close clinical follow-up to determine treatment effectiveness and safety; and (8) a reevaluation and, if necessary, a standard attended cpap titration should be performed if symptoms do not resolve or the apap treatment otherwise appears to lack efficacy. 2008-03-06 2023-08-12 Not clear
Babak Mokhlesi, Aiman Tulaimat, Ilja Faibussowitsch, Yue Wang, Arthur T Evan. Obesity hypoventilation syndrome: prevalence and predictors in patients with obstructive sleep apnea. Sleep & breathing = Schlaf & Atmung. vol 11. issue 2. 2007-10-19. PMID:17187265. patients with obesity hypoventilation syndrome (ohs) have a lower quality of life, more healthcare expenses, a greater risk of pulmonary hypertension, and a higher mortality compared to eucapnic patients with obstructive sleep apnea (osa). 2007-10-19 2023-08-12 Not clear
Wataru Hid. Quality of life in obesity hypoventilation syndrome. Sleep & breathing = Schlaf & Atmung. vol 7. issue 1. 2003-08-19. PMID:12712391. the first paper of this issue of sleep and breathing reports that the quality of life (qol) assessed by the sf-36 and the epworth sleepiness scale (ess) in obesity hypoventilation syndrome (ohs) was compared with age- and body mass index-matched patients without hypoventilation (obese osa), nonobese osa patients, and healthy subjects. 2003-08-19 2023-08-12 human
Wataru Hida, Shinichi Okabe, Koichiro Tatsumi, Hiroshi Kimura, Tsuneto Akasiba, Kazuo Chin, Motoharu Ohi, Hideaki Nakayama, Makoto Satoh, Takayuki Kuriyam. Nasal continuous positive airway pressure improves quality of life in obesity hypoventilation syndrome. Sleep & breathing = Schlaf & Atmung. vol 7. issue 1. 2003-08-19. PMID:12712392. we studied the quality of life of obesity hypoventilation syndrome (ohs) by comparing it with age- and body mass index-matched patients without hypoventilation and age-matched obstructive sleep apnea (osa) patients with body mass index (bmi) under 30, and the efficacy of nasal continuous positive airway pressure (cpap) therapy for 3 to 6 months on the quality of life in these patients. 2003-08-19 2023-08-12 human
Michael Littner, Maxwell Hirshkowitz, David Davila, W McDowell Anderson, Clete A Kushida, B Tucker Woodson, Stephen F Johnson, S Wise Merril. Practice parameters for the use of auto-titrating continuous positive airway pressure devices for titrating pressures and treating adult patients with obstructive sleep apnea syndrome. An American Academy of Sleep Medicine report. Sleep. vol 25. issue 2. 2002-09-11. PMID:11902424. 2) apap titration and apap treatment are not currently recommended for patients with congestive heart failure, significant lung disease (e.g., chronic obstructive pulmonary disease), daytime hypoxemia and respiratory failure from any cause, or prominent nocturnal desaturation other than from osa (e.g., obesity hypoventilation syndrome). 2002-09-11 2023-08-12 Not clear
C P O'Donnell, C G Tankersley, V P Polotsky, A R Schwartz, P L Smit. Leptin, obesity, and respiratory function. Respiration physiology. vol 119. issue 2-3. 2000-05-18. PMID:10722859. thus, the possibility exists that a relative deficiency in leptin, or a leptin resistance, may play a role in obesity-related breathing disorders such as obesity hypoventilation syndrome (ohs) or obstructive sleep apnea (osa). 2000-05-18 2023-08-12 mouse
W Pankow, N Hijjeh, F Schüttler, T Penzel, H F Becker, J H Peter, P von Wicher. Influence of noninvasive positive pressure ventilation on inspiratory muscle activity in obese subjects. The European respiratory journal. vol 10. issue 12. 1998-04-23. PMID:9493672. eighteen obese subjects with a body mass index > or = 40 kg x m(-2) were investigated during the daytime, which included five healthy controls (simple obesity (so)), seven patients with obstructive sleep apnoea (osa) and six patients with the obesity hypoventilation syndrome (ohs). 1998-04-23 2023-08-12 human