All Relations between apoptosis and met-2
| Publication | Sentence | Publish Date | Extraction Date | Species |
|---|---|---|---|---|
| Yuting Shao, Yunhui Li, Dayong Wan. Polylactic acid microplastics cause transgenerational reproductive toxicity associated with activation of insulin and hedgehog ligands in C. elegans. The Science of the total environment. 2024-06-08. PMID:38851356. | additionally, pla-mp caused transgenerational increase in expression of met-2 and set-6 encoding histone methylation transferases, and germline apoptosis induced by pla-mp could be suppressed by rnai of met-2 and set-6. | 2024-06-08 | 2024-06-11 | caenorhabditis_elegans |
| Yuting Shao, Yunhui Li, Dayong Wan. Polylactic acid microplastics cause transgenerational reproductive toxicity associated with activation of insulin and hedgehog ligands in C. elegans. The Science of the total environment. 2024-06-08. PMID:38851356. | dysregulated expressions of some apoptosis and dna damage related genes caused by pla-mp were reversed by rnai of ins-39, wrt-3, met-2, and set-6. | 2024-06-08 | 2024-06-11 | caenorhabditis_elegans |
| Peter Zeller, Jan Padeken, Robin van Schendel, Veronique Kalck, Marcel Tijsterman, Susan M Gasse. Histone H3K9 methylation is dispensable for Caenorhabditis elegans development but suppresses RNA:DNA hybrid-associated repeat instability. Nature genetics. vol 48. issue 11. 2017-09-08. PMID:27668659. | in met-2 set-25 double mutants, which lack all h3k9 methylation (h3k9me), embryos differentiate normally, although mutant adults are sterile owing to extensive dna-damage-driven apoptosis in the germ line. | 2017-09-08 | 2023-08-13 | caenorhabditis_elegans |