All Relations between Depression and cerebral cortex

Publication Sentence Publish Date Extraction Date Species
J Olesen, S Vorstrup, M Lauritze. rCBF after TIA and during migraine attacks. Cephalalgia : an international journal of headache. vol 5 Suppl 2. 1985-09-16. PMID:4016940. in classic migraine, alterations of rcbf are completely different, indicating a mechanism progressing in the cerebral cortex, probably the spreading depression of leao. 1985-09-16 2023-08-11 Not clear
C J Schmidt, J W Gib. Role of the serotonin uptake carrier in the neurochemical response to methamphetamine: effects of citalopram and chlorimipramine. Neurochemical research. vol 10. issue 5. 1985-08-22. PMID:3859754. in vivo, coadministration of citalopram or chlorimipramine with methamphetamine blocked the irreversible depression of tryptophan hydroxylase activity observed in the neostriatum and cerebral cortex after repeated administration of high doses of methamphetamine. 1985-08-22 2023-08-11 Not clear
L Csiba, W Paschen, G Mie. Regional changes in tissue pH and glucose content during cortical spreading depression in rat brain. Brain research. vol 336. issue 1. 1985-08-20. PMID:4005572. after eliciting a single passage of spreading depression in rat brain cortex, regional alterations in glucose content and tissue ph were studied in relation to changes in cortical steady potential. 1985-08-20 2023-08-11 rat
L Csiba, W Paschen, G Mie. Regional changes in tissue pH and glucose content during cortical spreading depression in rat brain. Brain research. vol 336. issue 1. 1985-08-20. PMID:4005572. migration of spreading depression into non-invaded cortex was accompanied by a drastic decrease in glucose concentration and tissue ph which persisted longer than the transient depolarization of cortical cells. 1985-08-20 2023-08-11 rat
K Tanaka, S C Jones, E Dora, J H Greenberg, M Reivic. Effect of iodoacetate on local cerebral blood flow and glucose metabolism in cats: a double-radionuclide autoradiographic study. Journal of cerebral blood flow and metabolism : official journal of the International Society of Cerebral Blood Flow and Metabolism. vol 5. issue 2. 1985-06-20. PMID:3988828. a marked increase in lcbf, accompanied by a moderate to severe depression of lcmrglu, was observed in the iaa-superfused cortex. 1985-06-20 2023-08-11 Not clear
G G Somjen, P G Aitke. The ionic and metabolic responses associated with neuronal depression of Leão's type in cerebral cortex and in hippocampal formation. Anais da Academia Brasileira de Ciencias. vol 56. issue 4. 1985-06-12. PMID:6442833. the ionic and metabolic responses associated with neuronal depression of leão's type in cerebral cortex and in hippocampal formation. 1985-06-12 2023-08-12 Not clear
M Ito, M Miyaoka, S Ishi. [Alterations in local cerebral glucose utilization during various anesthesia--the effect of urethane and a review]. No to shinkei = Brain and nerve. vol 36. issue 12. 1985-05-02. PMID:6529517. the neural structures in which no metabolic depression was observed were entorhinal cortex, many nuclei in hypothalamus, medial habenula-interpeduncular nucleus, nucleus tractus solitarius, and some white matters. 1985-05-02 2023-08-12 rat
V L Biank. [Lateralization factors in the vertebrate brain]. Fiziologicheskii zhurnal SSSR imeni I. M. Sechenova. vol 69. issue 7. 1985-01-31. PMID:6391964. conditioning in white rats and suppression of a hemisphere's cortex with spreading depression revealed that the right hemisphere prevailed in perception of space, in the simultaneous information processing, in perception of concrete signs and in deductive processing whereas the left hemisphere was predominant in perception of time, in successive information processing, in perception of abstract signs and in inductive processing. 1985-01-31 2023-08-12 rat
M Lauritze. Long-lasting reduction of cortical blood flow of the brain after spreading depression with preserved autoregulation and impaired CO2 response. Journal of cerebral blood flow and metabolism : official journal of the International Society of Cerebral Blood Flow and Metabolism. vol 4. issue 4. 1984-12-31. PMID:6438124. in control regions, cbf changed by 2.8-3.9% per mm hg paco2 change, whereas in the spreading depression cortex, cbf changed by only 1.3-1.5%. 1984-12-31 2023-08-12 rat
M Lauritze. Long-lasting reduction of cortical blood flow of the brain after spreading depression with preserved autoregulation and impaired CO2 response. Journal of cerebral blood flow and metabolism : official journal of the International Society of Cerebral Blood Flow and Metabolism. vol 4. issue 4. 1984-12-31. PMID:6438124. the difference between control and spreading depression cortex was significant at p less than 0.001, indicating a reduced co2 sensitivity of the cortical regions invaded by spreading depression. 1984-12-31 2023-08-12 rat
