All Relations between Dementia and amygdala

Publication Sentence Publish Date Extraction Date Species
C Maunoury, J L Michot, H Caillet, V Parlato, A Leroy-Willig, P Jehenson, A Syrota, F Bolle. Specificity of temporal amygdala atrophy in Alzheimer's disease: quantitative assessment with magnetic resonance imaging. Dementia (Basel, Switzerland). vol 7. issue 1. 1996-10-31. PMID:8788076. the aim of the study was to assess the specificity of temporal amygdala (ta) atrophy with magnetic resonance imaging (mri) by comparing a group of early impaired patients with alzheimer's disease (ad) with 'other types of dementia' and controls. 1996-10-31 2023-08-12 human
V E Anderson, N J Cairns, P N Leig. Involvement of the amygdala, dentate and hippocampus in motor neuron disease. Journal of the neurological sciences. vol 129 Suppl. 1995-11-24. PMID:7595627. we have observed that some cases of mnd with dementia (mnd-d), had ubiquitinated inclusions in the amygdala. 1995-11-24 2023-08-12 Not clear
C Bancher, K A Jellinge. Neurofibrillary tangle predominant form of senile dementia of Alzheimer type: a rare subtype in very old subjects. Acta neuropathologica. vol 88. issue 6. 1995-03-31. PMID:7879604. in a consecutive autopsy series of 580 demented elderly subjects, 256 with the clinical diagnosis of probable/possible alzheimer's disease (ad), there were 10 cases aged between 80 and 99 years with moderate to severe dementia or confusional state in which neuropathological studies revealed abundant neurofibrillary tangles with predominant involvement of the allocortex (entorhinal region, subiculum, ca 1 sector of hippocampus, amygdala) but no or only very few senile plaques. 1995-03-31 2023-08-12 human
D Nochlin, G van Belle, T D Bird, S M Sum. Comparison of the severity of neuropathologic changes in familial and sporadic Alzheimer's disease. Alzheimer disease and associated disorders. vol 7. issue 4. 1994-03-11. PMID:8305189. we compared the density of neurofibrillary tangles (nt) and neuritic (senile) plaques (np) in 10 cortical areas, the amygdala, the hippocampus, the parahippocampal gyrus, and the cerebellum in patients with familial (fad) and sporadic (sad) alzheimer's disease with early (< 55 years), intermediate (55-70 years) and late (> 70 years) ages of onset of dementia and age-matched controls. 1994-03-11 2023-08-12 Not clear
J P Aggleto. The contribution of the amygdala to normal and abnormal emotional states. Trends in neurosciences. vol 16. issue 8. 1993-10-21. PMID:7691009. in parallel with this, it appears increasingly likely that amygdala dysfunction contributes to the emotional changes that accompany certain neurological disorders, including dementia and schizophrenia. 1993-10-21 2023-08-12 monkey
P V Arriagada, J H Growdon, E T Hedley-Whyte, B T Hyma. Neurofibrillary tangles but not senile plaques parallel duration and severity of Alzheimer's disease. Neurology. vol 42. issue 3 Pt 1. 1992-04-23. PMID:1549228. we found that (1) the severity of dementia was positively related to the number of nfts in neocortex, but not to the degree of sp deposition; (2) nfts accumulate in a consistent pattern reflecting hierarchic vulnerability of individual cytoarchitectural fields; (3) nfts appeared in the entorhinal cortex, ca1/subiculum field of the hippocampal formation, and the amygdala early in the disease process; and (4) the degree of sp deposition was also related to a hierarchic vulnerability of certain brain areas to accumulate sps, but the pattern of sp distribution was different from that of nft. 1992-04-23 2023-08-11 Not clear
S M Sumi, T D Bird, D Nochlin, M A Raskin. Familial presenile dementia with psychosis associated with cortical neurofibrillary tangles and degeneration of the amygdala. Neurology. vol 42. issue 1. 1992-03-02. PMID:1734292. familial presenile dementia with psychosis associated with cortical neurofibrillary tangles and degeneration of the amygdala. 1992-03-02 2023-08-11 Not clear
T Matsuzawa, T Hishinuma, H Matsui, K Meguro, M Ueda, S Kinomura, K Yamad. Severe atrophy of amygdala and hippocampus in both Alzheimer's disease and multi-infarct dementia. The science reports of the research institutes, Tohoku University. Ser. C, Medicine. Tohoku Daigaku. vol 37. issue 1-4. 1991-10-10. PMID:2103656. severe atrophy of amygdala and hippocampus in both alzheimer's disease and multi-infarct dementia. 1991-10-10 2023-08-11 Not clear
T Matsuzawa, T Hishinuma, H Matsui, K Meguro, M Ueda, S Kinomura, K Yamad. Severe atrophy of amygdala and hippocampus in both Alzheimer's disease and multi-infarct dementia. The science reports of the research institutes, Tohoku University. Ser. C, Medicine. Tohoku Daigaku. vol 37. issue 1-4. 1991-10-10. PMID:2103656. with magnetic resonance imaging, severe atrophy of amygdala and hippocampus in both alzheimer's disease and multi-infarct dementia was observed. 1991-10-10 2023-08-11 Not clear
T Matsuzaw. Limbic system, the main focus of dementia syndrome--a study with MRI and PET. The science reports of the research institutes, Tohoku University. Ser. C, Medicine. Tohoku Daigaku. vol 37. issue 1-4. 1991-10-10. PMID:2103658. with mri there is drastic atrophy and destruction of the amygdala and hippocampus suggesting the limbic system as the focus of dementia syndrome. 1991-10-10 2023-08-11 Not clear