N Nisimaru, M Yamamoto, I Shimoyam. Inhibitory effects of cerebellar cortical stimulation on sympathetic nerve activity in rabbits. The Japanese journal of physiology. vol 34. issue 3. 1984-12-20. PMID:6492498. the effective sites which caused the marked depression on the sympathetic nerve activity were distributed in the ipsilateral vermal cortex, predominantly in the medial part. 1984-12-20 2023-08-12 Not clear
P F Warren, M A Peat, J W Gib. The effects of a single dose of amphetamine and iprindole on the serotonergic system of the rat brain. Neuropharmacology. vol 23. issue 7A. 1984-10-19. PMID:6206434. marked decreases in the activity of tryptophan hydroxylase (tph) were observed in both the cerebral cortex and neostriatum after 6 hr, with maximum depression observed at 24 hr. 1984-10-19 2023-08-12 rat
E Dóra, L Gyulai, A G Kovác. Determinants of brain activation-induced cortical NAD/NADH responses in vivo. Brain research. vol 299. issue 1. 1984-07-16. PMID:6326966. in order to elucidate that which are the factors that may influence the direction of brain activation-induced changes in the redox state of oxidized/reduced nicotinamide adenine dinucleotide (nad/nadh), the brain cortex was electrically stimulated during arterial hypotension and following reinfusion of the shed blood, during arterial hyper- and hypoxia, and during the second phase of spreading cortical depression (sd). 1984-07-16 2023-08-12 Not clear
G Mies, W Pasche. Regional changes of blood flow, glucose, and ATP content determined on brain sections during a single passage of spreading depression in rat brain cortex. Experimental neurology. vol 84. issue 2. 1984-06-07. PMID:6714339. regional changes of blood flow, glucose, and atp content determined on brain sections during a single passage of spreading depression in rat brain cortex. 1984-06-07 2023-08-12 rat
J Kriváne. Magnesium ions potentiate the formation of adenosine 3',5'-monophosphate induced by depolarizing agents in the rat cerebral cortex in vivo. Physiologia Bohemoslovaca. vol 33. issue 1. 1984-05-16. PMID:6324256. pretreatment of the rat cerebral cortex by topical application of 1.5 mol x 1(-1) mgcl2 in order to prevent the induction of spreading depression by depolarizing agents did not inhibit but potentiated the ability of 1.0 mol x 1(-1) k+ (39 pmol camp/mg prot.) 1984-05-16 2023-08-12 rat
J Kriváne. In vitro phosphorylation of proteins in the preparations of cerebral cortex invaded by spreading depression. Physiologia Bohemoslovaca. vol 33. issue 1. 1984-05-16. PMID:6709731. in vitro phosphorylation of proteins in the preparations of cerebral cortex invaded by spreading depression. 1984-05-16 2023-08-12 Not clear
V L Biank. Hemisphere specialization of the animal brain for information processing principles. The International journal of neuroscience. vol 20. issue 1-2. 1984-04-13. PMID:6668114. hemisphere cortex inactivation was carried out by means of spreading depression. 1984-04-13 2023-08-12 rat
W A Mutch, A J Hanse. Extracellular pH changes during spreading depression and cerebral ischemia: mechanisms of brain pH regulation. Journal of cerebral blood flow and metabolism : official journal of the International Society of Cerebral Blood Flow and Metabolism. vol 4. issue 1. 1984-03-15. PMID:6693512. the lactate concentration in brain cortex increased from baseline 1.2 mm to 7.0 mm (n = 6) during the maximal acidic change in spreading depression. 1984-03-15 2023-08-12 rat
W A Mutch, A J Hanse. Extracellular pH changes during spreading depression and cerebral ischemia: mechanisms of brain pH regulation. Journal of cerebral blood flow and metabolism : official journal of the International Society of Cerebral Blood Flow and Metabolism. vol 4. issue 1. 1984-03-15. PMID:6693512. superfusion of the cortex with inhibitors of carbonic anhydrase (acetazolamide), na+/h+ counter transport (amiloride), and cl-/hco-3 countertransport (4,4'-diisothiocyanostilbene-2,2'-disulfonic acid) altered the triphasic phe changes in a similar fashion for both spreading depression and cerebral ischemia, providing insights into the phe regulatory mechanisms in mammalian brain. 1984-03-15 2023-08-12 rat
A S Saratikov, T A Zamoshchin. [Efficacy of lithium hydroxybutyrate in reserpine depression]. Biulleten' eksperimental'noi biologii i meditsiny. vol 96. issue 11. 1984-01-27. PMID:6685545. long-term administration of lithium hydroxybutyrate (10 mg/kg, for 7 days) prevents the depression of avoidance behavior in rats and eeg alterations in the cortex and subcortical structures of rabbit brain induced by a single injection of 0.125 mg/kg reserpine. 1984-01-27 2023-08-12 rat