C T Dickson, C H Vanderwol. Animal models of human amnesia and dementia: hippocampal and amygdala ablation compared with serotonergic and cholinergic blockade in the rat. Behavioural brain research. vol 41. issue 3. 1991-04-03. PMID:2288673. animal models of human amnesia and dementia: hippocampal and amygdala ablation compared with serotonergic and cholinergic blockade in the rat. 1991-04-03 2023-08-11 human
C T Dickson, C H Vanderwol. Animal models of human amnesia and dementia: hippocampal and amygdala ablation compared with serotonergic and cholinergic blockade in the rat. Behavioural brain research. vol 41. issue 3. 1991-04-03. PMID:2288673. the behavioral effects of combined bilateral hippocampal and amygdala ablation (previously proposed as a model of human global amnesia) were compared to those seen with central blockade of the ascending cholinergic and serotonergic projections (a possible model of human global dementia) in male hooded rats. 1991-04-03 2023-08-11 human
P Cervera, C Duyckaerts, M Ruberg, E Hirsch, G Ransmayr, J J Hauw, Y Agi. Tyrosine hydroxylase-like immunoreactivity in senile plaques is not related to the density of tyrosine hydroxylase-positive fibers in patients with Alzheimer's disease. Neuroscience letters. vol 110. issue 1-2. 1990-05-24. PMID:1970142. the number of silver-stained senile plaques was lowest in the hippocampus and highest in the amygdala, and increased in all three structures in relation to the degree of dementia in the patients. 1990-05-24 2023-08-11 human
S M de la Monte, S E Wells, T Hedley-Whyte, J H Growdo. Neuropathological distinction between Parkinson's dementia and Parkinson's plus Alzheimer's disease. Annals of neurology. vol 26. issue 3. 1989-11-08. PMID:2802531. the findings suggest that dementia in pdd is mainly subcortical in origin and due to neuronal degeneration in basal ganglia, the amygdala, and thalamus. 1989-11-08 2023-08-11 Not clear
S J Kish, Y Robitaille, M el-Awar, J H Deck, J Simmons, L Schut, L J Chang, L DiStefano, M Freedma. Non-Alzheimer-type pattern of brain cholineacetyltransferase reduction in dominantly inherited olivopontocerebellar atrophy. Annals of neurology. vol 26. issue 3. 1989-11-08. PMID:2802535. the lack of severe disabling dementia in our opca patients compared with ad patients having a similar cortical cholinergic reduction could be explained by an absence of either a marked cholinergic loss in amygdala or hippocampus or significant loss of noncholinergic cerebral cortical and limbic neurons as occurs in ad brain. 1989-11-08 2023-08-11 Not clear
S Nakamura, T Kawamata, I Akiguchi, M Kameyama, H Kimur. [Catecholaminergic systems in the amygdaloid complex of SDAT and aged controls: tyrosine hydroxylase immunohistochemistry]. Rinsho shinkeigaku = Clinical neurology. vol 29. issue 3. 1989-08-31. PMID:2568896. catecholaminergic systems in the amygdaloid complex in patients with senile dementia of alzheimer type (sdat) and aged controls have been studied with tyrosine hydroxylase (th) immunohistochemistry. 1989-08-31 2023-08-11 human
S Nakamura, T Kawamata, I Akiguchi, M Kameyama, H Kimur. [Somatostatin-immunoreactive neurons in the amygdaloid complex in patients with Alzheimer type dementia and aged controls]. Rinsho shinkeigaku = Clinical neurology. vol 28. issue 2. 1988-09-29. PMID:2900710. [somatostatin-immunoreactive neurons in the amygdaloid complex in patients with alzheimer type dementia and aged controls]. 1988-09-29 2023-08-11 Not clear
R E Arbogast, M R Kozlowsk. Quantitative morphometric analysis of the neurotoxic effects of the excitotoxin, ibotenic acid, on the basal forebrain. Neurotoxicology. vol 9. issue 1. 1988-08-23. PMID:3393301. since the globus pallidus is spared in victims of senile dementia of the alzheimer type (sdat), although the adjacent nbm and amygdala are damaged, these data are not consistent with the theory that the damage to the basal forebrain seen in sdat victims is produced by elevated levels of endogenous excitotoxin. 1988-08-23 2023-08-11 Not clear
P K Mölsä, E Säkö, L Paljärvi, J O Rinne, U K Rinn. Alzheimer's disease: neuropathological correlates of cognitive and motor disorders. Acta neurologica Scandinavica. vol 75. issue 6. 1987-10-20. PMID:3630634. a multiple regression model consisting of plaques and tangles in amygdala, gyrus frontalis medius, gyrus angularis, and gyrus temporalis medius, plaques of gyrus rectus, tangles of the hippocampus, gyrus precentralis and gyrus cinguli together with brain weight, emerged to link dementia to neuropathological changes at the level of maximum significance. 1987-10-20 2023-08-11 Not clear
A M Palmer, A W Procter, G C Stratmann, D M Bowe. Excitatory amino acid-releasing and cholinergic neurones in Alzheimer's disease. Neuroscience letters. vol 66. issue 2. 1986-08-12. PMID:3014387. brains of normal controls and patients with primary degenerative dementia were investigated for choline acetyltransferase (chat) activity in the frontal, temporal and parietal cortex, hippocampus, amygdala and thalamus. 1986-08-12 2023-08-11 Not clear